Differential expression of genes related to levels of mucosal cell proliferation among multiple rat strains by using oligonucleotide microarrays

[1]  T. Tsukamoto,et al.  Global expression analysis of N-methyl-N'-nitro-N-nitrosoguanidine-induced rat stomach carcinomas using oligonucleotide microarrays. , 2003, Carcinogenesis.

[2]  Manel Esteller,et al.  Hypermethylation-associated Inactivation of the Cellular Retinol-Binding-Protein 1 Gene in Human Cancer. , 2002, Cancer research.

[3]  H. Yokozaki,et al.  Distinct promoter hypermethylation of p16INK4a, CDH1, and RAR‐beta in intestinal, diffuse–adherent, and diffuse–scattered type gastric carcinomas , 2002, The Journal of pathology.

[4]  Peng Liu,et al.  Selective Cooperation between Fatty Acid Binding Proteins and Peroxisome Proliferator-Activated Receptors in Regulating Transcription , 2002, Molecular and Cellular Biology.

[5]  K. Wakazono,et al.  Profiling and selection of genes differentially expressed in the pylorus of rat strains with different proliferative responses and stomach cancer susceptibility. , 2002, Carcinogenesis.

[6]  R. Lotan,et al.  Inactivation of retinoic acid receptor beta by promoter CpG hypermethylation in gastric cancer. , 2001, Differentiation; research in biological diversity.

[7]  D. Levinthal,et al.  Distinct Roles for Cellular Retinoic Acid-binding Proteins I and II in Regulating Signaling by Retinoic Acid* , 1999, The Journal of Biological Chemistry.

[8]  H. Iishi,et al.  Attenuation by all-trans-retinoic acid of sodium chloride-enhanced gastric carcinogenesis induced by N-methyl-N′-nitro-N-nitrosoguanidine in Wistar rats , 1999, British Journal of Cancer.

[9]  E. Frei,et al.  1-C-glucuronidation of N-nitrosodiethylamine and N-nitrosomethyl-n-pentylamine in vivo and in primary hepatocytes from rats pretreated with inducers. , 1992, Carcinogenesis.

[10]  L. Ellwein,et al.  Cell proliferation in carcinogenesis. , 1990, Science.

[11]  B. Ames,et al.  Too many rodent carcinogens: mitogenesis increases mutagenesis. , 1990, Science.

[12]  N. Bass,et al.  Functions of fatty acid binding proteins , 1990, Experientia.

[13]  P. Kleihues,et al.  Differential proliferative response of gastric mucosa during carcinogenesis induced by N-methyl-N'-nitro-N-nitrosoguanidine in susceptible ACI rats, resistant Buffalo rats, and their hybrid F1 cross. , 1988, Cancer research.

[14]  M. Tatematsu,et al.  Coefficient induction of pepsinogen 1-decreased pyloric glands and gastric cancers in five different strains of rats treated with N-methyl-N'-nitro-N-nitrosoguanidine. , 1988, Carcinogenesis.

[15]  N. Bass.  Function and regulation of hepatic and intestinal fatty acid binding proteins. , 1985, Chemistry and physics of lipids.

[16]  H. Ohgaki,et al.  Genetic control of susceptibility of rats to gastric carcinoma. , 1983, Cancer research.

[17]  D. Crowe,et al.  Transcriptional regulation of retinoic acid responsive genes by cellular retinoic acid binding protein-II modulates RA mediated tumor cell proliferation and invasion. , 1998, Anticancer research.

[18]  T. Sugimura.  Experimental Gastric Cancers , 1993 .

[19]  N. Bass,et al.  Fatty acid binding proteins: recent concepts of regulation and function. , 1992, Progress in clinical and biological research.

[20]  P. Kleihues,et al.  Experimental gastric cancer. , 1991, The Italian journal of gastroenterology.

[21]  N. Bass.  The cellular fatty acid binding proteins: aspects of structure, regulation, and function. , 1988, International review of cytology.

[22]  H. DeLuca,et al.  Biosynthesis of retinoyl-β-glucuronide, a biologically active metabolite of all-trans-retinoic acid , 1986 .

[23]  D. Meranze,et al.  Host resistance to gastric adenocarcinomatosis in three strains of rats ingesting N-methyl-N'-nitro-N-nitrosoguanidine. , 1973, Oncology.