NOX1-derived ROS drive the expression of Lipocalin-2 in colonic epithelial cells in inflammatory conditions
暂无分享,去创建一个
A. Tlili | A. Pelletier | P. Lettéron | Dan Liu | J. El-Benna | J. Marie | M. Hurtado-Nedelec | P. M. Dang | V. Marzaioli | A. Sannier | Coralie Pintard | Nesrine Makhezer | Marwa Ben Khemis | Yamina Khichane | Marjan Mojallali
[1] Subhasis Chaudhuri,et al. Current State of Art , 2019, Visual and Text Sentiment Analysis through Hierarchical Deep Learning Networks.
[2] A. Tlili,et al. NOX1-derived ROS drive the expression of Lipocalin-2 in colonic epithelial cells in inflammatory conditions , 2018, Mucosal Immunology.
[3] S. Gaffen,et al. IL-17 Signaling: The Yin and the Yang. , 2017, Trends in immunology.
[4] Daeui Park,et al. Involvement of NF-κBIZ and related cytokines in age-associated renal fibrosis , 2017, Oncotarget.
[5] Misaki Matsumoto,et al. NOX1/NADPH Oxidase Expressed in Colonic Macrophages Contributes to the Pathogenesis of Colonic Inflammation in Trinitrobenzene Sulfonic Acid–Induced Murine Colitis , 2017, The Journal of Pharmacology and Experimental Therapeutics.
[6] H. Tilg,et al. 3rd European Evidence-based Consensus on the Diagnosis and Management of Crohn’s Disease 2016: Part 1 Diagnosis and Medical Management , 2017, Journal of Crohn's & colitis.
[7] M. Gougerot-Pocidalo,et al. Priming of the neutrophil respiratory burst: role in host defense and inflammation , 2016, Immunological reviews.
[8] Alexander V. Zhdanov,et al. Defensive Mutualism Rescues NADPH Oxidase Inactivation in Gut Infection. , 2016, Cell host & microbe.
[9] H. Tilg,et al. Lipocalin 2 Protects from Inflammation and Tumorigenesis Associated with Gut Microbiota Alterations. , 2016, Cell host & microbe.
[10] B. Chassaing,et al. Microbiota-Inducible Innate Immune Siderophore Binding Protein Lipocalin 2 Is Critical for Intestinal Homeostasis , 2016, Cellular and molecular gastroenterology and hepatology.
[11] Su-Jae Lee,et al. NADPH Oxidase 1 Activity and ROS Generation Are Regulated by Grb2/Cbl-Mediated Proteasomal Degradation of NoxO1 in Colon Cancer Cells. , 2016, Cancer research.
[12] C. Fiocchi,et al. Immunopathogenesis of IBD: current state of the art , 2016, Nature Reviews Gastroenterology &Hepatology.
[13] J. Merchant,et al. Increased Expression of DUOX2 Is an Epithelial Response to Mucosal Dysbiosis Required for Immune Homeostasis in Mouse Intestine. , 2015, Gastroenterology.
[14] S. Brand,et al. Lipocalin-2 Is a Disease Activity Marker in Inflammatory Bowel Disease Regulated by IL-17A, IL-22, and TNF-&agr; and Modulated by IL23R Genotype Status , 2015, Inflammatory bowel diseases.
[15] Judy H. Cho,et al. Defects in Nicotinamide-adenine Dinucleotide Phosphate Oxidase Genes NOX1 and DUOX2 in Very Early Onset Inflammatory Bowel Disease , 2015, Cellular and molecular gastroenterology and hepatology.
[16] Tak W. Mak,et al. Regulation of tumour necrosis factor signalling: live or let die , 2015, Nature Reviews Immunology.
[17] R. Eliakim,et al. Expression of IL-2, IL-17 and TNF-alpha in patients with Crohn's disease treated with anti-TNF antibodies. , 2014, Clinics and research in hepatology and gastroenterology.
[18] Markus F. Neurath,et al. Cytokines in inflammatory bowel disease , 2014, Nature Reviews Immunology.
[19] J. Gálvez. Role of Th17 Cells in the Pathogenesis of Human IBD , 2014, ISRN inflammation.
[20] R. Poulsom,et al. DUOX2 and DUOXA2 Form the Predominant Enzyme System Capable of Producing the Reactive Oxygen Species H2O2 in Active Ulcerative Colitis and are Modulated by 5-Aminosalicylic Acid , 2014, Inflammatory bowel diseases.
[21] J. Doroshow,et al. Nox1 causes ileocolitis in mice deficient in glutathione peroxidase-1 and -2. , 2013, Free radical biology & medicine.
[22] J. Lambeth,et al. Symbiotic lactobacilli stimulate gut epithelial proliferation via Nox‐mediated generation of reactive oxygen species , 2013, The EMBO journal.
[23] Jung Mogg Kim,et al. Bacteroides fragilis enterotoxin upregulates lipocalin-2 expression in intestinal epithelial cells , 2013, Laboratory Investigation.
[24] M. Gougerot-Pocidalo,et al. NOXO1 phosphorylation on serine 154 is critical for optimal NADPH oxidase 1 assembly and activation , 2013, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[25] C. Reutelingsperger,et al. Annexin A1, formyl peptide receptor, and NOX1 orchestrate epithelial repair. , 2013, The Journal of clinical investigation.
