Long noncoding RNA Gm20319, acting as competing endogenous RNA, regulated GNE expression by sponging miR-7240-5p to involve in deoxynivalenol-induced liver damage in vitro.

[1]  Liegang Liu,et al.  Roles of microRNAs and prospective view of competing endogenous RNAs in mycotoxicosis. , 2019, Mutation research.

[2]  Liegang Liu,et al.  Heme oxygenase-1 attenuates low-dose of deoxynivalenol-induced liver inflammation potentially associating with microbiota. , 2019, Toxicology and applied pharmacology.

[3]  W. Cheng,et al.  Non-coding RNA-Associated ceRNA Networks in a New Contrast-Induced Acute Kidney Injury Rat Model , 2019, Molecular therapy. Nucleic acids.

[4]  Liang Wang,et al.  Long non-coding RNA AGAP2-AS1, functioning as a competitive endogenous RNA, upregulates ANXA11 expression by sponging miR-16-5p and promotes proliferation and metastasis in hepatocellular carcinoma , 2019, Journal of Experimental & Clinical Cancer Research.

[5]  S. Shi,et al.  Deoxynivalenol‐induced oxidative stress and Nrf2 translocation in maternal liver on gestation day 12.5 d and 18.5 d , 2019, Toxicon : official journal of the International Society on Toxinology.

[6]  Baofeng Yang,et al.  Long non-coding RNA Gm2199 rescues liver injury and promotes hepatocyte proliferation through the upregulation of ERK1/2 , 2018, Cell Death & Disease.

[7]  Kang-sheng Liu,et al.  Advances of Long Noncoding RNAs-mediated Regulation in Reproduction , 2018, Chinese medical journal.

[8]  C. Dinarello.  Overview of the IL‐1 family in innate inflammation and acquired immunity , 2018, Immunological reviews.

[9]  K. Kuča,et al.  Trichothecenes: immunomodulatory effects, mechanisms, and anti-cancer potential , 2017, Archives of Toxicology.

[10]  S. Madec,et al.  Hepatotoxicity of fusariotoxins, alone and in combination, towards the HepaRG human hepatocyte cell line. , 2017, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.

[11]  I. Nonaka,et al.  Sialic acid deficiency is associated with oxidative stress leading to muscle atrophy and weakness in GNE myopathy , 2017, Human molecular genetics.

[12]  Junfeng Zhang,et al.  TGF-β-induced hepatocyte lincRNA-p21 contributes to liver fibrosis in mice , 2017, Scientific Reports.

[13]  Liegang Liu,et al.  Embryotoxicity Caused by DON-Induced Oxidative Stress Mediated by Nrf2/HO-1 Pathway , 2017, Toxins.

[14]  Liegang Liu,et al.  Current sights for mechanisms of deoxynivalenol‐induced hepatotoxicity and prospective views for future scientific research: A mini review , 2017, Journal of applied toxicology : JAT.

[15]  S. Qin,et al.  Exogenous supplement of N-acetylneuraminic acid ameliorates atherosclerosis in apolipoprotein E-deficient mice. , 2016, Atherosclerosis.

[16]  Xiong Guo,et al.  Prevalence of Selenium, T-2 Toxin, and Deoxynivalenol in Kashin–Beck Disease Areas in Qinghai Province, Northwest China , 2016, Biological Trace Element Research.

[17]  Meng-yu Zhang,et al.  Overexpression of the long noncoding RNA TUG1 protects against cold‐induced injury of mouse livers by inhibiting apoptosis and inflammation , 2016, The FEBS journal.

[18]  Chi-Hung Huang,et al.  Mechanism and inhibition of human UDP-GlcNAc 2-epimerase, the key enzyme in sialic acid biosynthesis , 2016, Scientific Reports.

[19]  Bicheng Chen,et al.  MALAT1 functions as a competing endogenous RNA to mediate Rac1 expression by sequestering miR-101b in liver fibrosis , 2015, Cell cycle.

[20]  N. Ismail,et al.  High fat diet-induced inflammation and oxidative stress are attenuated by N-acetylneuraminic acid in rats , 2015, Journal of Biomedical Science.

[21]  Xiaoming Fan,et al.  Long Non-coding RNA Growth Arrest-specific Transcript 5 (GAS5) Inhibits Liver Fibrogenesis through a Mechanism of Competing Endogenous RNA* , 2015, The Journal of Biological Chemistry.

[22]  J. F. Burrows,et al.  Targeting Siglecs with a sialic acid–decorated nanoparticle abrogates inflammation , 2015, Science Translational Medicine.

