Hypoxia shapes the immune landscape in lung injury and promotes the persistence of inflammation
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J. Baillie | S. Forbes | P. Ratcliffe | J. Schwarze | T. Ly | C. Pugh | David Lewis | S. Walmsley | Christos Spanos | R. Dickinson | C. Pridans | L. Campana | C. Bain | S. Jenkins | M. Whyte | D. Dockrell | Leila Reyes | S. Clohisey | N. Hirani | Kamil R. Kranc | Manuel A. Sanchez-Garcia | P. Coelho | A. Mirchandani | Tyler Morrison | Emily R. Watts | F. Murphy | Ailiang Zhang | K. Musgrave | A. Rostron | P. Sadiku | A. J. Simpson | N. Parkinson | Hannah Lawson | D. Griffith | V. Kelly | J. Scott | P. S. Lewis | Simone Arienti | George Cooper | Sarah K Clark | Liam Delaney | Isla Harper | Pranvera Sadiku | Clare Pridans
[1] A. von Kriegsheim,et al. Hypoxia drives murine neutrophil protein scavenging to maintain central carbon metabolism. , 2021, The Journal of clinical investigation.
[2] A. Malik,et al. The angiocrine Rspondin3 instructs interstitial macrophage transition via metabolic–epigenetic reprogramming and resolves inflammatory injury , 2020, Nature Immunology.
[3] Eric Song,et al. Longitudinal analyses reveal immunological misfiring in severe COVID-19 , 2020, Nature.
[4] Nicolas Carlier,et al. Impaired type I interferon activity and inflammatory responses in severe COVID-19 patients , 2020, Science.
[5] T. Ly,et al. Low Cell Number Proteomic Analysis Using In-Cell Protease Digests Reveals a Robust Signature for Cell Cycle State Classification , 2020, bioRxiv.
[6] T. Otto,et al. Distinct synovial tissue macrophage subsets regulate inflammation and remission in rheumatoid arthritis , 2020, Nature Medicine.
[7] S. Dhingra,et al. Hypoxia-induced shift in the phenotype of proteasome from 26S toward immunoproteasome triggers loss of immunoprivilege of mesenchymal stem cells , 2020, Cell Death & Disease.
[8] I. Amit,et al. Single-cell landscape of bronchoalveolar immune cells in patients with COVID-19 , 2020, Nature Medicine.
[9] F. Ginhoux,et al. Two distinct interstitial macrophage populations coexist across tissues in specific subtissular niches , 2019, Science.
[10] E. Solito,et al. Annexin A1: Uncovering the Many Talents of an Old Protein , 2018, International journal of molecular sciences.
[11] M. Chamaillard,et al. Type I interferons drive inflammasome-independent emergency monocytopoiesis during endotoxemia , 2017, Scientific Reports.
[12] David E. Muench,et al. Granulocyte‐Monocyte Progenitors and Monocyte‐Dendritic Cell Progenitors Independently Produce Functionally Distinct Monocytes , 2017, Immunity.
[13] Amit A. Patel,et al. The fate and lifespan of human monocyte subsets in steady state and systemic inflammation , 2017, The Journal of experimental medicine.
[14] Ognjen Gajic,et al. An Official American Thoracic Society/European Society of Intensive Care Medicine/Society of Critical Care Medicine Clinical Practice Guideline: Mechanical Ventilation in Adult Patients with Acute Respiratory Distress Syndrome , 2017, American journal of respiratory and critical care medicine.
[15] F. Ginhoux,et al. Exposure to Bacterial CpG DNA Protects from Airway Allergic Inflammation by Expanding Regulatory Lung Interstitial Macrophages , 2017, Immunity.
[16] Amy R. Peck,et al. Inactivation of Interferon Receptor Promotes the Establishment of Immune Privileged Tumor Microenvironment. , 2017, Cancer cell.
[17] P. Carmeliet,et al. Hypoxia determines survival outcomes of bacterial infection through HIF-1α–dependent reprogramming of leukocyte metabolism , 2017, Science Immunology.
[18] A. Joshi,et al. Long-lived self-renewing bone marrow-derived macrophages displace embryo-derived cells to inhabit adult serous cavities , 2016, Nature Communications.
[19] Anders Larsson,et al. Epidemiology, Patterns of Care, and Mortality for Patients With Acute Respiratory Distress Syndrome in Intensive Care Units in 50 Countries. , 2016, JAMA.
[20] Wei-Kung Chen,et al. CREB Negatively Regulates IGF2R Gene Expression and Downstream Pathways to Inhibit Hypoxia-Induced H9c2 Cardiomyoblast Cell Death , 2015, International journal of molecular sciences.
[21] Lee B. Smith,et al. Characterisation of a Novel Fc Conjugate of Macrophage Colony-stimulating Factor , 2014, Molecular therapy : the journal of the American Society of Gene Therapy.
[22] L. King,et al. Diverse macrophage populations mediate acute lung inflammation and resolution. , 2014, American journal of physiology. Lung cellular and molecular physiology.
