Cross-Presented Antigen Be Deleted by Soluble , but Not T Cells Can + Tissue-Resident Memory CD 8

[1]  S. Bromley,et al.  Chemokine receptor CCR7 guides T cell exit from peripheral tissues and entry into afferent lymphatics , 2005, Nature Immunology.

[2]  E. Butcher,et al.  Chemokine receptor CCR7 required for T lymphocyte exit from peripheral tissues , 2005, Nature Immunology.

[3]  Leo Lefrançois,et al.  Initial T cell frequency dictates memory CD8+ T cell lineage commitment , 2005, Nature Immunology.

[4]  H. Pircher,et al.  Complex Memory T-Cell Phenotypes Revealed by Coexpression of CD62L and CCR7 , 2005, Journal of Virology.

[5]  J. Drijfhout,et al.  Cross-presentation by intercellular peptide transfer through gap junctions , 2005, Nature.

[6]  L. Sherman,et al.  Distinct Requirements for Deletion versus Anergy during CD8 T Cell Peripheral Tolerance In Vivo1 , 2005, The Journal of Immunology.

[7]  F. Celada,et al.  CD8 memory T cells: cross-reactivity and heterologous immunity , 2004, Seminars in Immunology.

[8]  D. Voehringer,et al.  Constitutive Expression of CCR7 Directs Effector CD8 T Cells into the Splenic White Pulp and Impairs Functional Activity1 , 2004, The Journal of Immunology.

[9]  D. Tscharke,et al.  CD8+ T Cell Cross-Priming via Transfer of Proteasome Substrates , 2004, Science.

[10]  L. Lefrançois,et al.  Dynamics of blood-borne CD8 memory T cell migration in vivo. , 2004, Immunity.

[11]  R. Ward,et al.  Activated Primary and Memory CD8 T Cells Migrate to Nonlymphoid Tissues Regardless of Site of Activation or Tissue of Origin1 , 2004, The Journal of Immunology.

[12]  K. Rock,et al.  Cellular protein is the source of cross-priming antigen in vivo. , 2004, Proceedings of the National Academy of Sciences of the United States of America.

[13]  A. Valujskikh,et al.  In remembrance of things past: memory T cells and transplant rejection , 2003, Immunological reviews.

[14]  R. Steinman,et al.  Tolerogenic dendritic cells. , 2003, Annual review of immunology.

[15]  M. Sayegh,et al.  Memory T cells: a hurdle to immunologic tolerance. , 2003, Journal of the American Society of Nephrology : JASN.

[16]  E. Wherry,et al.  Heterologous immunity provides a potent barrier to transplantation tolerance. , 2003, The Journal of clinical investigation.

[17]  D. Griffin.  Immune responses to RNA-virus infections of the CNS , 2003, Nature Reviews Immunology.

[18]  D. Greiner,et al.  Direct Visualization of Cross-Reactive Effector and Memory Allo-Specific CD8 T Cells Generated in Response to Viral Infections1 , 2003, The Journal of Immunology.

[19]  R. Steinman,et al.  Dendritic Cell Function in Vivo during the Steady State: A Role in Peripheral Tolerance , 2003, Annals of the New York Academy of Sciences.

[20]  Michel C. Nussenzweig,et al.  Efficient Targeting of Protein Antigen to the Dendritic Cell Receptor DEC-205 in the Steady State Leads to Antigen Presentation on Major Histocompatibility Complex Class I Products and Peripheral CD8+ T Cell Tolerance , 2002, The Journal of experimental medicine.

[21]  Alan D. Roberts,et al.  Long-Term Maintenance of Virus-Specific Effector Memory CD8+ T Cells in the Lung Airways Depends on Proliferation1 , 2002, The Journal of Immunology.

[22]  R. Steinman,et al.  Immune Tolerance After Delivery of Dying Cells to Dendritic Cells In Situ , 2002, The Journal of experimental medicine.

[23]  L. Sherman,et al.  Memory CD8(+) T cells undergo peripheral tolerance. , 2002, Immunity.

[24]  R. Steinman,et al.  The CD8+ Dendritic Cell Subset Selectively Endocytoses Dying Cells in Culture and In Vivo , 2002, The Journal of experimental medicine.

[25]  Wolfgang Weninger,et al.  Migratory Properties of Naive, Effector, and Memory Cd8+ T Cells , 2001, The Journal of experimental medicine.

