Sensitivity to N-methyl-d-aspartic acid-induced convulsions is genetically associated with resistance to ethanol withdrawal seizures

[1]  J. Crabbe,et al.  Neural sensitivity to pentylenetetrazol convulsions in inbred and selectively bred mice , 1992, Brain Research.

[2]  K. Gale,et al.  Subcortical structures and pathways involved in convulsive seizure generation. , 1992, Journal of clinical neurophysiology : official publication of the American Electroencephalographic Society.

[3]  K. Gulya,et al.  Chronic ethanol ingestion decreases vasopressin mRNA in hypothalamic and extrahypothalamic nuclei of mouse brain , 1991, Brain Research.

[4]  B. Meldrum.  Excitatory Amino Acid Transmitters in Epilepsy , 1991, Epilepsia.

[5]  J. Crabbe,et al.  Effects of convulsants on handling-induced convulsions in mice selected for ethanol withdrawal severity , 1991, Brain Research.

[6]  K. Grant,et al.  Brain regional specificity and time-course of changes in the NMDA receptor-ionophore complex during ethanol withdrawal , 1991, Brain Research.

[7]  J. Crabbe,et al.  Genetic correlations among inbred strain sensitivities to convulsions induced by 9 convulsant drugs , 1990, Brain Research.

[8]  J. Crabbe,et al.  NMDA receptors in mice bred to be prone or resistant to ethanol withdrawal seizures. , 1990, European journal of pharmacology.

[9]  K. Grant,et al.  Ethanol and the NMDA receptor. , 1990, Alcohol.

[10]  J. Crabbe,et al.  Estimation of genetic correlation: interpretation of experiments using selectively bred and inbred animals. , 1990, Alcoholism, clinical and experimental research.

[11]  K. Grant,et al.  Ethanol withdrawal seizures and the NMDA receptor complex. , 1990, European journal of pharmacology.

[12]  G White,et al.  Ethanol inhibits NMDA-activated ion current in hippocampal neurons. , 1989, Science.

[13]  L. Iversen,et al.  The anticonvulsant MK-801 is a potent N-methyl-D-aspartate antagonist. , 1986, Proceedings of the National Academy of Sciences of the United States of America.

[14]  J. Crabbe,et al.  Bidirectional selection for susceptibility to ethanol withdrawal seizures inMus musculus , 1985, Behavior genetics.

[15]  W. Freed,et al.  Glutamic acid and ethanol dependence , 1978, Pharmacology Biochemistry and Behavior.

[16]  J. Crabbe,et al.  Alcohol dependence and withdrawal: a genetic animal model. , 1990, Annals of medicine.

[17]  C. Cotman,et al.  The excitatory amino acid receptors: their classes, pharmacology, and distinct properties in the function of the central nervous system. , 1989, Annual review of pharmacology and toxicology.

[18]  H. Gerhard,et al.  Epilepsy and the Reticular Formation: The Role of the Reticular Core in Convulsive Seizures , 1987 .

[19]  B. Clineschmidt,et al.  Anticonvulsant activity of (+)‐5‐methyl‐10, 11‐dihydro‐5H‐dibenzo[a, d]cyclohepten‐5, 10‐imine (MK‐801), a substance with potent anticonvulsant, central sympathomimetic, and apparent anxiolytic properties , 1982 .

[20]  W. Freed,et al.  Effects of acute and chronic ethanol intake on synaptosomal glutamate binding activity. , 1978, Biochemical pharmacology.