Assessment of Demographic, Genetic, and Imaging Variables Associated With Brain Resilience and Cognitive Resilience to Pathological Tau in Patients With Alzheimer Disease
暂无分享,去创建一个
M. Gorno-Tempini | J. Kramer | C. Sudre | A. Boxer | G. Rabinovici | O. Hansson | Hanna Cho | S. Palmqvist | Ruben Smith | R. Ossenkoppele | O. Strandberg | R. La Joie | J. Choi | Y. Ryu | C. Lyoo | Jonas Jester-Broms | B. Miller | B. Miller | B. Miller
[1] C. Jack,et al. Association of Apolipoprotein E ɛ4, Educational Level, and Sex With Tau Deposition and Tau-Mediated Metabolic Dysfunction in Older Adults , 2019, JAMA network open.
[2] W. M. van der Flier,et al. Cognitive reserve and clinical progression in Alzheimer disease , 2019, Neurology.
[3] M. Ewers,et al. Left frontal connectivity attenuates the adverse effect of entorhinal tau pathology on memory , 2019, Neurology.
[4] David T. Jones,et al. Prevalence of Biologically vs Clinically Defined Alzheimer Spectrum Entities Using the National Institute on Aging–Alzheimer’s Association Research Framework , 2019, JAMA neurology.
[5] C. Jack,et al. White matter hyperintensities: relationship to amyloid and tau burden. , 2019, Brain : a journal of neurology.
[6] W. Jagust,et al. Biomarkers for tau pathology , 2019, Molecular and Cellular Neuroscience.
[7] Keith A. Johnson,et al. Sex Differences in the Association of Global Amyloid and Regional Tau Deposition Measured by Positron Emission Tomography in Clinically Normal Older Adults , 2019, JAMA neurology.
[8] C. Jack,et al. Limbic-predominant age-related TDP-43 encephalopathy (LATE): consensus working group report , 2019, Brain : a journal of neurology.
[9] Stephen Salloway,et al. A multicentre longitudinal study of flortaucipir (18F) in normal ageing, mild cognitive impairment and Alzheimer’s disease dementia , 2019, Brain : a journal of neurology.
[10] L. Beckett,et al. White matter hyperintensities in vascular contributions to cognitive impairment and dementia (VCID): Knowledge gaps and opportunities , 2019, Alzheimer's & dementia.
[11] Özgür A. Onur,et al. Level of education mitigates the impact of tau pathology on neuronal function , 2019, European Journal of Nuclear Medicine and Molecular Imaging.
[12] W. M. van der Flier,et al. Nature and implications of sex differences in AD pathology , 2018, Nature Reviews Neurology.
[13] B. Miller,et al. Discriminative Accuracy of [18F]flortaucipir Positron Emission Tomography for Alzheimer Disease vs Other Neurodegenerative Disorders , 2018, JAMA.
[14] Y. Stern,et al. Whitepaper: Defining and investigating cognitive reserve, brain reserve, and brain maintenance , 2018, Alzheimer's & Dementia.
[15] Rik Ossenkoppele,et al. Is intracranial volume a suitable proxy for brain reserve? , 2018, Alzheimer's Research & Therapy.
[16] O. Hansson,et al. Greater tau load and reduced cortical thickness in APOE ε4-negative Alzheimer’s disease: a cohort study , 2018, Alzheimer's Research & Therapy.
[17] Sterling C. Johnson,et al. Sex-Specific Association of Apolipoprotein E With Cerebrospinal Fluid Levels of Tau , 2018, JAMA neurology.
[18] Philip S. Insel,et al. Associations between tau, Aβ, and cortical thickness with cognition in Alzheimer disease , 2019, Neurology.
[19] Prashanthi Vemuri,et al. Resistance vs resilience to Alzheimer disease , 2018, Neurology.
[20] Christopher G Schwarz,et al. Longitudinal tau PET in ageing and Alzheimer’s disease , 2018, Brain : a journal of neurology.
[21] W. M. van der Flier,et al. Differential effects of cognitive reserve and brain reserve on cognition in Alzheimer disease , 2018, Neurology.
[22] Daniel R. Schonhaut,et al. Tau pathology and neurodegeneration contribute to cognitive impairment in Alzheimer’s disease , 2017, Brain : a journal of neurology.
[23] Rik Ossenkoppele,et al. Distinct 18F‐AV‐1451 tau PET retention patterns in early‐ and late‐onset Alzheimer's disease , 2017, Brain : a journal of neurology.
[24] K. Fliessbach,et al. Tau pathology and cognitive reserve in Alzheimer's disease , 2017, Neurobiology of Aging.
[25] W. M. van der Flier,et al. A neuroimaging approach to capture cognitive reserve: Application to Alzheimer's disease , 2017, Human brain mapping.
[26] William J. Jagust,et al. Comparison of multiple tau-PET measures as biomarkers in aging and Alzheimer's disease , 2017, NeuroImage.
[27] C. Jack,et al. Age and sex specific prevalences of cerebral β-amyloidosis, tauopathy and neurodegeneration among clinically normal individuals aged 50-95 years: a cross-sectional study , 2017, The Lancet Neurology.
[28] Mark E Bastin,et al. Sex Differences in the Adult Human Brain: Evidence from 5216 UK Biobank Participants , 2017, bioRxiv.
