Blockade of T Lymphocyte Costimulation with Cytotoxic T Lymphocyte–Associated Antigen 4–Immunoglobulin (Ctla4ig) Reverses the Cellular Pathology of Psoriatic Plaques, Including the Activation of Keratinocytes, Dendritic Cells, and Endothelial Cells
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M. Lebwohl | P. Linsley | Sewon Kang | S. Kelley | C. Guzzo | J. Krueger | B. Jegasothy | T. Kikuchi | E. Hayes | J. Abrams | Michael J. Brown
[1] R. Schwartz. The new immunology--the end of immunosuppressive drug therapy? , 1999, The New England journal of medicine.
[2] M. Lebwohl,et al. CTLA4Ig-mediated blockade of T-cell costimulation in patients with psoriasis vulgaris. , 1999, The Journal of clinical investigation.
[3] N. Kadowaki,et al. Reciprocal control of T helper cell and dendritic cell differentiation. , 1999, Science.
[4] J. Kieffer,et al. Interaction of Dendritic Cells with Skin Endothelium: A New Perspective on Immunosurveillance , 1999, Journal of Experimental Medicine.
[5] A. Lanzavecchia,et al. T lymphocyte costimulation mediated by reorganization of membrane microdomains. , 1999, Science.
[6] M. D. de Rie,et al. The pathogenesis of psoriasis: immunological facts and speculations. , 1999, Immunology today.
[7] G. Freeman,et al. The BB1 monoclonal antibody recognizes both cell surface CD74 (MHC class II-associated invariant chain) as well as B7-1 (CD80), resolving the question regarding a third CD28/CTLA-4 counterreceptor. , 1998, Journal of immunology.
[8] J. Banchereau,et al. A novel lysosome-associated membrane glycoprotein, DC-LAMP, induced upon DC maturation, is transiently expressed in MHC class II compartment. , 1998, Immunity.
[9] L. Turka,et al. The role of T-cell costimulatory activation pathways in transplant rejection. , 1998, The New England journal of medicine.
[10] R. Steinman,et al. A physiologic function for p-glycoprotein (MDR-1) during the migration of dendritic cells from skin via afferent lymphatic vessels. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[11] A. Lanzavecchia. Licence to kill , 1998 .
[12] Polly Matzinger,et al. A conditioned dendritic cell can be a temporal bridge between a CD4+ T-helper and a T-killer cell , 1998, Nature.
[13] R. Steinman,et al. Dendritic cells and the control of immunity , 1998, Nature.
[14] I. Kimber,et al. α6 Integrins Are Required for Langerhans Cell Migration from the Epidermis , 1997, The Journal of experimental medicine.
[15] D. Strunk,et al. A Skin Homing Molecule Defines the Langerhans Cell Progenitor in Human Peripheral Blood , 1997, The Journal of experimental medicine.
[16] S. Topino,et al. Kaposi's sarcoma cells express the macrophage-associated antigen mannose receptor and develop in peripheral blood cultures of Kaposi's sarcoma patients. , 1997, The American journal of pathology.
[17] A. Scheffold,et al. P- and E-selectin mediate recruitment of T-helper-1 but not T-helper-2 cells into inflamed tissues , 1997, Nature.
[18] P. Holt,et al. Brief Definitive Report Dendritic Cells Are Recruited into the Airway Epithelium during the Inflammatory Response to a Broad Spectrum of Stimuli , 2022 .
[19] B. Nickoloff,et al. Dermal injection of immunocytes induces psoriasis. , 1996, The Journal of clinical investigation.
[20] Alejandro Aruffo,et al. CD40 is functionally expressed on human keratinocytes , 1996, European journal of immunology.
[21] J. Pan,et al. Interleukin 4 or oncostatin M induces a prolonged increase in P- selectin mRNA and protein in human endothelial cells , 1996, The Journal of experimental medicine.
[22] A. Gaspari,et al. Human epidermal keratinocytes are induced to secrete interleukin‐6 and co‐stimulate T lymphocyte proliferation by a CD40‐dependent mechanism , 1996, European journal of immunology.
