Brain stress systems in the amygdala and addiction
暂无分享,去创建一个
[1] G. Koob. Neurobiological substrates for the dark side of compulsivity in addiction , 2009, Neuropharmacology.
[2] G. Koob,et al. PRECLINICAL STUDY: Corticotropin‐releasing factor‐1 receptor antagonists decrease heroin self‐administration in long‐ but not short‐access rats , 2009, Addiction biology.
[3] G. Koob,et al. The α1 adrenergic receptor antagonist prazosin reduces heroin self-administration in rats with extended access to heroin administration , 2009, Pharmacology Biochemistry and Behavior.
[4] G. Koob. A Role for Brain Stress Systems in Addiction , 2008, Neuron.
[5] G. Koob,et al. α1-Noradrenergic system role in increased motivation for cocaine intake in rats with prolonged access , 2008, European Neuropsychopharmacology.
[6] R. Rimondini,et al. PRECLINICAL STUDY: Long‐lasting tolerance to alcohol following a history of dependence , 2008, Addiction biology.
[7] G. Koob,et al. alpha1-noradrenergic receptor antagonism blocks dependence-induced increases in responding for ethanol. , 2008, Alcohol.
[8] G. Koob,et al. MPZP: A novel small molecule corticotropin-releasing factor type 1 receptor (CRF1) antagonist , 2008, Pharmacology Biochemistry and Behavior.
[9] P. Hipskind,et al. Upregulation of Voluntary Alcohol Intake, Behavioral Sensitivity to Stress, and Amygdala Crhr1 Expression Following a History of Dependence , 2008, Biological Psychiatry.
[10] M. Le Moal,et al. Addiction and the brain antireward system. , 2008, Annual review of psychology.
[11] G. Koob,et al. CRF(1) receptor antagonists attenuate escalated cocaine self-administration in rats. , 2008, Psychopharmacology.
[12] G. Koob,et al. CRF1 receptor antagonists attenuate escalated cocaine self-administration in rats , 2008, Psychopharmacology.
[13] L. Parsons,et al. CRF–CRF1 system activation mediates withdrawal-induced increases in nicotine self-administration in nicotine-dependent rats , 2007, Proceedings of the National Academy of Sciences.
[14] G. Koob,et al. A key role for corticotropin-releasing factor in alcohol dependence , 2007, Trends in Neurosciences.
[15] P. Hipskind,et al. 3-(4-Chloro-2-Morpholin-4-yl-Thiazol-5-yl)-8-(1-Ethylpropyl)-2,6-Dimethyl-Imidazo[1,2-b]Pyridazine: A Novel Brain-Penetrant, Orally Available Corticotropin-Releasing Factor Receptor 1 Antagonist with Efficacy in Animal Models of Alcoholism , 2007, The Journal of Neuroscience.
[16] G. Koob,et al. Corticotropin-Releasing Factor 1 Antagonists Selectively Reduce Ethanol Self-Administration in Ethanol-Dependent Rats , 2007, Biological Psychiatry.
[17] S. Matta,et al. Nicotinic activation of CRH neurons in extrahypothalamic regions of the rat brain , 1997, Endocrine.
[18] W. Vale,et al. The role of the hypothalamic-pituitary-adrenal axis in neuroendocrine responses to stress , 2006, Dialogues in clinical neuroscience.
[19] Antonio Alcaro,et al. Ethanol consumption and reward depend on norepinephrine in the prefrontal cortex , 2006, Neuroreport.
[20] G. Koob,et al. Corticotropin-Releasing Factor within the Central Nucleus of the Amygdala Mediates Enhanced Ethanol Self-Administration in Withdrawn, Ethanol-Dependent Rats , 2006, The Journal of Neuroscience.
[21] D. Winder,et al. Neuropeptide Y and corticotropin-releasing factor bi-directionally modulate inhibitory synaptic transmission in the bed nucleus of the stria terminalis , 2006, Neuropharmacology.
[22] Donald W. Pfaff,et al. Brain Arousal and Information Theory: Neural and Genetic Mechanisms , 2005 .
[23] G. Koob,et al. Plasticity of reward neurocircuitry and the 'dark side' of drug addiction , 2005, Nature Neuroscience.
[24] D. Overstreet,et al. Prior Multiple Ethanol Withdrawals Enhance Stress-Induced Anxiety-Like Behavior: Inhibition by CRF1- and Benzodiazepine-Receptor Antagonists and a 5-HT1a-Receptor Agonist , 2005, Neuropsychopharmacology.
[25] C. Ehlers,et al. Increased CRF-like and NPY-like immunoreactivity in adult rats exposed to nicotine during adolescence: Relation to anxiety-like and depressive-like behavior , 2005, Neuropeptides.
