Chapter 26 Neurotransmitters and cytokines in CNS pathology

[1]  L. Ignarro,et al.  Microglial cell cytotoxicity of oligodendrocytes is mediated through nitric oxide. , 1993, Journal of immunology.

[2]  D. Reis,et al.  Synthesis of nitric oxide in CNS glial cells , 1993, Trends in Neurosciences.

[3]  A. Nussler,et al.  Inflammation, immunoregulation, and inducible nitric oxide synthase , 1993, Journal of leukocyte biology.

[4]  J. Albina,et al.  Nitric oxide-mediated apoptosis in murine peritoneal macrophages. , 1993, Journal of immunology.

[5]  V. Souvannavong,et al.  Nitric oxide synthase induces macrophage death by apoptosis. , 1993, Biochemical and biophysical research communications.

[6]  M J Sanderson,et al.  Mechanisms of intercellular calcium signaling in glial cells studied with dantrolene and thapsigargin , 1993, Glia.

[7]  J. Gornbein,et al.  Production of interleukin-1 by microglia in response to substance P: role for a non-classical NK-1 receptor , 1993, Journal of Neuroimmunology.

[8]  M. Sanderson,et al.  Substance P stimulates IL-1 production by astrocytes via intracellular calcium , 1992, Brain Research.

[9]  T. Molitor,et al.  Activated microglia mediate neuronal cell injury via a nitric oxide mechanism. , 1992, Journal of immunology.

[10]  K. Boje,et al.  Microglial-produced nitric oxide and reactive nitrogen oxides mediate neuronal cell death , 1992, Brain Research.

[11]  J. Merrill,et al.  Autoimmune disease and the nervous system. Biochemical, molecular, and clinical update. , 1992, The Western journal of medicine.

[12]  H. Hartung,et al.  Production of nitrite by neonatal rat microglial cells/brain macrophages. , 1992, Cellular immunology.

[13]  Terry D. Lee,et al.  Cloning and characterization of inducible nitric oxide synthase from mouse macrophages. , 1992, Science.

[14]  N. Welsh,et al.  Interleukin-1 beta-induced nitric oxide production in isolated rat pancreatic islets requires gene transcription and may lead to inhibition of the Krebs cycle enzyme aconitase. , 1991, Endocrinology.

[15]  M. Wadhwa,et al.  Detection of interleukin-1 and interleukin-6 in adult rat brain, following mechanical injury, by in vivo microdialysis: evidence of a role for microglia in cytokine production , 1991, Journal of Neuroimmunology.

[16]  M. Sharief,et al.  Association between Tumor Necrosis Factor-α and Disease Progression in Patients with Multiple Sclerosis , 1991 .

[17]  A. Charles,et al.  Intercellular signaling in glial cells: Calcium waves and oscillations in response to mechanical stimulation and glutamate , 1991, Neuron.

[18]  P. Kubes,et al.  Nitric oxide: an endogenous modulator of leukocyte adhesion. , 1991, Proceedings of the National Academy of Sciences of the United States of America.

[19]  D. S. Webb,et al.  Cytokine-induced enhancement of ICAM-1 expression results in increased vulnerability of tumor cells to monocyte-mediated lysis. , 1991, Journal of immunology.

[20]  M. Iriki,et al.  Action site of circulating interleukin-1 on the rabbit brain , 1991, Brain Research.

[21]  A. Ganong,et al.  Activity and distribution of binding sites in brain of a nonpeptide substance P (NK1) receptor antagonist. , 1991, Science.

[22]  J. Lowe,et al.  A potent nonpeptide antagonist of the substance P (NK1) receptor. , 1991, Science.

[23]  J. Merrill,et al.  Natural and induced cytotoxicity of oligodendrocytes by microglia is inhibitable by TGFβ , 1991, Glia.

[24]  J. Merrill,et al.  Effects of interleukin-1 and tumor necrosis factor-alpha on astrocytes, microglia, oligodendrocytes, and glial precursors in vitro. , 1991, Developmental neuroscience.

[25]  S. Wahl,et al.  Antagonistic and agonistic effects of transforming growth factor-beta and IL-1 in rheumatoid synovium. , 1990, Journal of immunology.

[26]  S. Hoffman,et al.  Cellular mechanisms of nonspecific immunity to intracellular infection: cytokine-induced synthesis of toxic nitrogen oxides from L-arginine by macrophages and hepatocytes. , 1990, Immunology letters.

