An Artemisia scoparia extract attenuates glucocorticoid-induced lipolysis in adipocytes.
暂无分享,去创建一个
[1] K. Bohnert,et al. A fat-promoting botanical extract from Artemisia scoparia exerts geroprotective effects on C. elegans lifespan and stress resistance. , 2022, The journals of gerontology. Series A, Biological sciences and medical sciences.
[2] Allison J. Richard,et al. Artemisia scoparia and Metabolic Health: Untapped Potential of an Ancient Remedy for Modern Use , 2022, Frontiers in Endocrinology.
[3] Brett A. Collins,et al. FGF1 and insulin control lipolysis by convergent pathways. , 2022, Cell metabolism.
[4] J. Stephens,et al. Artemisia scoparia promotes adipogenesis in the absence of adipogenic effectors , 2021, Obesity.
[5] I. Raskin,et al. Prenylated Coumaric Acids from Artemisia scoparia Beneficially Modulate Adipogenesis. , 2021, Journal of natural products.
[6] T. Harris,et al. Adipocyte lipolysis drives acute stress-induced insulin resistance , 2020, Scientific Reports.
[7] Tenzin Dagpo,et al. Exploring Therapeutic Targets to Reverse or Prevent the Transition from Metabolically Healthy to Unhealthy Obesity , 2020, Cells.
[8] Alexander Yang,et al. Adipocyte lipolysis: from molecular mechanisms of regulation to disease and therapeutics. , 2020, The Biochemical journal.
[9] Joseph W. Jackson,et al. One week of continuous corticosterone exposure impairs hepatic metabolic flexibility, promotes islet β-cell proliferation, and reduces physical activity in male C57BL/6 J mice , 2019, The Journal of Steroid Biochemistry and Molecular Biology.
[10] H. Sørensen,et al. Fifteen-year nationwide trends in systemic glucocorticoid drug use in Denmark. , 2019, European journal of endocrinology.
[11] I. Raskin,et al. Distinct Fractions of an Artemisia scoparia Extract Contain Compounds With Novel Adipogenic Bioactivity , 2019, Front. Nutr..
[12] J. Rood,et al. An ethanolic extract of Artemisia scoparia inhibits lipolysis in vivo and has antilipolytic effects on murine adipocytes in vitro. , 2018, American journal of physiology. Endocrinology and metabolism.
[13] Nathan R. Qi,et al. Glucocorticoid-Induced Metabolic Disturbances Are Exacerbated in Obese Male Mice , 2018, Endocrinology.
[14] A. Pariente,et al. Prevalence and prescription patterns of oral glucocorticoids in adults: a retrospective cross-sectional and cohort analysis in France , 2017, BMJ Open.
[15] H. Sørensen,et al. Systemic glucocorticoid use in Denmark: a population-based prevalence study , 2017, BMJ Open.
[16] C. Diwoky,et al. Pharmacological inhibition of adipose triglyceride lipase corrects high-fat diet-induced insulin resistance and hepatosteatosis in mice , 2017, Nature Communications.
[17] G. Shulman,et al. Mechanism for leptin’s acute insulin-independent effect to reverse diabetic ketoacidosis , 2017, The Journal of clinical investigation.
[18] D. Langin,et al. Adipocyte lipolysis and insulin resistance. , 2016, Biochimie.
[19] I. Hochberg,et al. Gene expression changes in subcutaneous adipose tissue due to Cushing's disease , 2015, Journal of molecular endocrinology.
[20] B. Geng,et al. Thiazolidinediones attenuate lipolysis and ameliorate dexamethasone-induced insulin resistance. , 2015, Metabolism: clinical and experimental.
[21] T. Mikkelsen,et al. Identification of nuclear hormone receptor pathways causing insulin resistance by transcriptional and epigenomic analysis , 2014, Nature Cell Biology.
[22] R. MacPherson,et al. Evidence for fatty acids mediating CL 316,243-induced reductions in blood glucose in mice. , 2014, American journal of physiology. Endocrinology and metabolism.
[23] J. Alvarez‐Leite,et al. Low-Grade Inflammation, Obesity, and Diabetes , 2014, Current Obesity Reports.
[24] D. Ribnicky,et al. Artemisia extracts activate PPARγ, promote adipogenesis, and enhance insulin sensitivity in adipose tissue of obese mice. , 2014 .
[25] D. Ribnicky,et al. Artemisia scoparia Enhances Adipocyte Development and Endocrine Function In Vitro and Enhances Insulin Action In Vivo , 2014, PloS one.
[26] W. Johnson,et al. Artemisia scoparia extract attenuates non-alcoholic fatty liver disease in diet-induced obesity mice by enhancing hepatic insulin and AMPK signaling independently of FGF21 pathway. , 2013, Metabolism: clinical and experimental.
[27] C. Deal,et al. Prevalence of oral glucocorticoid usage in the United States: A general population perspective , 2013, Arthritis care & research.