[26]  Michel C. Nussenzweig,et al.  Dendritic Cells Induce Peripheral T Cell Unresponsiveness under Steady State Conditions in Vivo , 2001, The Journal of experimental medicine.

[27]  D. Woodland,et al.  Antigen-Specific CD8+ T Cells Persist in the Upper Respiratory Tract Following Influenza Virus Infection1 , 2001, The Journal of Immunology.

[28]  Simon C Watkins,et al.  Dendritic Cells Acquire Antigens from Live Cells for Cross-Presentation to CTL1 , 2001, The Journal of Immunology.

[29]  L. Lefrançois,et al.  Preferential Localization of Effector Memory Cells in Nonlymphoid Tissue , 2001, Science.

[30]  Alan D. Roberts,et al.  Activated Antigen-Specific CD8+ T Cells Persist in the Lungs Following Recovery from Respiratory Virus Infections1 , 2001, The Journal of Immunology.

[31]  J. Noseworthy.  Progress in determining the causes and treatment of multiple sclerosis , 1999, Nature.

[32]  H. Hartung,et al.  Immunopathogenesis of multiple sclerosis: the role of T cells. , 1999, Current opinion in neurology.

[33]  P. Debré,et al.  Chronic intravenous injections of antigen induce and maintain tolerance in T cell receptor‐transgenic mice , 1999, European journal of immunology.

[34]  J. Burns,et al.  Isolation of myelin basic protein–specific T cells predominantly from the memory T‐cell compartment in multiple sclerosis , 1999, Annals of neurology.

[35]  A. Khoruts,et al.  Direct evidence that functionally impaired CD4+ T cells persist in vivo following induction of peripheral tolerance. , 1998, Journal of immunology.

[36]  J. Ernerudh,et al.  CD4 and CD8 lymphocyte subsets in cerebrospinal fluid and peripheral blood from patients with multiple sclerosis, meningitis and normal controls , 1998, Acta neurologica Scandinavica.

[37]  S. Tonegawa,et al.  Tolerance Induction and Autoimmune Encephalomyelitis Amelioration After Administration of Myelin Basic Protein–derived Peptide , 1997, The Journal of experimental medicine.

[38]  R M Zinkernagel,et al.  Peptide antigen treatment of naive and virus-immune mice: antigen-specific tolerance versus immunopathology. , 1997, Immunity.

[39]  R. Steinman,et al.  Dendritic cells in the T‐cell areas of lymphoid organs , 1997, Immunological reviews.

[40]  S. Miller,et al.  Inhibition of relapsing experimental autoimmune encephalomyelitis in SJL mice by feeding the immunodominant PLP139‐151 peptide , 1996, Journal of neuroscience research.

[41]  D. Wraith,et al.  Mucosal Tolerance in a Murine Model of Experimental Autoimmune Encephalomyelitis a , 1996, Annals of the New York Academy of Sciences.

[42]  D. Wraith,et al.  Affinity for class II MHC determines the extent to which soluble peptides tolerize autoreactive T cells in naive and primed adult mice--implications for autoimmunity. , 1995, International immunology.

[43]  H. Pircher,et al.  T cell priming versus T cell tolerance induced by synthetic peptides , 1995, The Journal of experimental medicine.

[44]  P. Doherty,et al.  CD8+ T-cell memory to viruses. , 1994, Current opinion in immunology.

[45]  H. Pircher,et al.  Peptide-induced T-cell tolerance to prevent autoimmune diabetes in a transgenic mouse model. , 1994, Proceedings of the National Academy of Sciences of the United States of America.

[46]  Hans Hengartner,et al.  T cell immunity after a viral infection versus T cell tolerance induced by soluble viral peptides , 1993, European journal of immunology.

[47]  A. Gaur,et al.  Amelioration of autoimmune encephalomyelitis by myelin basic protein synthetic peptide-induced anergy. , 1992, Science.

[48]  J. Villadangos,et al.  Regulation of antigen presentation and cross-presentation in the dendritic cell network: facts, hypothesis, and immunological implications. , 2005, Advances in immunology.

[49]  L. Cauley,et al.  Memory T-cells in non-lymphoid tissues. , 2002, Current opinion in investigational drugs.

[50]  C Caux,et al.  Immunobiology of dendritic cells. , 2000, Annual review of immunology.

[51]  P. Knopf,et al.  Cervical lymphatics, the blood-brain barrier and the immunoreactivity of the brain: a new view. , 1992, Immunology today.