This information is current as Multiple Sclerosis Immune and Environmental Regulators in TCR Transgenic Mice That Shed Light on

[1]  L. Steinman The discovery of natalizumab, a potent therapeutic for multiple sclerosis , 2012, The Journal of cell biology.

[2]  L. Fugger,et al.  Bridging the gap from genetic association to functional understanding: the next generation of mouse models of multiple sclerosis , 2012, Immunological reviews.

[3]  H. Cantor,et al.  Regulation of self-tolerance by Qa-1-restricted CD8(+) regulatory T cells. , 2011, Seminars in immunology.

[4]  K. Berer,et al.  Commensal microbiota and myelin autoantigen cooperate to trigger autoimmune demyelination , 2011, Nature.

[5]  Simon C. Potter,et al.  Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis , 2011, Nature.

[6]  S. Kaveri,et al.  Physiological Induction of Regulatory Qa-1-Restricted CD8+ T Cells Triggered by Endogenous CD4+ T Cell Responses , 2011, PloS one.

[7]  V. Kuchroo,et al.  Gene-environment interaction in induction of autoimmunity. , 2011, Seminars in immunology.

[8]  A. Cooke,et al.  Infectious triggers protect from autoimmunity. , 2011, Seminars in immunology.

[9]  J. Ochoa-Repáraz,et al.  Gut, bugs, and brain: Role of commensal bacteria in the control of central nervous system disease , 2011, Annals of neurology.

[10]  S. Mazmanian,et al.  Proinflammatory T-cell responses to gut microbiota promote experimental autoimmune encephalomyelitis , 2010, Proceedings of the National Academy of Sciences.

[11]  S. Miller,et al.  Experimental Autoimmune Encephalomyelitis in the Mouse , 2007 .

[12]  Manuel A. Friese,et al.  From genes to function: the next challenge to understanding multiple sclerosis , 2009, Nature Reviews Immunology.

[13]  S. Sainsbury,et al.  T cell-mediated autoimmune disease due to low-affinity crossreactivity to common microbial peptides. , 2009, Immunity.

[14]  R. Ley,et al.  Innate immunity and intestinal microbiota in the development of Type 1 diabetes , 2008, Nature.

[15]  S. Mazmanian,et al.  An Immunomodulatory Molecule of Symbiotic Bacteria Directs Maturation of the Host Immune System , 2005, Cell.

[16]  R. Marrie,et al.  For Personal Use. Only Reproduce with Permission from Elsevier Ltd a Complex Disease Environmental Risk Factors in Multiple Sclerosis Aetiology , 2022 .

[17]  A. Macpherson,et al.  Interactions between commensal intestinal bacteria and the immune system , 2004, Nature Reviews Immunology.

[18]  R Balfour Sartor,et al.  Therapeutic manipulation of the enteric microflora in inflammatory bowel diseases: antibiotics, probiotics, and prebiotics. , 2004, Gastroenterology.

[19]  S. Miller,et al.  CD8-deficient SJL mice display enhanced susceptibility to Theiler’s virus infection and increased demyelinating pathology , 2001, Journal of NeuroVirology.

[20]  J. Goverman,et al.  A Pathogenic Role for Myelin-Specific Cd8+ T Cells in a Model for Multiple Sclerosis , 2001, The Journal of experimental medicine.

[21]  M. A. Curotto de Lafaille,et al.  Regulatory T cells in spontaneous autoimmune encephalomyelitis , 2001, Immunological reviews.

[22]  J. Gordon,et al.  Commensal Host-Bacterial Relationships in the Gut , 2001, Science.

[23]  H. Cantor,et al.  Suppression of immune responses by CD8 cells. II. Qa-1 on activated B cells stimulates CD8 cell suppression of T helper 2 responses. , 1998, Journal of immunology.

[24]  H. D. Liggitt,et al.  Triggers of autoimmune disease in a murine TCR-transgenic model for multiple sclerosis. , 1997, Journal of immunology.

[25]  Susumu Tonegawa,et al.  High incidence of spontaneous autoimmune encephalomyelitis in immunodeficient anti-myelin basic protein T cell receptor transgenic mice , 1994, Cell.

[26]  L. Hood,et al.  Transgenic mice that express a myelin basic protein-specific T cell receptor develop spontaneous autoimmunity , 1993, Cell.

[27]  Susumu Tonegawa,et al.  RAG-1-deficient mice have no mature B and T lymphocytes , 1992, Cell.