Oxidized α1-antitrypsin stimulates the release of monocyte chemotactic protein-1 from lung epithelial cells: potential role in emphysema
暂无分享,去创建一个
[1] John D. Johnson,et al. Neonatal lung neutrophils and elastase/proteinase inhibitor imbalance. , 2015, The American review of respiratory disease.
[2] T. Haas,et al. JNK as a positive regulator of angiogenic potential in endothelial cells , 2008, Cell biology international.
[3] D. Postma,et al. Nitrogen Dioxide Exposure Attenuates Cigarette Smoke-Induced Cytokine Production in Mice , 2008, Inhalation toxicology.
[4] G. Joos,et al. Cigarette Smoke-Induced Pulmonary Inflammation and Emphysema Are Attenuated in CCR6-Deficient Mice1 , 2006, The Journal of Immunology.
[5] P. Barnes,et al. Chemokine receptors as therapeutic targets in chronic obstructive pulmonary disease. , 2006, Trends in pharmacological sciences.
[6] T. Flotte,et al. α-1 Antitrypsin Inhibits Caspase-3 Activity, Preventing Lung Endothelial Cell Apoptosis , 2006 .
[7] M. Hollenberg,et al. Proteinase-activated receptor-1 mediates elastase-induced apoptosis of human lung epithelial cells. , 2005, American journal of respiratory cell and molecular biology.
[8] D. Lomas,et al. Polymers of Z alpha1-antitrypsin co-localize with neutrophils in emphysematous alveoli and are chemotactic in vivo. , 2005, The American journal of pathology.
[9] Tomoko Betsuyaku,et al. Chemokines in bronchiolar epithelium in the development of chronic obstructive pulmonary disease. , 2004, American journal of respiratory cell and molecular biology.
[10] Xiaomin Wang,et al. Leukocyte elastase induces epithelial apoptosis: role of mitochondial permeability changes and Akt. , 2004, American journal of physiology. Gastrointestinal and liver physiology.
[11] P. Paré,et al. The nature of small-airway obstruction in chronic obstructive pulmonary disease. , 2004, The New England journal of medicine.
[12] I. Adcock,et al. Effect of smoking on MAP kinase-induced modulation of IL‐8 in human alveolar macrophages , 2004, European Respiratory Journal.
[13] N. McElvaney,et al. Z alpha1-antitrypsin polymerizes in the lung and acts as a neutrophil chemoattractant. , 2004, Chest.
[14] U. Pastorino,et al. Primary human alveolar type II epithelial cell chemokine release: effects of cigarette smoke and neutrophil elastase. , 2004, American journal of respiratory cell and molecular biology.
[15] R. Pauwels,et al. Chronic obstructive pulmonary disease: molecular and cellularmechanisms , 2003, European Respiratory Journal.
[16] L. Kunz-Schughart,et al. Expression and Release of Interleukin-8 by Human Bronchial Epithelial Cells from Patients with Chronic Obstructive Pulmonary Disease, Smokers, and Never-Smokers , 2003, Respiration.
[17] D. Perlmutter. Liver injury in alpha1-antitrypsin deficiency: an aggregated protein induces mitochondrial injury. , 2002, The Journal of clinical investigation.
[18] D. Lomas,et al. Alpha1-antitrypsin polymerization and the serpinopathies: pathobiology and prospects for therapy. , 2002, The Journal of clinical investigation.
[19] T. Betsuyaku,et al. Increased levels of interleukin-8 in BAL fluid from smokers susceptible to pulmonary emphysema , 2002, Thorax.
[20] S. Lindgren,et al. Human monocyte activation by cleaved form of alpha-1-antitrypsin involvement of the phagocytic pathway. , 2001, European journal of biochemistry.
[21] J. Whisstock,et al. The Serpins Are an Expanding Superfamily of Structurally Similar but Functionally Diverse Proteins , 2001, The Journal of Biological Chemistry.
[22] P. Paré,et al. Amplification of inflammation in emphysema and its association with latent adenoviral infection. , 2001, American journal of respiratory and critical care medicine.
[23] K. Chung,et al. Cytokines in chronic obstructive pulmonary disease , 2001, European Respiratory Journal.
[24] P. Barnes. Cytokine modulators as novel therapies for airway disease , 2001, European Respiratory Journal.
[25] M. Wu,et al. Inhibitor of nuclear factor-kappaB induction by cAMP antagonizes interleukin-1-induced human macrophage-colony-stimulating-factor expression. , 2001, The Biochemical journal.
[26] S. Lindgren,et al. Effects of Noninhibitory α-1-Antitrypsin on Primary Human Monocyte Activation in Vitro , 2001 .
[27] R. Chambers,et al. Alpha‐1‐antitrypsin stimulates fibroblast proliferation and procollagen production and activates classical MAP kinase signalling pathways , 2001, Journal of cellular physiology.
[28] Randy J. Read,et al. Structure of a serpin–protease complex shows inhibition by deformation , 2000, Nature.
[29] P. Vandenabeele,et al. Functional Protection by Acute Phase Proteins &agr;1-Acid Glycoprotein and &agr;1-Antitrypsin Against Ischemia/Reperfusion Injury by Preventing Apoptosis and Inflammation , 2000, Circulation.
[30] J. Moss,et al. Oxidation of either methionine 351 or methionine 358 in alpha 1-antitrypsin causes loss of anti-neutrophil elastase activity. , 2000, The Journal of biological chemistry.
[31] R. Stockley,et al. Airways inflammation in chronic bronchitis: the effects of smoking and alpha1-antitrypsin deficiency. , 2000, The European respiratory journal.
[32] S. Janciauskiene,et al. Activation of Primary Human Monocytes by the Oxidized Form of α1-Antitrypsin* , 2000, The Journal of Biological Chemistry.
