Association study of childhood food allergy with genome-wide association studies-discovered loci of atopic dermatitis and eosinophilic esophagitis.

[1]  Komei Ito,et al.  Diagnosis of food allergy based on oral food challenge test. , 2009, Allergology international : official journal of the Japanese Society of Allergology.

[2]  Thomas Blicher,et al.  A genome-wide association study identifies CDHR3 as a susceptibility locus for early childhood asthma with severe exacerbations , 2013, Nature Genetics.

[3]  Miles Parkes,et al.  Genetic insights into common pathways and complex relationships among immune-mediated diseases , 2013, Nature Reviews Genetics.

[4]  D. Postma,et al.  Sequence variants affecting eosinophil numbers associate with asthma and myocardial infarction , 2009, Nature Genetics.

[5]  D. Venkataraman,et al.  Filaggrin loss-of-function mutations are associated with food allergy in childhood and adolescence. , 2014, The Journal of allergy and clinical immunology.

[6]  G. Lack.  Update on risk factors for food allergy. , 2012, The Journal of allergy and clinical immunology.

[7]  Andrew M. Rupert,et al.  Genome-wide association analysis of eosinophilic esophagitis provides insight into the tissue specificity of this allergic disease , 2014, Nature Genetics.

[8]  Christian Gieger,et al.  A genome-wide association study of plasma total IgE concentrations in the Framingham Heart Study. , 2012, The Journal of allergy and clinical immunology.

[9]  I Sayers,et al.  Genetic risk factors for the development of allergic disease identified by genome-wide association , 2014, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.

[10]  L. Liang,et al.  A genome-wide association study of atopic dermatitis identifies loci with overlapping effects on asthma and psoriasis , 2013, Human molecular genetics.

[11]  Manuel A. R. Ferreira,et al.  Genome-Wide Association Studies of Asthma in Population-Based Cohorts Confirm Known and Suggested Loci and Identify an Additional Association near HLA , 2012, PloS one.

[12]  Gonçalo R. Abecasis,et al.  Genetic variants regulating ORMDL3 expression contribute to the risk of childhood asthma , 2007, Nature.

[13]  M. Rothenberg.  Molecular, genetic, and cellular bases for treating eosinophilic esophagitis. , 2015, Gastroenterology.

[14]  Yusuke Nakamura,et al.  High-density genotyping study identifies four new susceptibility loci for atopic dermatitis , 2013, Nature Genetics.

[15]  Joseph T. Glessner,et al.  Common variants at 5q22 associate with pediatric eosinophilic esophagitis , 2010, Nature Genetics.

[16]  Florence Demenais,et al.  A large-scale, consortium-based genomewide association study of asthma. , 2010, The New England journal of medicine.

[17]  Chuong B. Do,et al.  A genome-wide association meta-analysis of self-reported allergy identifies shared and allergy-specific susceptibility loci , 2013, Nature Genetics.

[18]  E. Bleecker,et al.  Genome-wide association study of asthma identifies RAD50-IL13 and HLA-DR/DQ regions. , 2010, The Journal of allergy and clinical immunology.

[19]  D. Vercelli,et al.  IL-13 R130Q, a common variant associated with allergy and asthma, enhances effector mechanisms essential for human allergic inflammation. , 2005, The Journal of clinical investigation.

[20]  Manolis Kellis,et al.  FTO Obesity Variant Circuitry and Adipocyte Browning in Humans. , 2015, The New England journal of medicine.

[21]  Yusuke Nakamura,et al.  Genome-wide association study identifies eight new susceptibility loci for atopic dermatitis in the Japanese population , 2012, Nature Genetics.

[22]  A. Bredenoord,et al.  GWAS identifies four novel eosinophilic esophagitis loci , 2014, Nature Communications.

[23]  Yan Wang,et al.  Genome-wide association study identifies two new susceptibility loci for atopic dermatitis in the Chinese Han population , 2011, Nature Genetics.

[24]  Christian Gieger,et al.  META-ANALYSIS OF GENOME-WIDE ASSOCIATION STUDIES IDENTIFIES THREE NEW RISK LOCI FOR ATOPIC DERMATITIS , 2011, Nature Genetics.

[25]  L. Liang,et al.  Genome-Wide Scan on Total Serum IgE Levels Identifies FCER1A as Novel Susceptibility Locus , 2008, PLoS genetics.

[26]  J. Hayashi,et al.  Filaggrin loss-of-function mutations are not a predisposing factor for atopic dermatitis in an Ishigaki Island under subtropical climate. , 2014, Journal of dermatological science.

[27]  M. Nishimura,et al.  Japanese-specific filaggrin gene mutations in Japanese patients suffering from atopic eczema and asthma. , 2010, The Journal of investigative dermatology.

[28]  David J Balding,et al.  A genome-wide meta-analysis of genetic variants associated with allergic rhinitis and grass sensitization and their interaction with birth order. , 2011, The Journal of allergy and clinical immunology.

[29]  S. Heath,et al.  A common variant on chromosome 11q13 is associated with atopic dermatitis , 2009, Nature Genetics.

[30]  K. Bønnelykke,et al.  Atopic endotype in childhood. , 2016, The Journal of allergy and clinical immunology.

[31]  H. Hakonarson,et al.  HLA‐DQ strikes again: Genome‐wide association study further confirms HLA‐DQ in the diagnosis of asthma among adults , 2012, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.

[32]  Kari Stefansson,et al.  Meta-analysis of genome-wide association studies identifies ten loci influencing allergic sensitization , 2013, Nature Genetics.

[33]  L. Wain,et al.  Genome-wide association study to identify genetic determinants of severe asthma , 2011, Thorax.