[27]  J. Adams,et al.  hypotension: Implications for the involvement of nitric oxide , 2022 .

[28]  M. Graeber,et al.  New expression of myelomonocytic antigens by microglia and perivascular cells following lethal motor neuron injury , 1990, Journal of Neuroimmunology.

[29]  M. Persson,et al.  The Neuropeptide Substance P Stimulates Production of Interleukin 1 in Human Blood Monocytes: Activated Cells are Preferentially Influenced by the Neuropeptide , 1990, Scandinavian journal of immunology.

[30]  B. Freeman,et al.  Apparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxide. , 1990, Proceedings of the National Academy of Sciences of the United States of America.

[31]  A. Cross,et al.  Homing to central nervous system vasculature by antigen-specific lymphocytes. II. Lymphocyte/endothelial cell adhesion during the initial stages of autoimmune demyelination. , 1990, Laboratory investigation; a journal of technical methods and pathology.

[32]  C. Dinarello,et al.  Correlations and interactions in the production of interleukin-6 (IL-6), IL-1, and tumor necrosis factor (TNF) in human blood mononuclear cells: IL-6 suppresses IL-1 and TNF. , 1990, Blood.

[33]  S. Hauser,et al.  Substance P immunoreactive astrocytes are present in multiple sclerosis plaques , 1989, Brain Research.

[34]  M. Brockhaus,et al.  Cell surface tumor necrosis factor (TNF) accounts for monocyte- and lymphocyte-mediated killing of TNF-resistant target cells. , 1989, Cellular immunology.

[35]  M. Shin,et al.  Production of tumor necrosis factor and other cytokines by astrocytes stimulated with lipopolysaccharide or a neurotropic virus. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[36]  J. Merrill,et al.  Tumor necrosis factor identified in multiple sclerosis brain , 1989, The Journal of experimental medicine.

[37]  C. Boehmer,et al.  Substance P receptor binding sites are expressed by glia in vivo after neuronal injury. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[38]  M. Turner,et al.  Modulation of cytokine production by transforming growth factor-beta. , 1989, Journal of immunology.

[39]  J. Glowinski,et al.  Tachykinin Receptors of the NK1 Type (Substance P) Coupled Positively to Phospholipase C on Cortical Astrocytes from the Newborn Mouse in Primary Culture , 1989, Journal of neurochemistry.

[40]  P. J. Larsen,et al.  Binding of a iodinated substance P analog to a NK-1 receptor on isolated cell membranes from rat anterior pituitary. , 1989, Endocrinology.

[41]  H. Kettenmann,et al.  Activation of substance P receptors leads to membrane potential responses in cultured astrocytes , 1989, Glia.

[42]  D. Regoli,et al.  Receptors for substance P and related neurokinins. , 1989, Pharmacology.

[43]  M. Shingu,et al.  Possible role of H2O2-mediated complement activation and cytokines-mediated fibroblasts superoxide generation on skin inflammation. , 1989, Dermatologica.

[44]  E. S. Kimball,et al.  Substance P, neurokinin A, and neurokinin B induce generation of IL-1-like activity in P388D1 cells. Possible relevance to arthritic disease. , 1988, Journal of immunology.

[45]  H. Kirchner,et al.  Increased production of interferon gamma and tumor necrosis factor precedes clinical manifestation in multiple sclerosis: Do cytokines trigger off exacerbations? , 1988, Acta neurologica Scandinavica.

[46]  D. Carson,et al.  Effect of neuropeptides on production of inflammatory cytokines by human monocytes , 1988 .

[47]  P. Mantyh,et al.  Receptor binding sites for substance P, but not substance K or neuromedin K, are expressed in high concentrations by arterioles, venules, and lymph nodules in surgical specimens obtained from patients with ulcerative colitis and Crohn disease. , 1988, Proceedings of the National Academy of Sciences of the United States of America.

[48]  J. Drapier,et al.  Differentiation of murine macrophages to express nonspecific cytotoxicity for tumor cells results in L-arginine-dependent inhibition of mitochondrial iron-sulfur enzymes in the macrophage effector cells. , 1988, Journal of immunology.

[49]  K. Selmaj,et al.  Tumor necrosis factor mediates myelin and oligodendrocyte damage in vitro , 1988, Annals of neurology.