[23] W. Seeger,et al. Inhaled granulocyte/macrophage colony-stimulating factor as treatment of pneumonia-associated acute respiratory distress syndrome. , 2014, American journal of respiratory and critical care medicine.
[24] Bernard Malissen,et al. Alveolar macrophages develop from fetal monocytes that differentiate into long-lived cells in the first week of life via GM-CSF , 2013, The Journal of experimental medicine.
[25] S. Morrison,et al. SLAM family markers resolve functionally distinct subpopulations of hematopoietic stem cells and multipotent progenitors. , 2013, Cell stem cell.
[26] Y. Belkaid,et al. Inflammatory monocytes regulate pathologic responses to commensals during acute gastrointestinal infection , 2013, Nature Medicine.
[27] F. Ginhoux,et al. Tissue-resident macrophages self-maintain locally throughout adult life with minimal contribution from circulating monocytes. , 2013, Immunity.
[28] G. Melillo,et al. Autocrine production of IL-11 mediates tumorigenicity in hypoxic cancer cells. , 2013, The Journal of clinical investigation.
[29] B. Malissen,et al. Resident and pro-inflammatory macrophages in the colon represent alternative context-dependent fates of the same Ly6Chi monocyte precursors , 2012, Mucosal Immunology.
[30] Teppei Shimamura,et al. Dynamic Change of Chromatin Conformation in Response to Hypoxia Enhances the Expression of GLUT3 (SLC2A3) by Cooperative Interaction of Hypoxia-Inducible Factor 1 and KDM3A , 2012, Molecular and Cellular Biology.
[31] R. Hyzy,et al. A randomized trial of recombinant human granulocyte-macrophage colony stimulating factor for patients with acute lung injury* , 2012, Critical care medicine.
[32] L. Boon,et al. IFNγ induces monopoiesis and inhibits neutrophil development during inflammation. , 2011, Blood.
[33] S. Bhattacharyya,et al. Lipopolysaccharide-induced activation of NF-κB non-canonical pathway requires BCL10 serine 138 and NIK phosphorylations. , 2010, Experimental cell research.
[34] L. de Leval,et al. Lung interstitial macrophages alter dendritic cell functions to prevent airway allergy in mice. , 2009, The Journal of clinical investigation.
[35] A. Manivannan,et al. Critical but divergent roles for CD62L and CD44 in directing blood monocyte trafficking in vivo during inflammation , 2008, Blood.
[36] Mimi C Sammarco,et al. Ferritin L and H Subunits Are Differentially Regulated on a Post-transcriptional Level* , 2008, Journal of Biological Chemistry.
[37] Xiaojing Ma,et al. Interleukin-10 expression in macrophages during phagocytosis of apoptotic cells is mediated by homeodomain proteins Pbx1 and Prep-1. , 2007, Immunity.
[38] David Bryder,et al. Elucidation of the phenotypic, functional, and molecular topography of a myeloerythroid progenitor cell hierarchy. , 2007, Cell stem cell.
[39] M. Mann,et al. Higher-energy C-trap dissociation for peptide modification analysis , 2007, Nature Methods.
[40] M. Mann,et al. Protocol for micro-purification, enrichment, pre-fractionation and storage of peptides for proteomics using StageTips , 2007, Nature Protocols.
[41] Luigi Varesio,et al. Hypoxia Modifies the Transcriptome of Primary Human Monocytes: Modulation of Novel Immune-Related Genes and Identification Of CC-Chemokine Ligand 20 as a New Hypoxia-Inducible Gene1 , 2006, The Journal of Immunology.
[42] L. Nelin,et al. The Role of Mitogen-activated Protein Kinase Phosphatase-1 in the Response of Alveolar Macrophages to Lipopolysaccharide ATTENUATION OF PROINFLAMMATORY CYTOKINE BIOSYNTHESIS VIA FEEDBACK CONTROL OF p38* , 2005 .
[43] J. Lötvall,et al. A role of GM-CSF in the accumulation of neutrophils in the airways caused by IL-17 and TNF‐α , 2003, European Respiratory Journal.
[44] W. Muller,et al. CD99 plays a major role in the migration of monocytes through endothelial junctions , 2002, Nature Immunology.
[45] J. Pugin,et al. Cytokine balance in the lungs of patients with acute respiratory distress syndrome. , 2001, American journal of respiratory and critical care medicine.
[46] F. Stentz,et al. Persistent elevation of inflammatory cytokines predicts a poor outcome in ARDS. Plasma IL-1 beta and IL-6 levels are consistent and efficient predictors of outcome over time. , 1995, Chest.
[47] K. Steinberg,et al. Evolution of bronchoalveolar cell populations in the adult respiratory distress syndrome. , 1994, American journal of respiratory and critical care medicine.
[48] David A. Williams,et al. Molecular cloning of a cDNA encoding interleukin 11, a stromal cell-derived lymphopoietic and hematopoietic cytokine. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[49] S. Hsu,et al. Self‐Renewal of Pulmonary Alveolar Macrophages: Evidence From Radiation Chimera Studies , 1987, Journal of leukocyte biology.
[50] C. W. Harris,et al. Effects of altitude acclimatization on blood composition of women. , 1969, Journal of applied physiology.