[23] G. Schuler,et al. Human cutaneous dendritic cells migrate through dermal lymphatic vessels in a skin organ culture model. , 1996, The Journal of investigative dermatology.
[24] R. Lechler,et al. Major histocompatibility complex class II-expressing endothelial cells induce allospecific nonresponsiveness in naive T cells , 1996, The Journal of experimental medicine.
[25] P. Das,et al. Effects of contact allergens on human Langerhans cells in skin organ culture: migration, modulation of cell surface molecules, and early expression of interleukin-1 beta protein. , 1996, Laboratory investigation; a journal of technical methods and pathology.
[26] Systemic tacrolimus (FK 506) is effective for the treatment of psoriasis in a double-blind, placebo-controlled study. The European FK 506 Multicentre Psoriasis Study Group. , 1996, Archives of dermatology.
[27] K. Nishioka,et al. Functional CD86 (B7-2/B70) on cultured human Langerhans cells. , 1996, The Journal of investigative dermatology.
[28] T. Ruzicka. Systemic tacrolimus (FK506) is effective for the treatment of psoriasis in a double-blind, placebo-controlled study , 1995 .
[29] C Danieli,et al. Dendritic cells use macropinocytosis and the mannose receptor to concentrate macromolecules in the major histocompatibility complex class II compartment: downregulation by cytokines and bacterial products , 1995, The Journal of experimental medicine.
[30] D. Carlo,et al. CD8+ T‐Cells in Psoriatic Lesions Preferentially Use T‐Cell Receptors Vβ3 and/or Vβ13.1 Genes , 1995 .
[31] R. Steinman,et al. Maturation and migration of cutaneous dendritic cells. , 1995, The Journal of investigative dermatology.
[32] P. Morris,et al. Dendritic cell loss from nonlymphoid tissues after systemic administration of lipopolysaccharide, tumor necrosis factor, and interleukin 1 , 1995, The Journal of experimental medicine.
[33] Liangji Zhou,et al. Human blood dendritic cells selectively express CD83, a member of the immunoglobulin superfamily. , 1995, Journal of immunology.
[34] C. Janeway,et al. Expression of the Co-stimulator Molecule B7–1 in Pancreatic β-Cells Accelerates Diabetes in the NOD Mouse , 1995, Diabetes.
[35] R. Steinman,et al. Both dendritic cells and memory T lymphocytes emigrate from organ cultures of human skin and form distinctive dendritic-T-cell conjugates. , 1995, The Journal of investigative dermatology.
[36] J. Banchereau,et al. B70/B7-2 is identical to CD86 and is the major functional ligand for CD28 expressed on human dendritic cells , 1994, The Journal of experimental medicine.
[37] J. Banchereau,et al. Activation of human dendritic cells through CD40 cross-linking , 1994, The Journal of experimental medicine.
[38] D. Carlo,et al. CD8+ T cells in psoriatic lesions preferentially use T-cell receptor V beta 3 and/or V beta 13.1 genes. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[39] J. Gribben,et al. The B7-2 (B70) costimulatory molecule expressed by monocytes and activated B lymphocytes is the CD86 differentiation antigen. , 1994, Blood.
[40] F. Nestle,et al. Characterization of dermal dendritic cells in psoriasis. Autostimulation of T lymphocytes and induction of Th1 type cytokines. , 1994, The Journal of clinical investigation.
[41] A. Gottlieb,et al. PUVA bath therapy strongly suppresses immunological and epidermal activation in psoriasis: a possible cellular basis for remittive therapy , 1994, The Journal of experimental medicine.
[42] P. Linsley,et al. Regulation of immunostimulatory function and costimulatory molecule (B7-1 and B7-2) expression on murine dendritic cells. , 1994, Journal of immunology.
