Aspirin Use Post-Acute Coronary Syndromes: Intolerance, Bleeding and Discontinuation
暂无分享,去创建一个
R. Califf | L. Newby | P. Armstrong | H. White | D. Kandzari | N. LaPointe | D. Moliterno | F. Verheugt | J. Kramer | M. Bhapkar | R. Califf | H. White | L. K. Newby | V. Manjushri | Bhapkar | J. David | Moliterno | A. FreekW. | Verheugt | Judith M. Kramer | Paul | W. Armstrong | Paul
[1] L. Bouter,et al. Mild renal insufficiency is associated with increased cardiovascular mortality: The Hoorn Study. , 2002, Kidney international.
[2] V. Hasselblad,et al. Seeking the optimal aspirin dose in acute coronary syndromes. , 2002, The American journal of cardiology.
[3] S. Borzak,et al. Benefits of aspirin and beta-blockade after myocardial infarction in patients with chronic kidney disease. , 2002, American heart journal.
[4] R. Califf,et al. Patient-reported frequency of taking aspirin in a population with coronary artery disease. , 2002, The American journal of cardiology.
[5] R. Califf,et al. Underuse of aspirin in a referral population with documented coronary artery disease. , 2002, The American journal of cardiology.
[6] Catherine Sudlow,et al. Collaborative meta-analysis of randomised trials of antiplatelet therapy for prevention of death, myocardial infarction, and stroke in high risk patients , 2002, BMJ : British Medical Journal.
[7] S. Hernández-Díaz,et al. Association between aspirin and upper gastrointestinal complications: systematic review of epidemiologic studies. , 2001, British journal of clinical pharmacology.
[8] S. Yusuf,et al. Effects of clopidogrel in addition to aspirin in patients with acute coronary syndromes without ST-segment elevation. , 2001, The New England journal of medicine.
[9] Second Symphony Investigators. Randomized Trial of Aspirin, Sibrafiban, or Both for Secondary Prevention After Acute Coronary Syndromes , 2001, Circulation.
[10] A. Gawlinski,et al. Improved treatment of coronary heart disease by implementation of a Cardiac Hospitalization Atherosclerosis Management Program (CHAMP). , 2001, The American journal of cardiology.
[11] P. R. R. Ecurrent,et al. EFFECTS OF CLOPIDOGREL IN ADDITION TO ASPIRIN IN PATIENTS WITH ACUTE CORONARY SYNDROMES WITHOUT ST-SEGMENT ELEVATION , 2001 .
[12] Y. Loke,et al. Risk of gastrointestinal haemorrhage with long term use of aspirin: meta-analysis , 2000, BMJ : British Medical Journal.
[13] R. Stafford,et al. Aspirin use is low among United States outpatients with coronary artery disease. , 2000, Circulation.
[14] R. Califf,et al. Comparison of sibrafiban with aspirin for prevention of cardiovascular events after acute coronary syndromes: a randomised trial , 2000, The Lancet.
[15] G. Remuzzi,et al. Uremic bleeding: closing the circle after 30 years of controversies? , 1999, Blood.
[16] L. Newby,et al. Long-term oral platelet glycoprotein IIb/IIIa receptor antagonism with sibrafiban after acute coronary syndromes: Study design of the Sibrafiban versus aspirin to Yield Maximum Protection from ischemic Heart events post-acute cOroNary sYndromes (SYMPHONY) trial , 1999 .
[17] W. Aronow. Underutilization of Aspirin in Older Patients with Prior Myocardial Infarction at the Time of Admission to a Nursing Home , 1998, Journal of the American Geriatrics Society.
[18] C. Bode,et al. Inhaled nitric oxide inhibits human platelet aggregation, P-selectin expression, and fibrinogen binding in vitro and in vivo. , 1998, Circulation.
[19] M. Dennis. A randomised, blinded, trial of clopidogrel versus aspirin in patients at risk of ischaemic events (CAPRIE). CAPRIE Steering Committee , 1996 .
[20] P. Metcalf,et al. Patterns of aspirin use in middle-aged adults: the Atherosclerosis Risk in Communities (ARIC) Study. , 1996, American heart journal.
[21] D. Clement. A randomised, blinded, trial of Clopidogrel versus aspirin in patients at risk of ischaemic events (CAPRIE) , 1996 .
[22] J. Stamler,et al. Effect of nitric oxide synthase inhibition on bleeding time in humans. , 1995, Journal of cardiovascular pharmacology.
[23] Walker,et al. Collaborative overview of randomised trials of antiplatelet therapy Prevention of death, myocardial infarction, and stroke by prolonged antiplatelet therapy in various categories of patients , 1994 .
[24] G. Hedenstierna,et al. Bleeding time prolongation and NO inhalation , 1993, The Lancet.
[25] C. Cierniewski,et al. The Decreased Circulating Platelet Mass and Its Relation to Bleeding Time in Chronic Renal Failure , 1991, Thrombosis and Haemostasis.
[26] N. Perico,et al. Role of endothelium-derived nitric oxide in the bleeding tendency of uremia. , 1990, The Journal of clinical investigation.
[27] S. Moncada,et al. An L-arginine/nitric oxide pathway present in human platelets regulates aggregation. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[28] J. Sixma,et al. High von Willebrand factor concentration compensates a relative adhesion defect in uremic blood. , 1990, Blood.
[29] S. Moncada,et al. ENDOGENOUS NITRIC OXIDE INHIBITS HUMAN PLATELET ADHESION TO VASCULAR ENDOTHELIUM , 1987, The Lancet.
[30] M. Bonati,et al. Aspirin prolongs bleeding time in uremia by a mechanism distinct from platelet cyclooxygenase inhibition. , 1987, The Journal of clinical investigation.
[31] C. Brown,et al. Evidence against a platelet cyclooxygenase defect in uraemic subjects on chronic haemodialysis , 1986, British journal of haematology.
[32] G. Levy. Pharmacokinetics of salicylate in man. , 1979, Drug metabolism reviews.
[33] G. Levy,et al. Salicylate accumulation kinetics in man. , 1972, The New England journal of medicine.
[34] G. Levy,et al. Limited capacity for salicyl phenolic glucuronide formation and its effect on the kinetics of salicylate elimination in man , 1972, Clinical pharmacology and therapeutics.