[23]  Lvhui Sun,et al.  Individual and combined cytotoxic effects of aflatoxin B1, zearalenone, deoxynivalenol and fumonisin B1 on BRL 3A rat liver cells. , 2015, Toxicon : official journal of the International Society on Toxinology.

[24]  R. Barbouche,et al.  The food born mycotoxin deoxynivalenol induces low-grade inflammation in mice in the absence of observed-adverse effects. , 2015, Toxicology letters.

[25]  J. Mott,et al.  Overview of MicroRNA Biology , 2015, Seminars in Liver Disease.

[26]  M. Szmitkowski,et al.  Serum Sialic Acids Levels According to the Severity of Liver Cirrhosis , 2014, Journal of clinical laboratory analysis.

[27]  Mukul S. Bansal,et al.  A comparative encyclopedia of DNA elements in the mouse genome , 2014, Nature.

[28]  H. Yoon,et al.  Superoxide Dismutase Gene (SOD1, SOD2, and SOD3) Polymorphisms and Antituberculosis Drug-induced Hepatitis , 2014, Allergy, asthma & immunology research.

[29]  V. Dohnal,et al.  Deoxynivalenol: signaling pathways and human exposure risk assessment—an update , 2014, Archives of Toxicology.

[30]  C. Chien,et al.  Sialic acid rescues repurified lipopolysaccharide-induced acute renal failure via inhibiting TLR4/PKC/gp91-mediated endoplasmic reticulum stress, apoptosis, autophagy, and pyroptosis signaling. , 2014, Toxicological sciences : an official journal of the Society of Toxicology.

[31]  R. Flisiak,et al.  Total and Free Serum Sialic Acid Concentration in Liver Diseases , 2014, BioMed research international.

[32]  J. Groopman,et al.  Public health impacts of foodborne mycotoxins. , 2014, Annual review of food science and technology.

[33]  A. Jean,et al.  Central inflammation and sickness-like behavior induced by the food contaminant deoxynivalenol: a PGE2-independent mechanism. , 2011, Toxicological sciences : an official journal of the Society of Toxicology.

[34]  P. Pandolfi,et al.  A ceRNA Hypothesis: The Rosetta Stone of a Hidden RNA Language? , 2011, Cell.

[35]  X. Lei,et al.  Knockout of SOD1 altered murine hepatic glycolysis, gluconeogenesis, and lipogenesis , 2011, Free radical biology & medicine.

[36]  J. Pestka.  Deoxynivalenol: mechanisms of action, human exposure, and toxicological relevance , 2010, Archives of Toxicology.

[37]  Dirk E. Smith,et al.  The IL-1 family: regulators of immunity , 2010, Nature Reviews Immunology.

[38]  C. Chien,et al.  SIALIC ACID REDUCES ACUTE ENDOTOXEMIA-INDUCED LIVER DYSFUNCTION IN THE RAT , 2008, Shock.

[39]  Z. Islam,et al.  Immunochemical assessment of deoxynivalenol tissue distribution following oral exposure in the mouse. , 2008, Toxicology letters.

[40]  A. Varki,et al.  Diversity in cell surface sialic acid presentations: implications for biology and disease , 2007, Laboratory Investigation.

[41]  C. Epstein,et al.  CuZnSOD deficiency leads to persistent and widespread oxidative damage and hepatocarcinogenesis later in life , 2005, Oncogene.

[42]  V. Ambros.  The functions of animal microRNAs , 2004, Nature.

[43]  J. Bennett,et al.  Mycotoxins , 2003, Clinical Microbiology Reviews.

[44]  C. Wild,et al.  Development of a urinary biomarker of human exposure to deoxynivalenol. , 2003, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.

[45]  J. Briggs,et al.  Glycoengineering of therapeutic glycoproteins: in vitro galactosylation and sialylation of glycoproteins with terminal N-acetylglucosamine and galactose residues. , 2001, Biochemistry.

[46]  J. Pestka,et al.  Induction of cytokine mRNAs in mice after oral exposure to the trichothecene vomitoxin (deoxynivalenol): relationship to toxin distribution and protein synthesis inhibition. , 1995, Toxicology and applied pharmacology.

[47]  Liegang Liu,et al.  Food raw materials and food production occurrences of deoxynivalenol in different regions , 2019, Trends in Food Science & Technology.

[48]  M. Huizing,et al.  UDP-GlcNAc 2-Epimerase/ManNAc Kinase (GNE): A Master Regulator of Sialic Acid Synthesis. , 2015, Topics in current chemistry.

[49]  M. Florczyk,et al.  MicroRNA expression profiles in liver and colon of sexually immature gilts after exposure to Fusarium mycotoxins. , 2015, Polish journal of veterinary sciences.