Naringin Attenuates Autophagic Stress and Neuroinflammation in Kainic Acid-Treated Hippocampus In Vivo
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[1] K. Vafeiadou,et al. The neuroprotective potential of flavonoids: a multiplicity of effects , 2008, Genes & Nutrition.
[2] D. Saluja,et al. Naringin protects against kainic acid-induced status epilepticus in rats: evidence for an antioxidant, anti-inflammatory and neuroprotective intervention. , 2011, Biological & pharmaceutical bulletin.
[3] Á. Simonyi,et al. Kainic acid-mediated excitotoxicity as a model for neurodegeneration , 2007, Molecular Neurobiology.
[4] R. Sankar,et al. Inflammation enhances epileptogenesis in the developing rat brain , 2010, Neurobiology of Disease.
[5] C. Hamani,et al. Changes in Hippocampal Volume are Correlated with Cell Loss but Not with Seizure Frequency in Two Chronic Models of Temporal Lobe Epilepsy , 2014, Front. Neurol..
[6] Philippe Dessen,et al. Inhibition of Macroautophagy Triggers Apoptosis , 2005, Molecular and Cellular Biology.
[7] Wei Wang,et al. Excitotoxicity of TNFα derived from KA activated microglia on hippocampal neurons in vitro and in vivo , 2010, Journal of neurochemistry.
[8] N Guthrie,et al. Inhibition of human breast cancer cell proliferation and delay of mammary tumorigenesis by flavonoids and citrus juices. , 1996, Nutrition and cancer.
[9] Tallie Z. Baram,et al. The role of inflammation in epilepsy , 2011, Nature Reviews Neurology.
[10] R. Burke,et al. In vivo AAV1 transduction with hRheb(S16H) protects hippocampal neurons by BDNF production. , 2015, Molecular therapy : the journal of the American Society of Gene Therapy.
[11] Chi Li,et al. Growth Factor Regulation of Autophagy and Cell Survival in the Absence of Apoptosis , 2005, Cell.
[12] G. Yen,et al. Neuroprotective effects of citrus flavonoids. , 2012, Journal of agricultural and food chemistry.
[13] A. Vezzani,et al. Functional Role of Inflammatory Cytokines and Antiinflammatory Molecules in Seizures and Epileptogenesis , 2002, Epilepsia.
[14] R. Racine,et al. Modification of seizure activity by electrical stimulation. II. Motor seizure. , 1972, Electroencephalography and clinical neurophysiology.
[15] I. Módy,et al. The process of epileptogenesis: a pathophysiological approach , 2001, Current opinion in neurology.
[16] Z. Qin,et al. An autophagic mechanism is involved in apoptotic death of rat striatal neurons induced by the non-N-methyl-D-aspartate receptor agonist kainic acid , 2008, Autophagy.
[17] Anil Kumar,et al. Protective effect of naringin against ischemic reperfusion cerebral injury: possible neurobehavioral, biochemical and cellular alterations in rat brain. , 2009, European journal of pharmacology.
[18] Jie Zhu,et al. Kainic Acid-Induced Neurodegenerative Model: Potentials and Limitations , 2010, Journal of biomedicine & biotechnology.
[19] G. Sudhandiran,et al. Naringin modulates oxidative stress and inflammation in 3-nitropropionic acid-induced neurodegeneration through the activation of nuclear factor-erythroid 2-related factor-2 signalling pathway , 2012, Neuroscience.
[20] K. Roth,et al. Kainic acid induces early and transient autophagic stress in mouse hippocampus , 2007, Neuroscience Letters.
[21] Sang-Joon Park,et al. Naringin protects the nigrostriatal dopaminergic projection through induction of GDNF in a neurotoxin model of Parkinson's disease. , 2014, The Journal of nutritional biochemistry.
[22] A. Cuervo,et al. Autophagy gone awry in neurodegenerative diseases , 2010, Nature Neuroscience.
[23] A. Vezzani,et al. Brain Inflammation in Epilepsy: Experimental and Clinical Evidence , 2005, Epilepsia.
[24] Ji-Seon Seo,et al. Fluoxetine attenuates kainic acid-induced neuronal cell death in the mouse hippocampus , 2009, Brain Research.
[25] V. Perry,et al. The kinetics and morphological characteristics of the macrophage-microglial response to kainic acid-induced neuronal degeneration , 1991, Neuroscience.
[26] Lianfeng Zhang,et al. Long-term naringin consumption reverses a glucose uptake defect and improves cognitive deficits in a mouse model of Alzheimer's disease , 2012, Pharmacology Biochemistry and Behavior.