Insights into psoriasis and other inflammatory diseases from large-scale gene expression studies.
暂无分享,去创建一个
A. Bowcock | W. Shannon | A. Menter | J. Catier | F. Du | K. Cao | K. Aftergut | A M Bowcock | M A Fernandez-Vina | W Shannon | F Du | J Duncan | K Cao | K Aftergut | J Catier | A Menter | J. Duncan | M. Fernández-Viña | Alan Menter | William D. Shannon | Fenghe Du | Jill Duncan | Kai Cao | Kent Aftergut | Jennifer Catier | Marcelo Fernandez-Vina
[1] C. Carcassi,et al. Fine mapping of the PSORS4 psoriasis susceptibility region on chromosome 1q21. , 2001, The Journal of investigative dermatology.
[2] G. Abecasis,et al. Genetic linkage of childhood atopic dermatitis to psoriasis susceptibility loci , 2001, Nature Genetics.
[3] C I Amos,et al. A genomewide screen in multiplex rheumatoid arthritis families suggests genetic overlap with other autoimmune diseases. , 2001, American journal of human genetics.
[4] I. Lawrance,et al. Ulcerative colitis and Crohn's disease: distinctive gene expression profiles and novel susceptibility candidate genes. , 2001, Human molecular genetics.
[5] I. Marenholz,et al. Identification of human epidermal differentiation complex (EDC)-encoded genes by subtractive hybridization of entire YACs to a gridded keratinocyte cDNA library. , 2001, Genome research.
[6] M. Lovett,et al. Zeroing in on tolerance , 2001, Nature Medicine.
[7] C. Li,et al. Model-based analysis of oligonucleotide arrays: expression index computation and outlier detection. , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[8] T. Wienker,et al. A major susceptibility locus for atopic dermatitis maps to chromosome 3q21 , 2000, Nature Genetics.
[9] K. Wang,et al. Sequencing and expression analysis of the serine protease gene cluster located in chromosome 19q13 region. , 2000, Gene.
[10] T. Wienker,et al. Genomewide scan in german families reveals evidence for a novel psoriasis-susceptibility locus on chromosome 19p13. , 2000, American journal of human genetics.
[11] P. V. D. van de Kerkhof,et al. Changes in Keratin 6 and Keratin 10 (Co-)Expression in Lesional and Symptomless Skin of Spreading Psoriasis , 2000, Dermatology.
[12] J. Warrington,et al. Comparison of human adult and fetal expression and identification of 535 housekeeping/maintenance genes. , 2000, Physiological genomics.
[13] R. Pfundt,et al. TNF-α and serum induce SKALP/elafin gene expression in human keratinocytes by a p38 MAP kinase-dependent pathway , 2000, Archives of Dermatological Research.
[14] L. Austin,et al. CD69, HLA-DR and the IL-2R identify persistently activated T cells in psoriasis vulgaris lesional skin: blood and skin comparisons by flow cytometry. , 2000, Journal of autoimmunity.
[15] T. Kupper,et al. Inflammatory skin diseases, T cells, and immune surveillance. , 1999, The New England journal of medicine.
[16] K. Asadullah,et al. The pathophysiological role of cytokines in psoriasis. , 1999, Drugs of today.
[17] B. Dallapiccola,et al. Evidence for interaction between psoriasis-susceptibility loci on chromosomes 6p21 and 1q21. , 1999, American journal of human genetics.
[18] J. Warrington,et al. A high-density probe array sample preparation method using 10- to 100-fold fewer cells , 1999, Nature Biotechnology.
[19] E. Farmer,et al. Epidermal HLA-DR and the enhancement of cutaneous reactivity to superantigenic toxins in psoriasis. , 1999, The Journal of clinical investigation.
[20] Qiliang Li,et al. Locus control regions: coming of age at a decade plus. , 1999, Trends in genetics : TIG.
[21] J. Riley,et al. Psoriasis susceptibility locus in chromosome region 3q21 identified in patients from southwest Sweden , 1999, European Journal of Human Genetics.
[22] B. Nickoloff. The immunologic and genetic basis of psoriasis. , 1999, Archives of dermatology.
[23] G. Mirza,et al. Human epidermal differentiation complex in a single 2.5 Mbp long continuum of overlapping DNA cloned in bacteria integrating physical and transcript maps. , 1999, The Journal of investigative dermatology.
[24] J. Schröder,et al. Human beta-defensin-2. , 1999, The international journal of biochemistry & cell biology.
