Expression of c-Fos and c-Myc and deposition of beta-APP in neurons in the adult rat brain as a result of exposure to short-lasting impulse noise.
暂无分享,去创建一个
Jingshan Shi | Feng Bao | A. Hamberger | H. Hansson | K. Haglid | A. Säljö | Annette Säljö | Anders Hamberger | Hans-Arne Hansson | Kenneth G Haglid | Jingshan Shi | F. Bao
[1] N. Elsayed,et al. Toxicology of blast overpressure. , 1997, Toxicology.
[2] F. Holsboer,et al. Activation of c-Fos contributes to amyloid β-peptide-induced neurotoxicity , 1996, Brain Research.
[3] G. Žunić,et al. Indirect neurotrauma caused by pulmonary blast injury : Development and prognosis , 1997 .
[4] L. Rubin,et al. A c-jun dominant negative mutant protects sympathetic neurons against programmed cell death , 1995, Neuron.
[5] D. Graham,et al. Is There a Genetic Basis for the Deposition of β-Amyloid After Fatal Head Injury? , 1999, Cellular and Molecular Neurobiology.
[6] T. Yamaki,et al. Experimental brain injury induces expression of amyloid precursor protein, which may be related to neuronal loss in the hippocampus. , 1998, Journal of neurotrauma.
[7] J. Povlishock,et al. A new model for rapid stretch-induced injury of cells in culture: characterization of the model using astrocytes. , 1995, Journal of neurotrauma.
[8] L. W. Turner,et al. Quantities and Units , 1991 .
[9] K. Yoshikawa,et al. Degeneration in vitro of post-mitotic neurons overexpressing the Alzheimer amyloid protein precursor , 1992, Nature.
[10] J Q Trojanowski,et al. Accumulation of amyloid beta and tau and the formation of neurofilament inclusions following diffuse brain injury in the pig. , 1999, Journal of neuropathology and experimental neurology.
[11] F. Diaz,et al. Patterns of immediate early gene mRNA expression following rodent and human traumatic brain injury. , 1999, Neurological research.
[12] W J Mcfeely,et al. Otologic Injuries Caused by Airbag Deployment , 1999, Otolaryngology Head & Neck Surgery.
[13] P. Gass,et al. Neuronal expression of AP-1 proteins in excitotoxic-neurodegenerative disorders and following nerve fiber lesions , 1995, Progress in Neurobiology.
[14] E. Hölttä,et al. Caspases and mitochondria in c-Myc-induced apoptosis: identification of ATM as a new target of caspases , 2000, Oncogene.
[15] J. Wolf,et al. Evolution of neurofilament subtype accumulation in axons following diffuse brain injury in the pig. , 1999, Journal of neuropathology and experimental neurology.
[16] P. Gluckman,et al. Do c‐Jun, c‐Fos, and amyloid precursor protein play a role in neuronal death or survival? , 1998, Journal of neuroscience research.
[17] P M Fuller,et al. Airbag-induced eye injuries: a report of 25 cases. , 1996, The Journal of trauma.
[18] D. Selkoe,et al. Targeting of cell-surface β-amyloid precursor protein to lysosomes: alternative processing into amyloid-bearing fragments , 1992, Nature.
[19] G. Rosenberger,et al. The MYC dualism in growth and death. , 1999, Mutation research.
[20] P. Mehta,et al. Traumatic Brain Injury Elevates the Alzheimer's Amyloid Peptide Aβ42 in Human CSF: A Possible Role for Nerve Cell Injury , 2000, Annals of the New York Academy of Sciences.
[21] I. Verma,et al. Regulation of proto-oncogene fos: a paradigm for early response genes. , 1988, Cold Spring Harbor symposia on quantitative biology.
[22] T. D. Brown,et al. Airbag associated fatal head injury: case report and review of the literature on airbag injuries , 2000, Journal of accident & emergency medicine.
[23] T. Curran,et al. Fos: an immediate-early transcription factor in neurons. , 1995, Journal of neurobiology.
[24] J. Charron,et al. N-Myc shares cellular functions wiht c-Myc. , 2000, DNA and cell biology.
[25] S. Deeb,et al. Overexpression of amyloid precursor protein alters its normal processing and is associated with neurotoxicity. , 1992, Biochemical and biophysical research communications.
[26] A. Hamberger,et al. Exposure to short-lasting impulse noise causes microglial and astroglial cell activation in the adult rat brain. , 2001, Pathophysiology : the official journal of the International Society for Pathophysiology.
[27] J. Pickard,et al. Expression of the immediate early genes c-Fos and c-Jun after head injury in man , 2000, Neurological research.
[28] K. Horsburgh,et al. The role of apolipoprotein E in Alzheimer’s disease, acute brain injury and cerebrovascular disease: evidence of common mechanisms and utility of animal models , 2000, Neurobiology of Aging.
[29] S. Estus,et al. Aggregated Amyloid-β Protein Induces Cortical Neuronal Apoptosis and Concomitant “Apoptotic” Pattern of Gene Induction , 1997, The Journal of Neuroscience.
[30] M. Dragunow,et al. Induction of immediate-early genes and the control of neurotransmitter-regulated gene expression within the nervous system. , 1995, Pharmacological reviews.
[31] J. Finnie,et al. Upregulation of amyloid precursor protein and its mRNA in an experimental model of paediatric head injury , 2000, Journal of Clinical Neuroscience.
[32] H. Lodish. Molecular Cell Biology , 1986 .
[33] R. Mrak,et al. Neuritic plaque evolution in Alzheimer’s disease is accompanied by transition of activated microglia from primed to enlarged to phagocytic forms , 1997, Acta Neuropathologica.
[34] D. Ignjatovic,et al. Blast injury from explosive munitions. , 1999, The Journal of trauma.
[35] C. Masters,et al. The amyloid protein precursor of Alzheimer's disease is a mediator of the effects of nerve growth factor on neurite outgrowth , 1992, Neuron.
[36] Kou-Juey Wu,et al. Coordinated regulation of iron-controlling genes, H-ferritin and IRP2, by c-MYC. , 1999, Science.
[37] Feng Bao,et al. Exposure to short-lasting impulse noise causes neuronal c-Jun expression and induction of apoptosis in the adult rat brain. , 2002, Journal of neurotrauma.
[38] H. Hansson,et al. Blast exposure causes redistribution of phosphorylated neurofilament subunits in neurons of the adult rat brain. , 2000, Journal of neurotrauma.
[39] W H Slattery,et al. Automobile Airbag Impulse Noise: Otologic Symptoms in Six Patients , 1998, Otolaryngology--head and neck surgery : official journal of American Academy of Otolaryngology-Head and Neck Surgery.
[40] M. Mattson,et al. Evidence for excitoprotective and intraneuronal calcium-regulating roles for secreted forms of the β-amyloid precursor protein , 1993, Neuron.
[41] A. Dancer,et al. The specific problems of noise in military life. , 1998, Scandinavian audiology. Supplementum.
[42] K. Ryan,et al. Myc oncogenes: the enigmatic family. , 1996, The Biochemical journal.
[43] D. Selkoe,et al. Amyloid β-protein precursor: new clues to the genesis of Alzheimer's disease , 1994, Current Opinion in Neurobiology.
[44] H. Hydén,et al. S-100β has a neuronal localisation in the rat hindbrain revealed by an antigen retrieval method , 1995, Brain Research.
[45] W. Griffin,et al. Glial‐Neuronal Interactions in Alzheimer's Disease: The Potential Role of a ‘Cytokine Cycle’ in Disease Progression , 1998, Brain pathology.