[26] H. Becker. Positive relationship between the number of prior ethanol withdrawal episodes and the severity of subsequent withdrawal seizures , 1994, Psychopharmacology.
[27] G. Koob,et al. The roles of urocortins 1, 2, and 3 in the brain , 2005 .
[28] G. Aston-Jones,et al. β-adrenergic antagonists attenuate withdrawal anxiety in cocaine-and morphine-dependent rats , 2005, Psychopharmacology.
[29] G. Koob,et al. CRF antagonist reverses the “anxiogenic” response to ethanol withdrawal in the rat , 2005, Psychopharmacology.
[30] G. Koob,et al. Buprenorphine and a CRF1 Antagonist Block the Acquisition of Opiate Withdrawal-Induced Conditioned Place Aversion in Rats , 2005, Neuropsychopharmacology.
[31] L. Parsons,et al. Increased GABA Release in the Central Amygdala of Ethanol-Dependent Rats , 2004, The Journal of Neuroscience.
[32] John T. Williams,et al. Noradrenaline Triggers GABAA Inhibition of Bed Nucleus of the Stria Terminalis Neurons Projecting to the Ventral Tegmental Area , 2004, The Journal of Neuroscience.
[33] G. Koob,et al. Increased anxiety-like behavior and ethanol self-administration in dependent rats: reversal via corticotropin-releasing factor-2 receptor activation. , 2004, Alcoholism, clinical and experimental research.
[34] S. D. Moore,et al. Ethanol Augments GABAergic Transmission in the Central Amygdala via CRF1 Receptors , 2004, Science.
[35] D. Overstreet,et al. Modulation of multiple ethanol withdrawal-induced anxiety-like behavior by CRF and CRF1 receptors , 2004, Pharmacology Biochemistry and Behavior.
[36] D. Overstreet,et al. SB242084, flumazenil, and CRA1000 block ethanol withdrawal-induced anxiety in rats. , 2004, Alcohol.
[37] W. Vale,et al. CRF and CRF receptors: role in stress responsivity and other behaviors. , 2004, Annual review of pharmacology and toxicology.
[38] T. Baker,et al. Potentiation of ethanol withdrawal by prior dependence , 1979, Psychopharmacology.
[39] G. Koob. Allostatic view of motivation: implications for psychopathology. , 2004, Nebraska Symposium on Motivation. Nebraska Symposium on Motivation.
[40] R. Hale,et al. Ethanol-induced locomotor stimulation in C57BL/6 mice following RO15-4513 administration , 2004, Psychopharmacology.
[41] B. Kissin,et al. Modifications in the response to alcohol following the establishment of physical dependence , 2004, Psychopharmacologia.
[42] R. Bevins,et al. Motivational factors in the etiology of drug abuse , 2004 .
[43] S. Matta,et al. Norepinephrine release in amygdala of rats during chronic nicotine self-administration: an in vivo microdialysis study , 2003, Neuropharmacology.
[44] S. File,et al. Corticotropin releasing factor antagonist, α-helical CRF9–41, reverses nicotine-induced conditioned, but not unconditioned, anxiety , 2003, Psychopharmacology.
[45] R. Hauger,et al. International Union of Pharmacology. XXXVI. Current Status of the Nomenclature for Receptors for Corticotropin-Releasing Factor and Their Ligands , 2003, Pharmacological Reviews.
[46] G. Koob,et al. Antagonism of corticotropin-releasing factor attenuates the enhanced responsiveness to stress observed during protracted ethanol abstinence. , 2003, Alcohol.
[47] J. Pastor,et al. [Neurobiology of addiction to drugs of abuse]. , 2003, Revista de neurologia.
[48] Y. Shaham,et al. The reinstatement model of drug relapse: history, methodology and major findings , 2003, Psychopharmacology.
[49] F. Weiss,et al. Additive Effect of Stress and Drug Cues on Reinstatement of Ethanol Seeking: Exacerbation by History of Dependence and Role of Concurrent Activation of Corticotropin-Releasing Factor and Opioid Mechanisms , 2002, The Journal of Neuroscience.
[50] R. Jaenisch,et al. Brain-Derived Neurotrophic Factor Is Essential for Opiate-Induced Plasticity of Noradrenergic Neurons , 2002, The Journal of Neuroscience.
[51] C. Hodge,et al. Elevated extracellular CRF levels in the bed nucleus of the stria terminalis during ethanol withdrawal and reduction by subsequent ethanol intake , 2002, Pharmacology Biochemistry and Behavior.
[52] J. Vaughan,et al. Urocortin III-Immunoreactive Projections in Rat Brain: Partial Overlap with Sites of Type 2 Corticotrophin-Releasing Factor Receptor Expression , 2002, The Journal of Neuroscience.
