Activation of dendritic antigen‐presenting cells expressing common heat shock protein receptor CD91 during induction of psoriasis
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O. Boyman | C. Conrad | E. Kielhorn | B J Nickoloff | O Boyman | C Conrad | C Dudli | E Kielhorn | F O Nestle | C. Dudli | B. J. Nickoloff | F. Nestle | F. O. Nestlé | B. Nickoloff | Curdin Conrad
[1] P. Srivastava,et al. CD91 is a common receptor for heat shock proteins gp96, hsp90, hsp70, and calreticulin. , 2001, Immunity.
[2] J. Barker,et al. Pathophysiology of psoriasis. , 1977, Annual review of medicine.
[3] Frank O. Nestle,et al. Spontaneous Development of Psoriasis in a New Animal Model Shows an Essential Role for Resident T Cells and Tumor Necrosis Factor-α , 2004, The Journal of experimental medicine.
[4] Liangji Zhou,et al. CD14+ blood monocytes can differentiate into functionally mature CD83+ dendritic cells. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[5] F. Nestle,et al. Characterization of dermal dendritic cells in psoriasis. Autostimulation of T lymphocytes and induction of Th1 type cytokines. , 1994, The Journal of clinical investigation.
[6] R. Morimoto,et al. Expression of human HSP70 during the synthetic phase of the cell cycle. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[7] P. Ohashi,et al. Hsp70 promotes antigen-presenting cell function and converts T-cell tolerance to autoimmunity in vivo , 2003, Nature Medicine.
[8] B. Nickoloff. Skin innate immune system in psoriasis: friend or foe? , 1999, The Journal of clinical investigation.
[9] J. Ovigne,et al. Normal keratinocytes express Toll‐like receptors (TLRs) 1, 2 and 5: modulation of TLR expression in chronic plaque psoriasis , 2003, The British journal of dermatology.
[10] June K. Robinson,et al. Innate immune-related receptors in normal and psoriatic skin. , 2003, Archives of pathology & laboratory medicine.
[11] A. Bowcock,et al. Psoriasis vulgaris: cutaneous lymphoid tissue supports T-cell activation and "Type 1" inflammatory gene expression. , 2004, Trends in immunology.
[12] P. Srivastava,et al. Necrotic but not apoptotic cell death releases heat shock proteins, which deliver a partial maturation signal to dendritic cells and activate the NF-kappa B pathway. , 2000, International immunology.
[13] J. Voorhees,et al. Psoriasis, T Cells and Autoimmunity , 1996, Journal of the Royal Society of Medicine.
[14] B. Nickoloff,et al. Dermal injection of immunocytes induces psoriasis. , 1996, The Journal of clinical investigation.
[15] J. Fraki,et al. Transplantation of psoriatic skin onto nude mice. , 1983, The Journal of investigative dermatology.
[16] P. Srivastava,et al. Interaction of heat shock proteins with peptides and antigen presenting cells: chaperoning of the innate and adaptive immune responses. , 2002, Annual review of immunology.
[17] B. Gazzard,et al. Disease-associated dendritic cells respond to disease-specific antigens through the common heat shock protein receptor. , 2003, Blood.
[18] J. Gudjonsson,et al. Immunopathogenic mechanisms in psoriasis , 2004, Clinical and experimental immunology.
[19] C. Reis e Sousa,et al. Toll-like receptors and dendritic cells: for whom the bug tolls. , 2004, Seminars in immunology.
[20] D. Carlo,et al. CD8+ T cells in psoriatic lesions preferentially use T-cell receptor V beta 3 and/or V beta 13.1 genes. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[21] U. Chaudhari,et al. Efficacy and safety of infliximab monotherapy for plaque-type psoriasis: a randomised trial , 2001, The Lancet.
[22] S. Porcelli,et al. The CD1 system: antigen-presenting molecules for T cell recognition of lipids and glycolipids. , 1999, Annual review of immunology.
[23] P. Mease,et al. Etanercept in the treatment of psoriatic arthritis and psoriasis: a randomised trial , 2000, The Lancet.
[24] S. Pizzo,et al. A CD91-Positive Subset of CD11c+ Blood Dendritic Cells: Characterization of the APC that Functions to Enhance Adaptive Immune Responses against CD91-Targeted Antigens 1 , 2004, The Journal of Immunology.
[25] M. Karin,et al. Nuclear factor-kappaB: a pivotal transcription factor in chronic inflammatory diseases. , 1997, The New England journal of medicine.
[26] P. Matzinger,et al. Danger signals: SOS to the immune system. , 2001, Current opinion in immunology.
[27] J. Krueger. The immunologic basis for the treatment of psoriasis with new biologic agents. , 2002, Journal of the American Academy of Dermatology.
[28] B. Gazzard,et al. The heat-shock protein receptor CD91 is up-regulated in monocytes of HIV-1-infected "true" long-term nonprogressors. , 2003, Blood.
[29] W. Sterry,et al. Differential expression of heat shock protein 70 (HSP70) and heat shock cognate protein 70 (HSC70) in human epidermis , 2004, Archives of Dermatological Research.
[30] J. Nick,et al. By Binding SIRPα or Calreticulin/CD91, Lung Collectins Act as Dual Function Surveillance Molecules to Suppress or Enhance Inflammation , 2003, Cell.
[31] James G. Krueger,et al. Response of psoriasis to a lymphocyte-selective toxin (DAB389IL-2) suggests a primary immune, but not keratinocyte, pathogenic basis , 1995, Nature Genetics.
[32] Shizuo Akira,et al. TLR signaling pathways. , 2004, Seminars in immunology.
[33] H. Rammensee,et al. The heat shock protein gp96 induces maturation of dendritic cells and down‐regulation of its receptor , 2000, European journal of immunology.