Prevalence and prognostic significance of KIT mutations in pediatric patients with core binding factor AML enrolled on serial pediatric cooperative trials for de novo AML.
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I. Bernstein | S. Raimondi | J. Radich | R. Arceci | N. Heerema | S. Meshinchi | T. Alonzo | R. Gerbing | W. Woods | M. Heinrich | B. Lange | Y. Ravindranath | B. Hirsch | Myron N. Chang | H. Weinstein | G. Dahl | N. Lacayo | Phoenix A Ho | C. Hurwitz | J. Pollard | D. Becton | P. Ho | R. Zeng
[1] P. Chevallier,et al. A phase II trial of high-dose imatinib mesylate for relapsed or refractory c-kit positive and Bcr-Abl negative acute myeloid leukaemia: the AFR-15 trial. , 2009, Leukemia research.
[2] S. Raimondi,et al. Prevalence and prognostic implications of CEBPA mutations in pediatric acute myeloid leukemia (AML): a report from the Children's Oncology Group. , 2009, Blood.
[3] G. Lyman,et al. Impact of disease risk on efficacy of matched related bone marrow transplantation for pediatric acute myeloid leukemia: the Children's Oncology Group. , 2008, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[4] S. Raimondi,et al. Prevalence and Prognostic Implications of WT1 Mutations in Pediatric AML Â: Report from Children’s Oncology Group , 2008 .
[5] C. Preudhomme,et al. Cooperating gene mutations in acute myeloid leukemia: a review of the literature , 2008, Leukemia.
[6] D. Liang,et al. Cooperating mutations of receptor tyrosine kinases and Ras genes in childhood core-binding factor acute myeloid leukemia and a comparative analysis on paired diagnosis and relapse samples , 2008, Leukemia.
[7] M. Gordon. Adverse Prognostic Significance of KIT Mutations in Adult Acute Myeloid Leukemia With inv(16) and t(8;21): A Cancer and Leukemia Group B Study , 2008 .
[8] R. Arceci,et al. The incidence and clinical significance of nucleophosmin mutations in childhood AML. , 2007, Blood.
[9] T. Taki,et al. N822 mutation of KIT gene was frequent in pediatric acute myeloid leukemia patients with t(8;21) in Japan: a study of the Japanese childhood AML cooperative study group , 2007, Leukemia.
[10] J. Radich,et al. Clinical implications of FLT3 mutations in pediatric AML. , 2006, Blood.
[11] I. Bernstein,et al. FLT3 internal tandem duplication in CD34+/CD33- precursors predicts poor outcome in acute myeloid leukemia. , 2006, Blood.
[12] C. Bloomfield,et al. Adverse prognostic significance of KIT mutations in adult acute myeloid leukemia with inv(16) and t(8;21): a Cancer and Leukemia Group B Study. , 2006, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[13] H. Dombret,et al. Incidence and prognostic impact of c-Kit, FLT3, and Ras gene mutations in core binding factor acute myeloid leukemia (CBF-AML) , 2006, Leukemia.
[14] Y. Hayashi,et al. KIT mutations, and not FLT3 internal tandem duplication, are strongly associated with a poor prognosis in pediatric acute myeloid leukemia with t(8;21): a study of the Japanese Childhood AML Cooperative Study Group. , 2006, Blood.
[15] W. Hiddemann,et al. 1 KIT-D 816 mutations in AML 1-ETO positive AML are associated with impaired event-free and overall survival , 2005 .
[16] T. Brümmendorf,et al. Prognostic impact of c-KIT mutations in core binding factor leukemias: an Italian retrospective study. , 2004, Blood.
[17] C. Bokemeyer,et al. Dasatinib (BMS-354825), a dual SRC/ABL kinase inhibitor, inhibits the kinase activity of wild-type, juxtamembrane, and activation loop mutant KIT isoforms associated with human malignancies. , 2006, Cancer research.
[18] S. Raimondi,et al. Pediatric Oncology Group (POG) studies of acute myeloid leukemia (AML): a review of four consecutive childhood AML trials conducted between 1981 and 2000 , 2005, Leukemia.
[19] S. Meshinchi,et al. Mutations in KIT and RAS are frequent events in pediatric core-binding factor acute myeloid leukemia , 2005, Leukemia.
[20] H. Mitsuya,et al. Imatinib mesylate for refractory acute myeloblastic leukemia harboring inv(16) and a C-KIT exon 8 mutation , 2005, Leukemia.
[21] H. Mitsuya,et al. Mutations in the receptor tyrosine kinase pathway are associated with clinical outcome in patients with acute myeloblastic leukemia harboring t(8;21)(q22;q22) , 2005, Leukemia.
[22] Doriano Fabbro,et al. Activation mutations of human c-KIT resistant to imatinib mesylate are sensitive to the tyrosine kinase inhibitor PKC412. , 2005, Blood.
[23] W. Hiddemann,et al. KIT exon 8 mutations associated with core-binding factor (CBF)-acute myeloid leukemia (AML) cause hyperactivation of the receptor in response to stem cell factor. , 2005, Blood.
[24] R. Cairoli,et al. Imatinib mesylate in the treatment of Core Binding Factor leukemias with KIT mutations. A report of three cases. , 2005, Leukemia research.
[25] Guoqiang Chen,et al. AML1-ETO and C-KIT mutation/overexpression in t(8;21) leukemia: implication in stepwise leukemogenesis and response to Gleevec. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[26] F. Galibert,et al. Localization of the human c-kit protooncogene on the q11–q12 region of chromosome 4 , 1988, Human Genetics.
[27] C. Harrison,et al. MRC trials in childhood acute myeloid leukaemia. , 2004, Annals of hematology.
[28] S. Feig,et al. Pilot study of idarubicin-based intensive-timing induction therapy for children with previously untreated acute myeloid leukemia: Children's Cancer Group Study 2941. , 2004, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[29] J. Reilly,et al. Incidence and prognosis of c‐KIT and FLT3 mutations in core binding factor (CBF) acute myeloid leukaemias , 2003, British journal of haematology.
[30] E Leonard,et al. Phosphatidylinositol 3 kinase contributes to the transformation of hematopoietic cells by the D816V c-Kit mutant. , 2001, Blood.
[31] R. Arceci,et al. STAT3 activation is required for Asp816 mutant c-Kit induced tumorigenicity , 2001, Oncogene.
[32] I. Bernstein,et al. Prevalence and prognostic significance of Flt3 internal tandem duplication in pediatric acute myeloid leukemia. , 2001, Blood.
[33] J. Buckley,et al. A comparison of allogeneic bone marrow transplantation, autologous bone marrow transplantation, and aggressive chemotherapy in children with acute myeloid leukemia in remission. , 2001, Blood.
[34] T. Naoe,et al. Prognostic implication of FLT3 and N-RAS gene mutations in acute myeloid leukemia. , 1999, Blood.
[35] K Wheatley,et al. The importance of diagnostic cytogenetics on outcome in AML: analysis of 1,612 patients entered into the MRC AML 10 trial. The Medical Research Council Adult and Children's Leukaemia Working Parties. , 1998, Blood.
[36] R. Gray. A Class of $K$-Sample Tests for Comparing the Cumulative Incidence of a Competing Risk , 1988 .
[37] W. Travis,et al. A Report of Three Cases , 1986 .
[38] E. Kaplan,et al. Nonparametric Estimation from Incomplete Observations , 1958 .
[39] H. B. Mann,et al. On a Test of Whether one of Two Random Variables is Stochastically Larger than the Other , 1947 .
[40] D.,et al. Regression Models and Life-Tables , 2022 .