Redox Metals in Alzheimer's Disease
暂无分享,去创建一个
[1] T. Bayer,et al. Dietary Cu stabilizes brain superoxide dismutase 1 activity and reduces amyloid Aβ production in APP23 transgenic mice , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[2] J. Connor. Evidence for Iron Mismanagement in the Brain in Neurological Disorders , 1997 .
[3] F. Ye,et al. Increasing striatal iron content associated with normal aging , 1998, Movement disorders : official journal of the Movement Disorder Society.
[4] P. Doraiswamy,et al. Current Status of Metals as Therapeutic Targets in Alzheimer's Disease , 2003, Journal of the American Geriatrics Society.
[5] J. Cammermeyer. DEPOSITION OF IRON IN PARAVENTRICULAR AREAS OF THE HUMAN BRAIN IN HEMOCHROMATOSIS , 1947 .
[6] J. Andersen,et al. Ironing out Parkinson's disease: is therapeutic treatment with iron chelators a real possibility? , 2002, Aging cell.
[7] Xudong Huang,et al. The A beta peptide of Alzheimer's disease directly produces hydrogen peroxide through metal ion reduction. , 1999, Biochemistry.
[8] Or Kakhlon,et al. The labile iron pool: characterization, measurement, and participation in cellular processes(1). , 2002, Free radical biology & medicine.
[9] D. Sparks,et al. Trace amounts of copper in water induce β-amyloid plaques and learning deficits in a rabbit model of Alzheimer's disease , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[10] Jae-Young Koh,et al. Contribution by synaptic zinc to the gender-disparate plaque formation in human Swedish mutant APP transgenic mice , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[11] D. McLachlan,et al. Intramuscular desferrioxamine in patients with Alzheimer's disease , 1991, The Lancet.
[12] G. Bartzokis,et al. MR evaluation of age-related increase of brain iron in young adult and older normal males. , 1997, Magnetic resonance imaging.
[13] Or Kakhlon,et al. Serial review: iron and cellular redox statusThe labile iron pool: characterization, measurement, and participation in cellular processes1 , 2002 .
[14] J. Connor,et al. Iron and neurodegenerative disorders , 2001, Brain Research Bulletin.
[15] D. Westaway,et al. In vivo reduction of amyloid-β by a mutant copper transporter , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[16] J. Connor,et al. Cellular distribution of iron in the brain of the Belgrade rat , 1999, Neuroscience.
[17] James R. Connor,et al. Iron in the Brain: An Important Contributor in Normal and Diseased States , 2000 .
[18] Xudong Huang,et al. An Iron-responsive Element Type II in the 5′-Untranslated Region of the Alzheimer's Amyloid Precursor Protein Transcript* , 2002, The Journal of Biological Chemistry.
[19] M. Giorgi,et al. Differential decrease of copper content and of copper binding to superoxide dismutase in liver, heart and brain of copper-deficient rats. , 1994, Biochemical and biophysical research communications.
[20] J. Connor,et al. Iron S Involvement in the Molecular Mechanisms and Pathogenesis of Alzheimers Disease , 2003 .
[21] D. Mason,et al. Kupffer cell staining by an HFE‐specific monoclonal antibody: implications for hereditary haemochromatosis , 1998, British journal of haematology.
[22] J. Connor,et al. Distribution of divalent metal transporter 1 and metal transport protein 1 in the normal and Belgrade rat , 2001, Journal of neuroscience research.
[23] R. Scott,et al. Is hemochromatosis a risk factor for Alzheimer's disease? , 2001, Journal of Alzheimer's disease : JAD.
[24] A. Terashi,et al. Association of transferrin C2 allele with late-onset Alzheimer’s disease , 1997, Human Genetics.
[25] P. Pasqualetti,et al. Red blood cell copper, zinc superoxide dismutase activity is higher in Alzheimer's disease and is decreased by d-penicillamine , 2002, Neuroscience Letters.
[26] J. Connor,et al. The case for iron chelation and/or antioxidant therapy in Alzheimer's disease , 2002 .
[27] G. Perry,et al. Iron accumulation in Alzheimer disease is a source of redox-generated free radicals. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[28] J. Connor,et al. Ferritin, transferrin, and iron in selected regions of the adult and aged rat brain , 1993, The Journal of comparative neurology.
[29] T. Montine,et al. Association of HFE mutations with neurodegeneration and oxidative stress in Alzheimer's disease and correlation with APOE , 2003, American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics.
[30] W. Burke,et al. HFE gene and hereditary hemochromatosis: a HuGE review. Human Genome Epidemiology. , 2001, American journal of epidemiology.
[31] J. Connor,et al. Alterations in the interaction between iron regulatory proteins and their iron responsive element in normal and Alzheimer's diseased brains. , 2000, Cellular and molecular biology.
[32] C. Culmsee,et al. Cellular and Molecular Mechanisms Underlying Perturbed Energy Metabolism and Neuronal Degeneration in Alzheimer's and Parkinson's Diseases , 1999, Annals of the New York Academy of Sciences.
[33] O. Combarros,et al. Interaction of the H63D Mutation in the Hemochromatosis Gene with the Apolipoprotein E Epsilon 4 Allele Modulates Age at Onset of Alzheimer’s Disease , 2003, Dementia and Geriatric Cognitive Disorders.
[34] J. Drapier,et al. Iron Regulatory Proteins as NO Signal Transducers , 2003, Science's STKE.
[35] W. Stremmel,et al. Pathogenesis of genetic haemochromatosis , 1993, European journal of clinical investigation.
[36] J. Connor,et al. Iron status and neural functioning. , 2003, Annual review of nutrition.
[37] Joseph F. Clark,et al. Heme and Iron Metabolism: Role in Cerebral Hemorrhage , 2003, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[38] M. Failla. Trace elements and host defense: recent advances and continuing challenges. , 2003, The Journal of nutrition.
[39] J. Connor,et al. A Quantitative Analysis of Isoferritins in Select Regions of Aged, Parkinsonian, and Alzheimer's Diseased Brains , 1995, Journal of neurochemistry.
[40] A. Warren,et al. Are hereditary hemochromatosis mutations involved in Alzheimer disease? , 2000, American journal of medical genetics.
[41] A. Bush. Metal complexing agents as therapies for Alzheimer’s disease , 2002, Neurobiology of Aging.
[42] N. Andrews. The iron transporter DMT1. , 1999, The international journal of biochemistry & cell biology.
[43] R. Tanzi,et al. Role of free radicals and metal ions in the pathogenesis of Alzheimer's disease. , 1999, Metal ions in biological systems.
[44] S. Toyokuni. Iron and carcinogenesis: from Fenton reaction to target genes , 2002, Redox report : communications in free radical research.