17q12‐21 amplicon, a novel recurrent genetic change in intestinal type of gastric carcinoma: A comparative genomic hybridization study
暂无分享,去创建一个
S. Knuutila | O. Monni | S. Nordling | A. Kokkola | P. Puolakkainen | E. Kivilaakso | M. Larramendy | M. Victorzon | R. Haapiainen
[1] B. Vogelstein,et al. Nomenclature: Vertebrate Mediators of TGFβ Family Signals , 1996, Cell.
[2] Irene L Andrulis,et al. MADR2 Maps to 18q21 and Encodes a TGFβ–Regulated MAD–Related Protein That Is Functionally Mutated in Colorectal Carcinoma , 1996, Cell.
[3] P. Meltzer,et al. Independent amplification and frequent co-amplification of three nonsyntenic regions on the long arm of chromosome 20 in human breast cancer. , 1996, Cancer research.
[4] P. Meltzer,et al. Hybrid selection of transcribed sequences from microdissected DNA: isolation of genes within amplified region at 20q11-q13.2 in breast cancer. , 1996, Cancer research.
[5] S. Knuutila,et al. DNA copy number losses in chromosome 14: an early change in gastrointestinal stromal tumors. , 1996, Cancer research.
[6] Scott E. Kern,et al. DPC4, A Candidate Tumor Suppressor Gene at Human Chromosome 18q21.1 , 1996, Science.
[7] H. Scherthan,et al. Advances in Brief Comparative Genomic in Situ Hybridization of Colon Carcinomas with Replication Error ' , 2006 .
[8] H. Yokozaki,et al. Frequent Amplification of the Cyclin E Gene in Human Gastric Carcinomas , 1995, Japanese journal of cancer research : Gann.
[9] E. Tahara. Molecular biology of gastric cancer , 1995, World journal of surgery.
[10] J Piper,et al. Optimizing comparative genomic hybridization for analysis of DNA sequence copy number changes in solid tumors , 1994, Genes, chromosomes & cancer.
[11] J Piper,et al. Detection and mapping of amplified DNA sequences in breast cancer by comparative genomic hybridization. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[12] M. Fukayama,et al. Molecular genetics for clinical management of colorectal carcinoma. 17p, 18q, and 22q loss of heterozygosity and decreased DCC expression are correlated with the metastatic potential , 1994, Cancer.
[13] S. Fushida,et al. Amplification of the c-erbB-2 gene in gastric carcinoma: correlation with survival. , 1993, Oncology.
[14] H. Kim,et al. Expression of cellular oncogenes in human gastric carcinoma: c‐myc, c–erb B2′ and c‐Ha‐ras , 1993, Journal of surgical oncology.
[15] H. Ito,et al. Frequent amplification of the c-met gene in scirrhous type stomach cancer. , 1992, Biochemical and biophysical research communications.
[16] S. Hirohashi,et al. Frequent loss of heterozygosity at the DCC locus in gastric cancer. , 1992, Cancer research.
[17] S. Rakic,et al. Serum gastrin levels in patients with intestinal and diffuse type of gastric cancer. , 1991, British Journal of Cancer.
[18] G. Friedman,et al. Helicobacter pylori infection and the risk of gastric carcinoma. , 1991, The New England journal of medicine.
[19] M. Blaser,et al. Helicobacter pylori infection and gastric carcinoma among Japanese Americans in Hawaii. , 1991, The New England journal of medicine.
[20] J. Rhim,et al. Amplification, overexpression, and rearrangement of the erbB-2 protooncogene in primary human stomach carcinomas. , 1989, Cancer research.
[21] S. Watson,et al. The in vitro growth response of primary human colorectal and gastric cancer cells to gastrin , 1989, International journal of cancer.
[22] J. Mecklin,et al. Carcinoma of the stomach and its heredity in young patients. , 1988, Scandinavian journal of gastroenterology.
[23] Shirley A. Miller,et al. A simple salting out procedure for extracting DNA from human nucleated cells. , 1988, Nucleic acids research.
[24] P. Laurén,et al. THE TWO HISTOLOGICAL MAIN TYPES OF GASTRIC CARCINOMA: DIFFUSE AND SO-CALLED INTESTINAL-TYPE CARCINOMA. AN ATTEMPT AT A HISTO-CLINICAL CLASSIFICATION. , 1965, Acta pathologica et microbiologica Scandinavica.