[43] P. Linsley,et al. Costimulator B7-1 confers antigen-presenting-cell function to parenchymal tissue and in conjunction with tumor necrosis factor alpha leads to autoimmunity in transgenic mice. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[44] R. Flavell,et al. The role of the T cell costimulator B7-1 in autoimmunity and the induction and maintenance of tolerance to peripheral antigen. , 1994, Immunity.
[45] D. Harlan,et al. Mice expressing both B7-1 and viral glycoprotein on pancreatic beta cells along with glycoprotein-specific transgenic T cells develop diabetes due to a breakdown of T-lymphocyte unresponsiveness. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[46] P. Singer. Licence to kill , 1994, Nature.
[47] F. Nestle,et al. T lymphocytes in skin lesions of psoriasis and mycosis fungoides express B7-1: a ligand for CD28. , 1994, Blood.
[48] L. Turka,et al. Keratinocytes: key immunocytes of the integument. , 1993, The American journal of pathology.
[49] M. Bigby,et al. T-cell receptor V beta expression in normal human skin. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[50] P. Linsley,et al. Expression and function of B7 on human epidermal Langerhans cells. , 1993, Journal of immunology.
[51] P. Linsley,et al. The role of the CD28 receptor during T cell responses to antigen. , 1993, Annual review of immunology.
[52] L. Orci,et al. Expression of a tumor necrosis factor alpha transgene in murine pancreatic beta cells results in severe and permanent insulitis without evolution towards diabetes , 1992, The Journal of experimental medicine.
[53] A. Demidem,et al. T-lymphocyte-activating properties of epidermal antigen-presenting cells from normal and psoriatic skin: evidence that psoriatic epidermal antigen-presenting cells resemble cultured normal Langerhans cells. , 1991, The Journal of investigative dermatology.
[54] P. Linsley,et al. CTLA-4 is a second receptor for the B cell activation antigen B7 , 1991, The Journal of experimental medicine.
[55] L. Picker,et al. ELAM-1 is an adhesion molecule for skin-homing T cells , 1991, Nature.
[56] T. Annesley,et al. Cyclosporine for plaque-type psoriasis. Results of a multidose, double-blind trial. , 1991, The New England journal of medicine.
[57] R. Steinman,et al. Migration and maturation of Langerhans cells in skin transplants and explants , 1990, The Journal of experimental medicine.
[58] James T. Elder,et al. UM4D4+ (CDw60) T cells are compartmentalized into psoriatic skin and release lymphokines that induce a keratinocyte phenotype expressed in psoriatic lesions. , 1990, The Journal of investigative dermatology.
[59] R. Steinman,et al. The distinct leukocyte integrins of mouse spleen dendritic cells as identified with new hamster monoclonal antibodies , 1990, The Journal of experimental medicine.
[60] O. Majdic,et al. Cultured human Langerhans cells resemble lymphoid dendritic cells in phenotype and function. , 1989, The Journal of investigative dermatology.
[61] B. Nickoloff,et al. Characterization of factor XIIIa positive dermal dendritic cells in normal and inflamed skin , 1989, The British journal of dermatology.
[62] R. Hall,et al. Epidermal keratinocytes express the adhesion molecule intercellular adhesion molecule-1 in inflammatory dermatoses. , 1989, The Journal of investigative dermatology.
[63] B. Seed,et al. Endothelial leukocyte adhesion molecule 1: an inducible receptor for neutrophils related to complement regulatory proteins and lectins. , 1989, Science.
[64] C W Smith,et al. Recognition of an endothelial determinant for CD 18-dependent human neutrophil adherence and transendothelial migration. , 1988, The Journal of clinical investigation.
[65] R. Palmiter,et al. Diabetes and tolerance in transgenic mice expressing class II MHC molecules in pancreatic beta cells , 1988, Cell.
[66] Michael Loran Dustin,et al. Induction by IL 1 and interferon-gamma: tissue distribution, biochemistry, and function of a natural adherence molecule (ICAM-1). , 1986, Journal of immunology.
[67] N. Romani,et al. HLA‐DR expression on keratinocytes is a common feature of diseased skin , 1986, The British journal of dermatology.