Impaired wound healing.
暂无分享,去创建一个
Tarynn M Witten | Kevin R Ward | K. Ward | T. Witten | D. Bonchev | N. Menke | R. Diegelmann | Robert F Diegelmann | Nathan B Menke | Danail G Bonchev
[1] Ali A. Minai,et al. Unifying Themes in Complex Systems , 2006 .
[2] M. Kobayashi,et al. Marjolin's Ulcer In Patients With Spinal Cord Injury , 2002, The journal of spinal cord medicine.
[3] I. K. Cohen,et al. Wound fluids from human pressure ulcers contain elevated matrix metalloproteinase levels and activity compared to surgical wound fluids. , 1996, The Journal of investigative dermatology.
[4] L. Golub,et al. Pharmacologic suppression of experimental abdominal aortic aneurysms: acomparison of doxycycline and four chemically modified tetracyclines. , 1998, Journal of vascular surgery.
[5] D. M. Cooper,et al. Determination of Endogenous Cytokines in Chronic Wounds , 1994, Annals of surgery.
[6] G. Schultz,et al. Proteases and the diabetic foot syndrome: mechanisms and therapeutic implications. , 2005, Diabetes care.
[7] D. Bonchev,et al. Complexity in chemistry, biology, and ecology , 2005 .
[8] F. Grinnell,et al. Wound fluid from chronic leg ulcers contains elevated levels of metalloproteinases MMP-2 and MMP-9. , 1993, The Journal of investigative dermatology.
[9] I. K. Cohen,et al. Physiology of the chronic wound. , 1998, Clinics in plastic surgery.
[10] Y. Konttinen,et al. Tetracycline inhibition identifies the cellular sources of collagenase in gingival crevicular fluid in different forms of periodontal diseases. , 1992, Drugs under experimental and clinical research.
[11] Simona Storti,et al. SERCA2a, Phospholamban, Sarcolipin, and Ryanodine Receptors Gene Expression in Children with Congenital Heart Defects , 2007, Molecular medicine.
[12] Danail Bonchev,et al. Complexity of Protein-Protein Interaction Networks, Complexes, and Pathways , 2003 .
[13] Michael F. Shlesinger,et al. Chaotic and Fractal Dynamics: An Introduction for Applied Scientists and Engineers , 1993 .
[14] L. Golub,et al. Tetracyclines suppress matrix metalloproteinase activity in adjuvant arthritis and in combination with flurbiprofen, ameliorate bone damage. , 1992, The Journal of rheumatology.
[15] Henrik Jeldtoft Jensen,et al. Self-Organized Criticality: Emergent Complex Behavior in Physical and Biological Systems , 1998 .
[16] D. Steed. Wound-healing trajectories. , 2003, The Surgical clinics of North America.
[17] I. K. Cohen,et al. Dynamics of the matrix metalloproteinases MMP‐1 and MMP‐8 in acute open human dermal wounds , 1998, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.
[18] V. Falanga. Growth Factors and Chronic Wounds: The Need to Understand the Microenvironment , 1992, The Journal of dermatology.
[19] H. Brem,et al. Healing of diabetic foot ulcers and pressure ulcers with human skin equivalent: a new paradigm in wound healing. , 2000, Archives of surgery.
[20] H. Lehnert,et al. Expression of matrix-metalloproteinases and their inhibitors in the wounds of diabetic and non-diabetic patients , 2002, Diabetologia.
[21] I. K. Cohen,et al. MMP-8 is the predominant collagenase in healing wounds and nonhealing ulcers. , 1999, The Journal of surgical research.
[22] D. Steed,et al. Wound healing trajectories as predictors of effectiveness of therapeutic agents. , 2000, Archives of surgery.
[23] B. Baxter,et al. MMP Inhibition in Abdominal Aortic Aneurysms: Rationale for a Prospective Randomized Clinical Trial , 1999, Annals of the New York Academy of Sciences.
[24] S. Weiss. Tissue destruction by neutrophils. , 1989, The New England journal of medicine.
[25] A. Vaheri,et al. Proteolytic activity in leg ulcer exudate , 1993, Experimental dermatology.
[26] M. Longaker,et al. Tissue inhibitor of metalloproteinases-1 is decreased and activated gelatinases are increased in chronic wounds. , 1995, The Journal of investigative dermatology.
[27] R. Diegelmann. Excessive neutrophils characterize chronic pressure ulcers , 2003, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.
[28] G. Schultz,et al. Growth factors and wound healing: Part II. Role in normal and chronic wound healing. , 1993, American journal of surgery.
[29] R. Diegelmann,et al. Ability of chronic wound fluids to degrade peptide growth factors is associated with increased levels of elastase activity and diminished levels of proteinase inhibitors , 1997, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.
[30] Stephen Wiggins. Global Bifurcations and Chaos: Analytical Methods , 1988 .
[31] L. Teot,et al. The diagnosis and treatment of carcinomas occurring at the sites of chronic pressure ulcers. , 2004, Journal of wound care.
[32] G. Cherbit,et al. Fractals: Non-Integral Dimensions and Applications , 1991 .
[33] D Bergqvist,et al. Chronic leg ulcers: an underestimated problem in primary health care among elderly patients. , 1991, Journal of epidemiology and community health.
[34] C. N. Rao,et al. Alpha 1-antitrypsin is degraded and non-functional in chronic wounds but intact and functional in acute wounds: the inhibitor protects fibronectin from degradation by chronic wound fluid enzymes. , 1995, The Journal of investigative dermatology.
[35] G. Schultz,et al. Interactions of cytokines, growth factors, and proteases in acute and chronic wounds , 1996, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.
[36] Matthew Witten. A note on the structure of system state spaces and its implications on the existence of non-repeatable experiments , 1980 .
[37] Matthew Witten,et al. A return to time, cells, systems, and aging: III. Gompertzian models of biological aging and some possible roles for critical elements , 1985, Mechanisms of Ageing and Development.
[38] S. Del Prato,et al. Semiquantitative analysis of the histopathological features of the neuropathic foot ulcer: effects of pressure relief. , 2003, Diabetes care.
[39] D. M. Cooper,et al. Definitions and guidelines for assessment of wounds and evaluation of healing. , 1994, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.
[40] F. Grinnell,et al. Fibronectin Degradation in Chronic Wounds Depends on the Relative Levels of Elastase, α1-Proteinase Inhibitor, and α2-Macroglobulin , 1996 .
[41] R. Landauer,et al. Thinking in Complexity: The Complex Dynamics of Matter, Mind, and Mankind , 1995 .