[53] R. Rimondini,et al. Long‐lasting increase in voluntary ethanol consumption and transcriptional regulation in the rat brain after intermittent exposure to alcohol , 2002, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[54] S. Matta,et al. Norepinephrine Secretion in the Hypothalamic Paraventricular Nucleus of Rats during Unlimited Access to Self-Administered Nicotine: An In Vivo Microdialysis Study , 2001, The Journal of Neuroscience.
[55] M. Wilson,et al. Neurotrophin-3 modulates noradrenergic neuron function and opiate withdrawal , 2001, Molecular Psychiatry.
[56] C. Donaldson,et al. Identification of urocortin III, an additional member of the corticotropin-releasing factor (CRF) family with high affinity for the CRF2 receptor , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[57] L. Parsons,et al. Compulsive Drug‐Seeking Behavior and Relapse , 2001 .
[58] J. Vaughan,et al. Urocortin II: A member of the corticotropin-releasing factor (CRF) neuropeptide family that is selectively bound by type 2 CRF receptors , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[59] G. Koob,et al. Drug Addiction, Dysregulation of Reward, and Allostasis , 2001, Neuropsychopharmacology.
[60] Vanya Qui ones-Jenab. The biological basis of cocaine addiction , 2001 .
[61] C. Heyser,et al. Excessive Ethanol Drinking Following a History of Dependence: Animal Model of Allostasis , 2000, Neuropsychopharmacology.
[62] Nicole C. Rust,et al. Ethanol-Associated Behaviors of Mice Lacking Norepinephrine , 2000, The Journal of Neuroscience.
[63] G. Aston-Jones,et al. Noradrenaline in the ventral forebrain is critical for opiate withdrawal-induced aversion , 2000, Nature.
[64] G. Koob,et al. Corticotropin-releasing factor, norepinephrine, and stress , 1999, Biological Psychiatry.
[65] H. Akil,et al. Distinct neurochemical populations in the rat central nucleus of the amygdala and bed nucleus of the stria terminalis: Evidence for their selective activation by interleukin‐1β , 1999, The Journal of comparative neurology.
[66] G. Koob,et al. Corticotropin-releasing factor antagonist attenuates the “anxiogenic-like” effect in the defensive burying paradigm but not in the elevated plus-maze following chronic cocaine in rats , 1999, Psychopharmacology.
[67] F Weiss,et al. In vivo CRF release in rat amygdala is increased during cocaine withdrawal in self‐administering rats , 1999, Synapse.
[68] G. Koob,et al. Clonidine Blocks Acquisition But not Expression of Conditioned Opiate Withdrawal in Rats , 1998, Neuropsychopharmacology.
[69] E. V. Van Bockstaele,et al. Amygdaloid Corticotropin‐Releasing Factor Targets Locus Coeruleus Dendrites: Substrate for the Co‐ordination of Emotional and Cognitive Limbs of the Stress Response , 1998, Journal of neuroendocrinology.
[70] H Selye,et al. A syndrome produced by diverse nocuous agents. 1936. , 1998, The Journal of neuropsychiatry and clinical neurosciences.
[71] H. Annis,et al. Gender in relation to relapse crisis situations, coping, and outcome among treated alcoholics. , 1998, Addictive behaviors.
[72] B. Kobilka,et al. Insights from in vivo modification of adrenergic receptor gene expression. , 1998, Annual review of pharmacology and toxicology.
[73] G F Koob,et al. Drug abuse: hedonic homeostatic dysregulation. , 1997, Science.
[74] G. Koob,et al. Activation of corticotropin-releasing factor in the limbic system during cannabinoid withdrawal. , 1997, Science.
[75] R. Stout,et al. Replication and extension of Marlatt's taxonomy of relapse precipitants: overview of procedures and results. The Relapse Research Group. , 1996, Addiction.
[76] S. Maisto,et al. Relapse research and the Reasons for Drinking Questionnaire: a factor analysis of Marlatt's relapse taxonomy. , 1996, Addiction.
[77] G. Koob,et al. Increase of extracellular corticotropin-releasing factor-like immunoreactivity levels in the amygdala of awake rats during restraint stress and ethanol withdrawal as measured by microdialysis , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[78] Z. Sarnyai,et al. Brain corticotropin-releasing factor mediates ‘anxiety-like’ behavior induced by cocaine withdrawal in rats , 1995, Brain Research.
[79] G. Koob,et al. Suppression of corticotropin‐releasing factor in the amygdala attenuates aversive consequences of morphine withdrawal , 1995, Behavioural pharmacology.
[80] G. Aghajanian,et al. Molecular and cellular mechanisms of opiate action: Studies in the rat locus coeruleus , 1994, Brain Research Bulletin.