[29] Keith A. Johnson,et al. Cognitive resilience in clinical and preclinical Alzheimer’s disease: the Association of Amyloid and Tau Burden on cognitive performance , 2017, Brain Imaging and Behavior.
[30] Timothy J. Hohman,et al. Asymptomatic Alzheimer disease , 2016, Neurology.
[31] Hanna Cho,et al. In vivo cortical spreading pattern of tau and amyloid in the Alzheimer disease spectrum , 2016, Annals of neurology.
[32] Daniel R. Schonhaut,et al. Tau PET patterns mirror clinical and neuroanatomical variability in Alzheimer's disease. , 2016, Brain : a journal of neurology.
[33] Daniel R. Schonhaut,et al. PET Imaging of Tau Deposition in the Aging Human Brain , 2016, Neuron.
[34] D. Y. Lee,et al. Prevalence of cerebral amyloid pathology in persons without dementia: a meta-analysis. , 2015, JAMA.
[35] Sébastien Ourselin,et al. Bayesian Model Selection for Pathological Neuroimaging Data Applied to White Matter Lesion Segmentation , 2015, IEEE Transactions on Medical Imaging.
[36] Michel Goedert,et al. Tau pathology and neurodegeneration , 2013, The Lancet Neurology.
[37] William Jagust,et al. Vulnerable Neural Systems and the Borderland of Brain Aging and Neurodegeneration , 2013, Neuron.
[38] Y. Stern. Cognitive reserve in ageing and Alzheimer's disease , 2012, The Lancet Neurology.
[39] C. Jack,et al. Neuroimaging correlates of pathologically defined subtypes of Alzheimer's disease: a case-control study , 2012, The Lancet Neurology.
[40] Yuan Yuan,et al. Accelerated aging-related transcriptome changes in the female prefrontal cortex , 2012, Aging cell.
[41] L. Nyberg,et al. Memory aging and brain maintenance , 2012, Trends in Cognitive Sciences.
[42] K. Jellinger,et al. Correlation of Alzheimer Disease Neuropathologic Changes With Cognitive Status: A Review of the Literature , 2012, Journal of neuropathology and experimental neurology.
[43] Nick C Fox,et al. The diagnosis of mild cognitive impairment due to Alzheimer’s disease: Recommendations from the National Institute on Aging-Alzheimer’s Association workgroups on diagnostic guidelines for Alzheimer's disease , 2011, Alzheimer's & Dementia.
[44] J. Morris,et al. The diagnosis of dementia due to Alzheimer’s disease: Recommendations from the National Institute on Aging-Alzheimer’s Association workgroups on diagnostic guidelines for Alzheimer's disease , 2011, Alzheimer's & Dementia.
[45] Nick C Fox,et al. Early-onset versus late-onset Alzheimer's disease: the case of the missing APOE ɛ4 allele , 2011, The Lancet Neurology.
[46] David A Wolk,et al. Apolipoprotein E (APOE) genotype has dissociable effects on memory and attentional–executive network function in Alzheimer’s disease , 2010, Proceedings of the National Academy of Sciences.
[47] B. Winblad,et al. Donepezil treatment in severe Alzheimer's disease: a pooled analysis of three clinical trials , 2009, Current medical research and opinion.
[48] J. Durlak. How to select, calculate, and interpret effect sizes. , 2009, Journal of pediatric psychology.
[49] C. Pike,et al. Protective actions of sex steroid hormones in Alzheimer’s disease , 2009, Frontiers in Neuroendocrinology.
[50] Paul M. Thompson,et al. Mapping the effect of APOE ε4 on gray matter loss in Alzheimer's disease in vivo , 2009, NeuroImage.
[51] P. Matthews,et al. Distinct patterns of brain activity in young carriers of the APOE e4 allele , 2009, NeuroImage.
[52] Carl W. Cotman,et al. Gene expression changes in the course of normal brain aging are sexually dimorphic , 2008, Proceedings of the National Academy of Sciences.
[53] S. Pocock,et al. The Strengthening the Reporting of Observational Studies in Epidemiology (STROBE) statement: guidelines for reporting observational studies. , 2007, Preventive medicine.
[54] Matthias Egger,et al. The Strengthening the Reporting of Observational Studies in Epidemiology (STROBE) Statement: Guidelines for Reporting Observational Studies , 2007, PLoS medicine.
[55] R. Woods,et al. Sex differences in cortical thickness mapped in 176 healthy individuals between 7 and 87 years of age. , 2007, Cerebral cortex.
[56] Sun I. Kim,et al. Gender difference analysis of cortical thickness in healthy young adults with surface-based methods , 2006, NeuroImage.
[57] T. Kirkwood,et al. Understanding the Odd Science of Aging , 2005, Cell.
[58] K. Rockwood,et al. Size of the treatment effect on cognition of cholinesterase inhibition in Alzheimer’s disease , 2004, Journal of Neurology, Neurosurgery & Psychiatry.
[59] G. Alexander,et al. Functional brain abnormalities in young adults at genetic risk for late-onset Alzheimer's dementia , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[60] M. Todd,et al. Human Brain Mapping , 2003 .
[61] K. Hall,et al. The relationships between age, sex, and the incidence of dementia and Alzheimer disease: a meta-analysis. , 1998, Archives of general psychiatry.