Influence of genetic variations in IL1B on brain region volumes in bipolar patients and controls

[1]  Sarah L Bergen,et al.  Longitudinal Cortical Thickness Changes in Bipolar Disorder and the Relationship to Genetic Risk, Mania, and Lithium Use , 2020, Biological Psychiatry.

[2]  J. Hsu,et al.  Inflammation associated with volume reduction in the gray matter and hippocampus of older patients with bipolar disorder. , 2019, Journal of affective disorders.

[3]  C. Pantelis,et al.  An Interleukin-1 beta (IL1B) haplotype linked with psychosis transition is associated with IL1B gene expression and brain structure , 2019, Schizophrenia Research.

[4]  K. Simonyan.  Recent advances in understanding the role of the basal ganglia , 2019, F1000Research.

[5]  Haihua Qian,et al.  Two variants of Interleukin-1B gene are associated with the decreased risk, clinical features, and better overall survival of colorectal cancer: a two-center case-control study , 2018, Aging.

[6]  P. Petrovic,et al.  Bipolar disorder type I and II show distinct relationships between cortical thickness and executive function , 2018, Acta psychiatrica Scandinavica.

[7]  M. Maes,et al.  The Compensatory Immune-Regulatory Reflex System (CIRS) in Depression and Bipolar Disorder , 2018, Molecular Neurobiology.

[8]  C. Hultman,et al.  Prevalences of autoimmune diseases in schizophrenia, bipolar I and II disorder, and controls , 2017, Psychiatry Research.

[9]  A. Carvalho,et al.  Cognitive Impairment in Bipolar Disorder: Treatment and Prevention Strategies , 2017, The international journal of neuropsychopharmacology.

[10]  Erick Jorge Canales-Rodríguez,et al.  Cortical abnormalities in bipolar disorder: an MRI analysis of 6503 individuals from the ENIGMA Bipolar Disorder Working Group , 2017, Molecular Psychiatry.

[11]  M. Corredor,et al.  A Review of Polymorphisms in Genes Involved in the Development of Gastric Cancer , 2016 .

[12]  E. Olsson,et al.  Cognitive reserve lessens the burden of white matter lesions on executive functions in bipolar disorder , 2016, Psychological Medicine.

[13]  P. Petrovic,et al.  Cortical thickness, volume and surface area in patients with bipolar disorder types I and II. , 2016, Journal of psychiatry & neuroscience : JPN.

[14]  Amit Anand,et al.  Differential Resting-State Functional Connectivity of Striatal Subregions in Bipolar Depression and Hypomania , 2016, Brain Connect..

[15]  Xiaoping P. Hu,et al.  Inflammation is associated with decreased functional connectivity within corticostriatal reward circuitry in depression , 2015, Molecular Psychiatry.

[16]  K. Blennow,et al.  CSF neuroinflammatory biomarkers in bipolar disorder are associated with cognitive impairment , 2015, European Neuropsychopharmacology.

[17]  D. Woods,et al.  Hemispheric asymmetries in cortical and subcortical anatomy , 2015, Laterality.

[18]  S. Hansen,et al.  Cognitive Functioning in Clinically Stable Patients with Bipolar Disorder I and II , 2015, PloS one.

[19]  M. Pawlak,et al.  Basal Ganglia Volumes: MR-Derived Reference Ranges and Lateralization Indices for Children and Young Adults , 2014, The neuroradiology journal.

[20]  C. Ekman,et al.  Vertex-based morphometry in euthymic bipolar disorder implicates striatal regions involved in psychomotor function , 2014, Psychiatry Research: Neuroimaging.

[21]  C. Davis,et al.  Obesity Elicits Interleukin 1-Mediated Deficits in Hippocampal Synaptic Plasticity , 2014, The Journal of Neuroscience.

[22]  T. Robbins,et al.  Neuroscience and Biobehavioral Reviews Review from the Ventral to the Dorsal Striatum: Devolving Views of Their Roles in Drug Addiction , 2022 .

[23]  D. Walker,et al.  Cytokines and the neurodevelopmental basis of mental illness , 2013, Front. Neurosci..

[24]  Teresa A. Victor,et al.  Inflammation and neurological disease-related genes are differentially expressed in depressed patients with mood disorders and correlate with morphometric and functional imaging abnormalities , 2013, Brain, Behavior, and Immunity.

[25]  E. Brietzke,et al.  Cytokine Alterations in Bipolar Disorder: A Meta-Analysis of 30 Studies , 2013, Biological Psychiatry.

[26]  P. Calabresi,et al.  Effects of central and peripheral inflammation on hippocampal synaptic plasticity , 2013, Neurobiology of Disease.

[27]  B. Czéh,et al.  Antidepressants act directly on astrocytes: Evidences and functional consequences , 2013, European Neuropsychopharmacology.

[28]  R. Nitsch,et al.  Absence of IL-1β positively affects neurological outcome, lesion development and axonal plasticity after spinal cord injury , 2013, Journal of Neuroinflammation.

[29]  C. Ekman,et al.  Neurocognitive function in bipolar disorder: a comparison between bipolar I and II disorder and matched controls , 2012, BMC Psychiatry.