[33] R. Stockley,et al. Evidence for excessive bronchial inflammation during an acute exacerbation of chronic obstructive pulmonary disease in patients with alpha(1)-antitrypsin deficiency (PiZ). , 1999, American journal of respiratory and critical care medicine.
[34] D. Lomas,et al. Heteropolymerization of S, I, and Z α1-antitrypsin and liver cirrhosis , 1999 .
[35] D. Mattey,et al. Oxidation of alpha1-proteinase inhibitor by the myeloperoxidase-hydrogen peroxidase system promotes binding to immunoglobulin A. , 1999, Biochemical and biophysical research communications.
[36] W. MacNee,et al. Neutrophil chemokines in bronchoalveolar lavage fluid and leukocyte-conditioned medium from nonsmokers and smokers. , 1998, The European respiratory journal.
[37] D. Lomas,et al. Lung polymers in Z alpha1-antitrypsin deficiency-related emphysema. , 1998, American journal of respiratory cell and molecular biology.
[38] W. Vogel,et al. The acute-phase protein α1-antitrypsin inhibits transferrin-receptor binding and proliferation of human skin fibroblasts , 1998 .
[39] M. Karin,et al. Regulation and function of the JNK subgroup of MAP kinases. , 1997, Biochimica et biophysica acta.
[40] W. Fiers,et al. Alpha 1-acid glycoprotein and alpha 1-antitrypsin inhibit TNF-induced but not anti-Fas-induced apoptosis of hepatocytes in mice. , 1997, Journal of immunology.
[41] E. Zandi,et al. AP-1 function and regulation. , 1997, Current opinion in cell biology.
[42] P. Gettins,et al. Major proteinase movement upon stable serpin-proteinase complex formation. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[43] R. Kettritz,et al. Apoptosis of endothelial cells induced by the neutrophil serine proteases proteinase 3 and elastase. , 1996, The American journal of pathology.
[44] R. Crystal,et al. Systemic deficiency of glutathione in cystic fibrosis. , 1993, Journal of applied physiology.
[45] R. Senior,et al. The serpin-enzyme complex (SEC) receptor mediates the neutrophil chemotactic effect of alpha-1 antitrypsin-elastase complexes and amyloid-beta peptide. , 1992, The Journal of clinical investigation.
[46] D. Lomas,et al. The mechanism of Z alpha 1-antitrypsin accumulation in the liver. , 1992, Nature.
[47] R. Crystal,et al. Aerosol α1 -antitrypsin treatment for cystic fibrosis , 1991, The Lancet.
[48] R. Crystal,et al. Molecular basis of alpha-1-antitrypsin deficiency. , 1988, The American journal of medicine.
[49] G. K. Scott,et al. Modulation of cell proliferation by protein proteinase inhibitors. A new analytical approach. , 1988, Biological chemistry Hoppe-Seyler.
[50] R. Senior,et al. The inhibitory complex of human alpha 1-proteinase inhibitor and human leukocyte elastase is a neutrophil chemoattractant , 1988, The Journal of experimental medicine.
[51] S. Weiss,et al. Oxidative regulation of neutrophil elastase-alpha-1-proteinase inhibitor interactions. , 1986, The Journal of clinical investigation.
[52] J. Carlson,et al. Risk of cirrhosis and primary liver cancer in alpha 1-antitrypsin deficiency. , 1986, The New England journal of medicine.
[53] R. Senior,et al. Determination of oxidized alpha-1-proteinase inhibitor in serum. , 1982, The Journal of laboratory and clinical medicine.
[54] J. Hoidal,et al. Potential mechanism of emphysema: alpha 1-proteinase inhibitor recovered from lungs of cigarette smokers contains oxidized methionine and has decreased elastase inhibitory capacity. , 1982, Proceedings of the National Academy of Sciences of the United States of America.
[55] R. Spragg,et al. Studies on the pathogenesis of the adult respiratory distress syndrome. , 1982, The Journal of clinical investigation.
[56] J. Travis,et al. Isolation and properties of oxidized alpha-1-proteinase inhibitor from human rheumatoid synovial fluid. , 1980, Biochemical and biophysical research communications.
[57] J. Travis,et al. Kinetics of association of serine proteinases with native and oxidized alpha-1-proteinase inhibitor and alpha-1-antichymotrypsin. , 1980, The Journal of biological chemistry.
[58] R. Crystal,et al. Cigarette smoking induces functional antiprotease deficiency in the lower respiratory tract of humans. , 1979, Science.
[59] D. Johnson,et al. The oxidative inactivation of human alpha-1-proteinase inhibitor. Further evidence for methionine at the reactive center. , 1979, The Journal of biological chemistry.
[60] W. Krivit,et al. Cirrhosis associated with alpha-1-antitrypsin deficiency: a previously unrecognized inherited disorder. , 1969, The Journal of laboratory and clinical medicine.
[61] P. Gross,et al. EXPERIMENTAL LUNG CANCER IN HAMSTERS. REPETITIVE INTRATRACHEAL APPLICATIONS OF TWO CARCINOGENIC HYDROCARBONS. , 1965, Archives of environmental health.
[62] B. Balbi,et al. Increased MCP-1 and MIP-1b in bronchoalveolar lavage fluid of chronic bronchitics , 1999 .
[63] J. Travis,et al. Kinetic and chemical evidence for the inability of oxidized alpha 1-proteinase inhibitor to protect lung elastin from elastolytic degradation. , 1984, Hoppe-Seyler's Zeitschrift fur physiologische Chemie.
[64] C. Laurell,et al. The Electrophoretic α;1-Globulin Pattern of Serum in α;1-Antitrypsin Deficiency , 1963 .