[81] B. Mason,et al. A Double‐Blind, Placebo‐Controlled Pilot Study to Evaluate the Efficacy and Safety of Oral Nalmefene HCI for Alcohol Dependence , 1994 .
[82] S. Foote,et al. The Locus Coeruleus as a Site for Integrating Corticotropin‐Releasing Factor and Noradrenergic Mediation of Stress Responses a , 1993, Annals of the New York Academy of Sciences.
[83] S. Matta,et al. Detection by in vivo microdialysis of nicotine-induced norepinephrine secretion from the hypothalamic paraventricular nucleus of freely moving rats: dose-dependency and desensitization. , 1993, Endocrinology.
[84] M. Cassell,et al. Intrinsic GABAergic neurons in the rat central extended amygdala , 1993, The Journal of comparative neurology.
[85] G. Koob,et al. Microinjection of a corticotropin-releasing factor antagonist into the central nucleus of the amygdala reverses anxiogenic-like effects of ethanol withdrawal , 1993, Brain Research.
[86] M. Page,et al. Activation of noradrenergic locus coeruleus neurons by hemodynamic stress is due to local release of corticotropin-releasing factor , 1991, Brain Research.
[87] P. Kalivas,et al. The basal forebrain : anatomy to function , 1991 .
[88] L. Heimer,et al. Piecing together the puzzle of basal forebrain anatomy. , 1991, Advances in experimental medicine and biology.
[89] G. Koob,et al. Nucleus accumbens and amygdala are possible substrates for the aversive stimulus effects of opiate withdrawal , 1990, Neuroscience.
[90] L. Heimer,et al. New perspectives in basal forebrain organization of special relevance for neuropsychiatric disorders: The striatopallidal, amygdaloid, and corticopetal components of substantia innominata , 1988, Neuroscience.
[91] J. T. Reason,et al. Handbook of Life Stress, Cognition and Health , 1988 .
[92] P. Sterling,et al. Allostasis: A new paradigm to explain arousal pathology. , 1988 .
[93] I. Ferrier,et al. Distribution of corticotropin-releasing factor-like immunoreactivity in human brain , 1987, Neuropeptides.
[94] I. Assenmacher,et al. Immunocytochemical evidence for stimulatory control by the ventral noradrenergic bundle of parvocellular neurons of the paraventricular nucleus secreting corticotropin releasing hormone and vasopressin in rats , 1986, Brain Research.
[95] S. Roelofs. Hyperventilation, anxiety, craving for alcohol: a subacute alcohol withdrawal syndrome. , 1985, Alcohol.
[96] P. Cushman,et al. Alcohol withdrawal syndromes: clinical management with lofexidine. , 1985, Alcoholism, clinical and experimental research.
[97] S. Otsuki,et al. Cerebrospinal fluid monoamine metabolites during alcohol withdrawal syndrome and recovered state. , 1983, Biological psychiatry.
[98] G. Sedvall,et al. Clinical conditions and concentrations of MOPEG in the cerebrospinal fluid and urine of male alcoholic patients during withdrawal. , 1983, Alcoholism, clinical and experimental research.
[99] L. Swanson,et al. Organization of ovine corticotropin-releasing factor immunoreactive cells and fibers in the rat brain: an immunohistochemical study. , 1983, Neuroendocrinology.
[100] C. Alling,et al. Studies on duration of a late recovery period after chronic abuse of ethanol , 1982, Acta psychiatrica Scandinavica.
[101] G. Sedvall,et al. Central norepinephrine metabolism during alcohol intoxication in addicts and healthy volunteers. , 1981, Science.
[102] S. Burchfield. The Stress Response: A New Perspective , 1979, Psychosomatic medicine.
[103] T. E. Werner,et al. Reinforcement with intragastric infusions of ethanol: Blocking effect of FLA 57 , 1979, Pharmacology Biochemistry and Behavior.
[104] S. T. Mason,et al. Noradrenaline and ethanol intake in the rat , 1979, Neuroscience Letters.
[105] S. Ogren,et al. Noradrenergic mediation of the positive reinforcing properties of ethanol: I. Suppression of ethanol consumption in laboratory rats following dopamine-beta-hydroxylase inhibition. , 1977, Archives internationales de pharmacodynamie et de therapie.
[106] Z. Amit,et al. The effects of selective catecholamine depletions by 6-hydroxydopamine on ethanol preference in rats , 1977, Neuroscience Letters.
[107] H I Hershon,et al. Alcohol withdrawal symptoms and drinking behavior. , 1977, Journal of studies on alcohol.
[108] H. Selye. A Syndrome produced by Diverse Nocuous Agents , 1936, Nature.
[109] J. Johnston. Further contributions to the study of the evolution of the forebrain , 1923 .