[28] S. Collins,et al. β-Adrenoceptor Signaling Networks in Adipocytes for Recruiting Stored Fat and Energy Expenditure , 2011, Front. Endocrin..
[29] I. Petersen,et al. Prevalence of long-term oral glucocorticoid prescriptions in the UK over the past 20 years. , 2011, Rheumatology.
[30] J. Graff,et al. Thiazolidinediones regulate adipose lineage dynamics. , 2011, Cell metabolism.
[31] Terence P. Speed,et al. Genome-Wide Analysis of Glucocorticoid Receptor Binding Regions in Adipocytes Reveal Gene Network Involved in Triglyceride Homeostasis , 2010, PloS one.
[32] P. Strålfors,et al. Differential regulation of adipocyte PDE3B in distinct membrane compartments by insulin and the beta3-adrenergic receptor agonist CL316243: effects of caveolin-1 knockdown on formation/maintenance of macromolecular signalling complexes. , 2009, The Biochemical journal.
[33] Guoheng Xu,et al. Direct effect of glucocorticoids on lipolysis in adipocytes. , 2009, Molecular endocrinology.
[34] C. Rosen. The rosiglitazone story--lessons from an FDA Advisory Committee meeting. , 2007, The New England journal of medicine.
[35] S. Nissen,et al. Effect of rosiglitazone on the risk of myocardial infarction and death from cardiovascular causes. , 2007, The New England journal of medicine.
[36] P. Arner,et al. NF-kappaB is important for TNF-alpha-induced lipolysis in human adipocytes. , 2007, Journal of lipid research.
[37] F. Sundler,et al. Alterations in regulation of energy homeostasis in cyclic nucleotide phosphodiesterase 3B-null mice. , 2006, The Journal of clinical investigation.
[38] E. Lander,et al. Reactive oxygen species have a causal role in multiple forms of insulin resistance , 2006, Nature.
[39] J. Cidlowski,et al. Antiinflammatory action of glucocorticoids--new mechanisms for old drugs. , 2005, The New England journal of medicine.
[40] J. Jessurun,et al. Sources of fatty acids stored in liver and secreted via lipoproteins in patients with nonalcoholic fatty liver disease. , 2005, The Journal of clinical investigation.
[41] M. Conti,et al. The Oligomerization State Determines Regulatory Properties and Inhibitor Sensitivity of Type 4 cAMP-specific Phosphodiesterases* , 2004, Journal of Biological Chemistry.
[42] J. Jameson,et al. A dominant negative PPARγ mutant shows altered cofactor recruitment and inhibits adipogenesis in 3T3-L1 cells , 2003, Diabetologia.
[43] V. Manganiello,et al. Tumor necrosis factor-alpha stimulates lipolysis in differentiated human adipocytes through activation of extracellular signal-related kinase and elevation of intracellular cAMP. , 2002, Diabetes.
[44] L. Witters. The blooming of the French lilac. , 2001, The Journal of clinical investigation.
[45] J. Mei,et al. Down-regulation of cyclic-nucleotide phosphodiesterase 3B in 3T3-L1 adipocytes induced by tumour necrosis factor alpha and cAMP. , 2000, The Biochemical journal.
[46] D. Matthews,et al. Systemic resistance to the antilipolytic effect of insulin in black and white women with visceral obesity. , 1999, American journal of physiology. Endocrinology and metabolism.
[47] W Ogawa,et al. Insulin-Induced Phosphorylation and Activation of Cyclic Nucleotide Phosphodiesterase 3B by the Serine-Threonine Kinase Akt , 1999, Molecular and Cellular Biology.
[48] G. Boden. Role of Fatty Acids in the Pathogenesis of Insulin Resistance and NIDDM , 1997, Diabetes.
[49] S. Rössner,et al. Effects of weight reduction on the regulation of lipolysis in adipocytes of women with upper-body obesity. , 1995, Clinical science.
[50] A. Brasier,et al. Tumor necrosis factor increases the rate of lipolysis in primary cultures of adipocytes without altering levels of hormone-sensitive lipase. , 1994, Endocrinology.
[51] R. Bone,et al. Role of tumor necrosis factor-α in disease states and inflammation , 1993 .
[52] M. Jensen,et al. Influence of body fat distribution on free fatty acid metabolism in obesity. , 1989, The Journal of clinical investigation.
[53] L. Poellinger,et al. Regulation of glucocorticoid receptor expression: evidence for transcriptional and posttranslational mechanisms. , 1988, Molecular endocrinology.
[54] J. Stevens,et al. Insulin resistance in adipocytes from fed and fasted obese rats: Dissociation of two insulin actions , 1981, Molecular and Cellular Biochemistry.
[55] T. Hawke,et al. Adipogenic and lipolytic effects of chronic glucocorticoid exposure. , 2011, American journal of physiology. Cell physiology.
[56] F. Pi-Sunyer,et al. Impaired insulin action in subcutaneous adipocytes from women with visceral obesity. , 2001, American journal of physiology. Endocrinology and metabolism.