Gene expression profiling of human pancreatic islets undergoing a simulated process of instant blood-mediated inflammatory reaction.

[1]  Allan Vaag,et al.  Interleukin-1-receptor antagonist in type 2 diabetes mellitus. , 2007, The New England journal of medicine.

[2]  K. Ekdahl,et al.  Tissue factor produced by the endocrine cells of the islets of Langerhans is associated with a negative outcome of clinical islet transplantation. , 2005 .

[3]  P. Halban,et al.  Leptin modulates beta cell expression of IL-1 receptor antagonist and release of IL-1beta in human islets. , 2004, Proceedings of the National Academy of Sciences of the United States of America.

[4]  Dolca Thomas,et al.  The Abundant NK Cells in Human Secondary Lymphoid Tissues Require Activation to Express Killer Cell Ig-Like Receptors and Become Cytolytic1 , 2004, The Journal of Immunology.

[5]  K. Ekdahl,et al.  Production of tissue factor by pancreatic islet cells as a trigger of detrimental thrombotic reactions in clinical islet transplantation , 2002, The Lancet.

[6]  J F Elliott,et al.  Clinical outcomes and insulin secretion after islet transplantation with the Edmonton protocol. , 2001, Diabetes.

[7]  G. Elgue,et al.  Incompatibility between human blood and isolated islets of Langerhans: a finding with implications for clinical intraportal islet transplantation? , 1999, Diabetes.

[8]  David A. Williams,et al.  Molecular cloning of a cDNA encoding interleukin 11, a stromal cell-derived lymphopoietic and hematopoietic cytokine. , 1990, Proceedings of the National Academy of Sciences of the United States of America.

[9]  Camillo Ricordi,et al.  Automated Method for Isolation of Human Pancreatic Islets , 1988, Diabetes.

[10]  P. Allavena,et al.  Human pancreatic islets produce and secrete MCP-1/CCL2: relevance in human islet transplantation. , 2002, Diabetes.