[25] S. Uccini,et al. Immunohistochemical study of the early histopathologic changes occurring in trauma-injured skin of psoriatic patients. , 1999, European journal of dermatology : EJD.
[26] B. Doğan,et al. Keratin 15 expression in stratified epithelia: downregulation in activated keratinocytes. , 1999, The Journal of investigative dermatology.
[27] R. Benner,et al. IL‐1β and IFN‐γ induce the regenerative epidermal phenotype of psoriasis in the transwell skin organ culture system. IFN‐γ up‐regulates the expression of keratin 17 and keratinocyte transglutaminase via endogenous IL‐1 production , 1999 .
[28] D. Botstein,et al. Cluster analysis and display of genome-wide expression patterns. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[29] K. Miyazono,et al. Increased numbers of CD68 antigen positive dendritic epidermal cells and upregulation of CLA (cutaneous lymphocyte-associated antigen) expression on these cells in various skin diseases. , 1998, Journal of dermatological science.
[30] Luis R. Espinoza,et al. Insights into the pathogenesis of psoriasis and psoriatic arthritis. , 1998, The American journal of the medical sciences.
[31] A. Bowcock,et al. The genetics of psoriasis: a complex disorder of the skin and immune system. , 1998, Human molecular genetics.
[32] G. Gyapay,et al. New susceptibility locus for rheumatoid arthritis suggested by a genome-wide linkage study. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[33] J. Trent,et al. Clustering of non-major histocompatibility complex susceptibility candidate loci in human autoimmune diseases. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[34] James T. Elder,et al. Linkage analysis of human leukocyte antigen (HLA) markers in familial psoriasis: strong disequilibrium effects provide evidence for a major determinant in the HLA-B/-C region. , 1998, American journal of human genetics.
[35] H. Prydz,et al. Constitutive and interferon-gamma-induced expression of the human proteasome subunit multicatalytic endopeptidase complex-like 1. , 1998, Biochimica et biophysica acta.
[36] H. Prydz,et al. Constitutive and interferon-γ-induced expression of the human proteasome subunit multicatalytic endopeptidase complex-like 11EMBL accession number for MECL1 cDNA: Y13640.1 , 1998 .
[37] R. Steinman,et al. Intraepidermal lymphocytes in psoriatic lesions are activated GMP‐17(TIA‐1)+CD8+CD3+ CTLs as determined by phenotypic analysis , 1998, Journal of cutaneous pathology.
[38] D. Creamer,et al. Overexpression of the angiogenic factor platelet‐derived endothelial cell growth factor/thymidine phosphorylase in psoriatic epidermis , 1997, The British journal of dermatology.
[39] James T. Elder,et al. Evidence for two psoriasis susceptibility loci (HLA and 17q) and two novel candidate regions (16q and 20p) by genome-wide scan. , 1997, Human molecular genetics.
[40] J. Browne,et al. Identification of a major susceptibility locus on chromosome 6p and evidence for further disease loci revealed by a two stage genome-wide search in psoriasis. , 1997, Human molecular genetics.
[41] G. Barbesino,et al. Mapping of a major susceptibility locus for Graves' disease (GD-1) to chromosome 14q31. , 1997, The Journal of clinical endocrinology and metabolism.
[42] G. Varigos,et al. Expression of insulin-like growth factor binding protein-3 (IGFBP-3) in the psoriatic lesion. , 1997, The Journal of investigative dermatology.
[43] A. Gottlieb,et al. Identification of aberrantly regulated genes in diseased skin using the cDNA differential display technique. , 1997, The Journal of investigative dermatology.
[44] K. Matsushima,et al. Localization of monocyte chemotactic and activating factor (MCAF/MCP-1) in psoriasis. , 1996, Journal of dermatological science.
[45] E. Fisher,et al. Evidence that a locus for familial psoriasis maps to chromosome 4q , 1996, Nature Genetics.
[46] M. Blumenberg,et al. Regulation of epidermal expression of keratin K17 in inflammatory skin diseases. , 1996, The Journal of investigative dermatology.
[47] P. Kerkhof,et al. Transition from symptomless to lesional psoriatic skin , 1996, Clinical and experimental dermatology.
[48] J. Schalkwijk,et al. Induction of normal and psoriatic phenotypes in submerged keratinocyte cultures , 1996, Journal of cellular physiology.