[50]  H. Hartung,et al.  Substance P and astrocytes: stimulation of the cyclooxygenase pathway of arachidonic acid metabolism , 1988, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[51]  M. Shin,et al.  Production of cytotoxic factor for oligodendrocytes by stimulated astrocytes. , 1987, Journal of immunology.

[52]  M. Nakano,et al.  Calcium ionophore A23187 does not stimulate lipopolysaccharide nonresponsive C3H/HeJ peritoneal macrophages to produce interleukin 1. , 1987, Journal of immunology.

[53]  S. Pizzo,et al.  Effects of bacterial lipopolysaccharide on the hydrolysis of phosphatidylinositol-4,5-bisphosphate in murine peritoneal macrophages. , 1987, Journal of immunology.

[54]  M. Marletta,et al.  Induction of nitrite/nitrate synthesis in murine macrophages by BCG infection, lymphokines, or interferon-gamma. , 1987, Journal of immunology.

[55]  R. Hirsch,et al.  Treatment of multiple sclerosis with gamma interferon , 1987, Neurology.

[56]  R. Peck.  Neuropeptides Modulating Macrophage Function , 1987, Annals of the New York Academy of Sciences.

[57]  D. Carson,et al.  Substance P activation of rheumatoid synoviocytes: neural pathway in pathogenesis of arthritis. , 1987, Science.

[58]  T. Decker,et al.  Cell-associated tumor necrosis factor (TNF) as a killing mechanism of activated cytotoxic macrophages. , 1987, Journal of immunology.

[59]  J. Glowinski,et al.  Substance P receptors in primary cultures of cortical astrocytes from the mouse. , 1986, Proceedings of the National Academy of Sciences of the United States of America.

[60]  E. Burcher,et al.  Multiple tachykinin binding sites in hamster, rat and guinea-pig urinary bladder. , 1986, European journal of pharmacology.

[61]  D Giulian,et al.  Characterization of ameboid microglia isolated from developing mammalian brain , 1986, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[62]  G. Conrad,et al.  Platelet activating factor raises intracellular calcium ion concentration in macrophages , 1986, The Journal of cell biology.

[63]  S. Brandwein.  Regulation of interleukin 1 production by mouse peritoneal macrophages. Effects of arachidonic acid metabolites, cyclic nucleotides, and interferons. , 1986, The Journal of biological chemistry.

[64]  Michael Loran Dustin,et al.  Induction by IL 1 and interferon-gamma: tissue distribution, biochemistry, and function of a natural adherence molecule (ICAM-1). , 1986, Journal of immunology.

[65]  D. Faller,et al.  Interferon-gamma induction of LFA-1-mediated homotypic adhesion of human monocytes. , 1986, Journal of immunology.

[66]  H. Hartung,et al.  Substance P: binding properties and studies on cellular responses in guinea pig macrophages. , 1986, Journal of immunology.

[67]  J. Merrill,et al.  Immunoregulatory molecules and IL 2 receptors identified in multiple sclerosis brain. , 1986, Journal of immunology.

[68]  R. Featherstone,et al.  An examination of the pharmacology of two substance P antagonists and the evidence for tachykinin receptor subtypes , 1986, British journal of pharmacology.

[69]  R. Ekman,et al.  Substance P and vasoactive intestinal peptide in bullous and inflammatory skin disease. , 1986, Acta dermato-venereologica.

[70]  C. Pert,et al.  Substance P receptor-mediated chemotaxis of human monocytes , 1985, Peptides.

[71]  J. Denburg,et al.  Mast cell heterogeneity: effects of neuroenteric peptides on histamine release. , 1985, Journal of immunology.

[72]  J. Glowinski,et al.  Pharmacological characterisation of two tachykinin binding sites in the rat cerebral cortex , 1985, Neuropeptides.

[73]  E. Goetzl,et al.  Substance P recognition by a subset of human T lymphocytes. , 1984, The Journal of clinical investigation.

[74]  F. Lembeck,et al.  Effects of capsaicin on inflammation and on the substance P content of nervous tissues in rats with adjuvant arthritis. , 1983, Life sciences.

[75]  R. Goldman,et al.  Enhancement of phagocytosis - a newly found activity of substance P residing in its N-terminal tetrapeptide sequence. , 1980, Biochemical and biophysical research communications.