[30]  L. Fañanás,et al.  Effect of the Interleukin-1β Gene on Dorsolateral Prefrontal Cortex Function in Schizophrenia: A Genetic Neuroimaging Study , 2012, Biological Psychiatry.

[31]  T. Suslow,et al.  Tumor Necrosis Factor Gene Variation Predicts Hippocampus Volume in Healthy Individuals , 2012, Biological Psychiatry.

[32]  Walter Heindel,et al.  Interleukin-6 gene (IL-6): a possible role in brain morphology in the healthy adult brain , 2012, Journal of Neuroinflammation.

[33]  J. Kowalski,et al.  Interleukin-1beta Promoter (−31T/C and −511C/T) Polymorphisms in Major Recurrent Depression , 2011, Journal of Molecular Neuroscience.

[34]  Jin Fan,et al.  Common and distinct networks underlying reward valence and processing stages: A meta-analysis of functional neuroimaging studies , 2011, Neuroscience & Biobehavioral Reviews.

[35]  M. Landén,et al.  Elevation of cerebrospinal fluid interleukin-1ß in bipolar disorder. , 2011, Journal of psychiatry & neuroscience : JPN.

[36]  J. Ordovás,et al.  Interleukin 1B variant -1473G/C (rs1143623) influences triglyceride and interleukin 6 metabolism. , 2011, The Journal of clinical endocrinology and metabolism.

[37]  M. Berk,et al.  eview athways underlying neuroprogression in bipolar disorder : Focus on nflammation , oxidative stress and neurotrophic factors , 2010 .

[38]  M. Ingvar,et al.  Manic episodes are associated with grey matter volume reduction — a voxel‐based morphometry brain analysis , 2010, Acta psychiatrica Scandinavica.

[39]  Lin He,et al.  Convergent Evidence Shows a Positive Association of Interleukin-1 Gene Complex Locus with Susceptibility to Schizophrenia in the Caucasian population , 2010, Schizophrenia Research.

[40]  L. Boulanger,et al.  Immune Proteins in Brain Development and Synaptic Plasticity , 2009, Neuron.

[41]  S. Tsai,et al.  Interleukin-1 Beta −511C/T Genetic Polymorphism is Associated with Age of Onset of Geriatric Depression , 2009, NeuroMolecular Medicine.

[42]  R. McIntyre,et al.  Inflammation and the phenomenology, pathophysiology, comorbidity, and treatment of bipolar disorder: a systematic review of the literature. , 2009, The Journal of clinical psychiatry.

[43]  L. Fañanás,et al.  Gray matter deficits in bipolar disorder are associated with genetic variability at interleukin‐1 beta gene (2q13) , 2008, Genes, brain, and behavior.

[44]  A. Galor,et al.  Transient band keratopathy associated with ocular inflammation and systemic hypercalcemia , 2008, Clinical ophthalmology.

[45]  Matthias J. Müller,et al.  Association analysis between variants of the interleukin-1β and the interleukin-1 receptor antagonist gene and antidepressant treatment response in major depression , 2008, Neuropsychiatric disease and treatment.

[46]  N. Rothwell,et al.  Interleukin-1 and inflammatory neurodegeneration. , 2007, Biochemical Society transactions.

[47]  C. Cannings,et al.  Single nucleotide polymorphisms in the human interleukin-1B gene affect transcription according to haplotype context. , 2006, Human molecular genetics.

[48]  P. Renshaw,et al.  Basal ganglia shape alterations in bipolar disorder. , 2006, The American journal of psychiatry.

[49]  Olga V. Demler,et al.  Lifetime prevalence and age-of-onset distributions of DSM-IV disorders in the National Comorbidity Survey Replication. , 2005, Archives of general psychiatry.

[50]  Mark Daly,et al.  Haploview: analysis and visualization of LD and haplotype maps , 2005, Bioinform..

[51]  B. Gutiérrez,et al.  Interleukin-1 cluster is associated with genetic risk for schizophrenia and bipolar disorder , 2004, Journal of Medical Genetics.

[52]  S. Tsai,et al.  Association Study of the Interleukin-1beta (C-511T) Genetic Polymorphism with Major Depressive Disorder, Associated Symptomatology, and Antidepressant Response , 2003, Neuropsychopharmacology.

[53]  U Hegerl,et al.  Association of an interleukin-1beta genetic polymorphism with altered brain structure in patients with schizophrenia. , 2001, The American journal of psychiatry.

[54]  M. Carrington,et al.  Interleukin-1 polymorphisms associated with increased risk of gastric cancer , 2000, Nature.

[55]  N. Herrmann,et al.  Global grey matter volume in adult bipolar patients with and without lithium treatment: A meta-analysis. , 2018, Journal of affective disorders.

[56]  D. Steffens,et al.  Putamen Volume Differences Among Older Adults: Depression Status, Melancholia, and Age , 2018, Journal of geriatric psychiatry and neurology.

[57]  J. Price,et al.  Neurocircuitry of Mood Disorders , 2010, Neuropsychopharmacology.

[58]  Patricia A. Berglund,et al.  Lifetime prevalence and age of onset distributions of mental disorders in the World Mental Health Survey Initiative. , 2008 .