[26] Pierluigi Gambetti,et al. Physiology and pathophysiology , 2013 .
[27] G. Yeretssian,et al. Intestinal antimicrobial peptides during homeostasis, infection, and disease , 2012, Front. Immun..
[28] T. Sharp,et al. Mucosal reactive oxygen species decrease virulence by disrupting Campylobacter jejuni phosphotyrosine signaling. , 2012, Cell host & microbe.
[29] J. Cowland,et al. Induction of Neutrophil Gelatinase-associated Lipocalin Expression by Co-stimulation with Interleukin-17 and Tumor Necrosis Factor-α Is Controlled by IκB-ζ but neither by C/EBP-β nor C/EBP-δ* , 2010, The Journal of Biological Chemistry.
[30] S. Gaffen,et al. Interleukin‐17 and its target genes: mechanisms of interleukin‐17 function in disease , 2010, Immunology.
[31] E. Marcotte,et al. Global signatures of protein and mRNA expression levelsw , 2009 .
[32] Judy H. Cho,et al. Interleukin-23/Th17 pathways and inflammatory bowel disease. , 2009, Inflammatory bowel diseases.
[33] P. Alex,et al. Distinct Cytokine Patterns Identified from Multiplex Profiles of Murine DSS and TNBS‐Induced Colitis , 2009, Inflammatory bowel diseases.
[34] P. Anderson. Post-transcriptional control of cytokine production , 2008, Nature Immunology.
[35] T. Leto,et al. Oxidative innate immune defenses by Nox/Duox family NADPH oxidases. , 2008, Contributions to microbiology.
[36] Y. Kuwahara,et al. Oxidative stress induced lipocalin 2 gene expression: addressing its expression under the harmful conditions. , 2007, Journal of radiation research.
[37] K. Krause,et al. The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology. , 2007, Physiological reviews.
[38] K. Rokutan,et al. NADPH oxidases in the gastrointestinal tract: a potential role of Nox1 in innate immune response and carcinogenesis. , 2006, Antioxidants & redox signaling.
[39] J. Bidart,et al. Dual oxidase2 is expressed all along the digestive tract. , 2005, American journal of physiology. Gastrointestinal and liver physiology.
[40] T. Muta,et al. Positive and Negative Regulation of Nuclear Factor-κB-mediated Transcription by IκB-ζ, an Inducible Nuclear Protein* , 2005, Journal of Biological Chemistry.
[41] T. Muta,et al. Positive and negative regulation of nuclear factor-kappaB-mediated transcription by IkappaB-zeta, an inducible nuclear protein. , 2005, The Journal of biological chemistry.
[42] Shizuo Akira,et al. Lipocalin 2 mediates an innate immune response to bacterial infection by sequestrating iron , 2004, Nature.
[43] A. Andoh,et al. Increased expression of interleukin 17 in inflammatory bowel disease , 2003, Gut.
[44] R. Strong,et al. The neutrophil lipocalin NGAL is a bacteriostatic agent that interferes with siderophore-mediated iron acquisition. , 2002, Molecular cell.
[45] Li Yan,et al. The High Molecular Weight Urinary Matrix Metalloproteinase (MMP) Activity Is a Complex of Gelatinase B/MMP-9 and Neutrophil Gelatinase-associated Lipocalin (NGAL) , 2001, The Journal of Biological Chemistry.
[46] Y. Suh,et al. Cell transformation by the superoxide-generating oxidase Mox1 , 1999, Nature.
[47] S. Targan,et al. A Short-Term Study of Chimeric Monoclonal Antibody cA2 to Tumor Necrosis Factor α for Crohn's Disease , 1997 .
[48] S. Targan,et al. A short-term study of chimeric monoclonal antibody cA2 to tumor necrosis factor alpha for Crohn's disease. Crohn's Disease cA2 Study Group. , 1997, The New England journal of medicine.
[49] M. Baker,et al. Evidence of oxidant-induced injury to epithelial cells during inflammatory bowel disease. , 1996, The Journal of clinical investigation.
[50] B. Nielsen,et al. Induction of NGAL synthesis in epithelial cells of human colorectal neoplasia and inflammatory bowel diseases. , 1996, Gut.
[51] H. Sengeløv,et al. Isolation and primary structure of NGAL, a novel protein associated with human neutrophil gelatinase. , 1993, The Journal of biological chemistry.
[52] D. Rampton,et al. Chemiluminescence assay of mucosal reactive oxygen metabolites in inflammatory bowel disease. , 1992, Gastroenterology.
[53] A. Keshavarzian,et al. Excessive production of reactive oxygen metabolites by inflamed colon: analysis by chemiluminescence probe. , 1992, Gastroenterology.
[54] C. Laurent. Live or let die? , 1992, Nursing times.
[55] H. Towbin,et al. Electrophoretic transfer of proteins from polyacrylamide gels to nitrocellulose sheets: procedure and some applications. , 1979, Proceedings of the National Academy of Sciences of the United States of America.
[56] U. K. Laemmli,et al. Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4 , 1970, Nature.