[49] Jo Whittaker,et al. Human Molecular Genetics , 1996, Nature Medicine.
[50] H. Takahashi,et al. Decreased β2‐adrenergic receptor‐mRNA and Ioricrin‐mRNA, and increased involucrin‐mRNA transcripts in psoriatic epidermis: analysis by reverse transcription–polymerase chain reaction , 1996 .
[51] R. Benner,et al. Increased expression of interleukin-4 receptors on psoriatic epidermal cells. , 1996, The American journal of pathology.
[52] S. Plon,et al. Susceptibility to insulin-dependent diabetes mellitus maps to a locus (IDDM11) on human chromosome 14q24.3-q31. , 1996, Genomics.
[53] James T. Elder,et al. Assignment of psoriasin to human chromosomal band 1q21: coordinate overexpression of clustered genes in psoriasis. , 1996, The Journal of investigative dermatology.
[54] Jean Weissenbach,et al. Mapping of a susceptibility locus for Crohn's disease on chromosome 16 , 1996, Nature.
[55] Irene M. Leigh,et al. Keratins (Kl6 and Kl7) as markers of keratinocyte hyperproliferation in psoriasis in vivo and in vitro , 1995 .
[56] C. Slaughter,et al. Primary structures of two homologous subunits of PA28, a γ‐interferon‐inducible protein activator of the 20S proteasome , 1995, FEBS letters.
[57] K. Tanaka,et al. cDNA cloning and interferon gamma down-regulation of proteasomal subunits X and Y. , 1994, Science.
[58] A. Bowcock,et al. Gene for familial psoriasis susceptibility mapped to the distal end of human chromosome 17q. , 1994, Science.
[59] V. Dixit,et al. Aberrant production of interleukin-8 and thrombospondin-1 by psoriatic keratinocytes mediates angiogenesis. , 1994, The American journal of pathology.
[60] K. Hashimoto,et al. Differential expression of transforming growth factor-alpha (TGF-alpha) and EGF receptor in transitional area of psoriatic epidermis. , 1994, Journal of dermatological science.
[61] T. Iseki. [Vitamin B12 and transcobalamin in chronic myeloproliferative disorders]. , 1993, Rinsho byori. The Japanese journal of clinical pathology.
[62] R U Peter,et al. Increased expression of epidermal IL-8 receptor in psoriasis. Down-regulation by FK-506 in vitro. , 1993, Journal of immunology.
[63] W. Fujimoto,et al. Expression of cornifin in squamous differentiating epithelial tissues, including psoriatic and retinoic acid-treated skin. , 1993, The Journal of investigative dermatology.
[64] P. Chambon,et al. Identification of a new interferon-alpha-inducible gene (p27) on human chromosome 14q32 and its expression in breast carcinoma. , 1993, Cancer research.
[65] J. Garioch,et al. Group A streptococcal antigen‐specific T lymphocytes in guttate psoriatic lesions , 1993, The British journal of dermatology.
[66] R. Leube,et al. Characterization of the human gene encoding cytokeratin 17 and its expression pattern. , 1992, European journal of cell biology.
[67] H. Leffers,et al. Molecular cloning and expression of a novel keratinocyte protein (psoriasis-associated fatty acid-binding protein [PA-FABP]) that is highly up-regulated in psoriatic skin and that shares similarity to fatty acid-binding proteins. , 1992, The Journal of investigative dermatology.
[68] J. Lindemans,et al. Elevated serum transcobalamin levels in anaemia of rheumatoid arthritis: correlation with disease activity but not with serum tumour necrosis factor alpha and interleukin 6 , 1992, Journal of internal medicine.
[69] N. Basset-Seguin,et al. C-fos and c-jun proto-oncogene expression is decreased in psoriasis: an in situ quantitative analysis. , 1991, The Journal of investigative dermatology.
[70] H. Leffers,et al. Molecular cloning, occurrence, and expression of a novel partially secreted protein "psoriasin" that is highly up-regulated in psoriatic skin. , 1991, The Journal of investigative dermatology.
[71] A. Menter,et al. Psoriasis in practice , 1991, The Lancet.
[72] Peter J. Rousseeuw,et al. Finding Groups in Data: An Introduction to Cluster Analysis , 1990 .
[73] S. P. Fodor,et al. Light-directed, spatially addressable parallel chemical synthesis. , 1991, Science.
[74] M. Schlup,et al. Measurements of unsaturated vitamin B12-binding capacity and myeloperoxidase as indices of severity of acute inflammation in serial colonoscopy biopsy specimens from patients with inflammatory bowel disease. , 1990, Scandinavian journal of gastroenterology.
[75] M. Duvic,et al. Immunology of AIDS related to psoriasis. , 1990, The Journal of investigative dermatology.
[76] James T. Elder,et al. Growth factor and proto-oncogene expression in psoriasis. , 1990, The Journal of investigative dermatology.
[77] A. Gottlieb,et al. HLA region genes and immune activation in the pathogenesis of psoriasis. , 1990, Archives of dermatology.
[78] James T. Elder,et al. Decreased growth inhibition by recombinant gamma interferon is associated with increased transforming growth factor‐α production in keratinocytes cultured from psoriatic lesions , 1989, The British journal of dermatology.
[79] C. Figueroa,et al. Identification of a tissue kallikrein in human polymorphonuclear leucocytes , 1989, British journal of haematology.
[80] B. Bernard,et al. Precocious appearance of involucrin and epidermal transglutaminase during differentiation of psoriatic skin , 1986, The British journal of dermatology.
[81] T. Russell,et al. Histocompatibility (HL-A) antigens associated with psoriasis. , 1972, The New England journal of medicine.
[82] M. R. Mickey,et al. Disturbance of HL-A antigen frequency in Psoriasis. , 1972, The New England journal of medicine.
[83] F. Ruissen,et al. Serial analysis of gene expression in differentiated cultures of human epidermal keratinocytes. , 2001, The Journal of investigative dermatology.
[84] R. Pfundt,et al. TNF-alpha and serum induce SKALP/elafin gene expression in human keratinocytes by a p38 MAP kinase-dependent pathway. , 2000, Archives of dermatological research.
[85] R. Trembath,et al. Identification of a novel psoriasis susceptibility locus at 1p and evidence of epistasis between PSORS1 and candidate loci , 2001, Journal of medical genetics.
[86] S. P. Fodor,et al. High density synthetic oligonucleotide arrays , 1999, Nature Genetics.
[87] K. Cao,et al. High and intermediate resolution DNA typing systems for class I HLA-A, B, C genes by hybridization with sequence-specific oligonucleotide probes (SSOP). , 1999, Reviews in immunogenetics.
[88] B. Dallapiccola,et al. Searching for psoriasis susceptibility genes in Italy: genome scan and evidence for a new locus on chromosome 1. , 1999, The Journal of investigative dermatology.
[89] R. Benner,et al. IL-1 beta and IFN-gamma induce the regenerative epidermal phenotype of psoriasis in the transwell skin organ culture system. IFN-gamma up-regulates the expression of keratin 17 and keratinocyte transglutaminase via endogenous IL-1 production. , 1999, The Journal of pathology.
[90] M. Duvic,et al. Distinct serum cytokines in AIDS-related skin diseases. , 1998, Viral immunology.
[91] H. Takahashi,et al. Decreased beta 2-adrenergic receptor-mRNA and loricrin-mRNA, and increased involucrin-mRNA transcripts in psoriatic epidermis: analysis by reverse transcription-polymerase chain reaction. , 1996, The British journal of dermatology.
[92] I. Leigh,et al. Keratins (K16 and K17) as markers of keratinocyte hyperproliferation in psoriasis in vivo and in vitro. , 1995, The British journal of dermatology.
[93] B. Bonnekoh,et al. Up-regulation of keratin 17 expression in human HaCaT keratinocytes by interferon-gamma. , 1995, The Journal of investigative dermatology.
[94] S. Y. Yang,et al. Locus-specific amplification of HLA class I genes from genomic DNA: locus-specific sequences in the first and third introns of HLA-A, -B, and -C alleles. , 1995, Tissue antigens.
[95] J. Wilson,et al. Epidermis contains platelet-type 12-lipoxygenase that is overexpressed in germinal layer keratinocytes in psoriasis. , 1994, The American journal of physiology.
[96] A. Bowcock,et al. HLA genes in familial psoriasis vulgaris (PV): Evidence for genetic heterogeneity , 1994 .
[97] M. Sattar,et al. Plasma vitamin B12 binding proteins correlate with disease activity in patients with rheumatoid arthritis. , 1991, Medical laboratory sciences.
[98] Sattar Ma,et al. Plasma vitamin B12 binding proteins correlate with disease activity in patients with rheumatoid arthritis. , 1991 .