Abnormal sensitivity of cannabinoid CB1 receptors in the striatum of mice with experimental amyotrophic lateral sclerosis
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D. Centonze | G. Bernardi | N. Mercuri | S. Rossi | M. Cozzolino | M. Carrì | M. Maccarrone | A. Musella | V. De Chiara
[1] Qing-song Liu,et al. D2 Dopamine Receptor Activation Facilitates Endocannabinoid-Mediated Long-Term Synaptic Depression of GABAergic Synaptic Transmission in Midbrain Dopamine Neurons via cAMP-Protein Kinase A Signaling , 2008, The Journal of Neuroscience.
[2] T. Montine,et al. The prostaglandin E2 EP2 receptor accelerates disease progression and inflammation in a model of amyotrophic lateral sclerosis , 2008, Annals of neurology.
[3] D. Centonze,et al. Chronic Psychoemotional Stress Impairs Cannabinoid-Receptor-Mediated Control of GABA Transmission in the Striatum , 2008, The Journal of Neuroscience.
[4] G. Marsicano,et al. Cannabinoids enhance susceptibility of immature brain to ethanol neurotoxicity , 2008, Annals of neurology.
[5] E. Beattie,et al. Altered presymptomatic AMPA and cannabinoid receptor trafficking in motor neurons of ALS model mice: implications for excitotoxicity , 2008, The European journal of neuroscience.
[6] M. Cozzolino,et al. Cysteine 111 Affects Aggregation and Cytotoxicity of Mutant Cu,Zn-superoxide Dismutase Associated with Familial Amyotrophic Lateral Sclerosis* , 2008, Journal of Biological Chemistry.
[7] Mauro Maccarrone,et al. The endocannabinoid system is dysregulated in multiple sclerosis and in experimental autoimmune encephalomyelitis. , 2007, Brain : a journal of neurology.
[8] M. S. Petrik,et al. Magnetic Resonance Microscopy and Immunohistochemistry of the CNS of the Mutant SOD Murine Model of ALS Reveals Widespread Neural Deficits , 2007, NeuroMolecular Medicine.
[9] A. Gowran,et al. The tumour suppressor protein, p53, is involved in the activation of the apoptotic cascade by Delta9-tetrahydrocannabinol in cultured cortical neurons. , 2007, European journal of pharmacology.
[10] D. Centonze,et al. Chronic cocaine sensitizes striatal GABAergic synapses to the stimulation of cannabinoid CB1 receptors , 2007, The European journal of neuroscience.
[11] Kristen K. Ade,et al. Anandamide Regulates Postnatal Development of Long-Term Synaptic Plasticity in the Rat Dorsolateral Striatum , 2007, The Journal of Neuroscience.
[12] S. Thayer,et al. Δ9-Tetrahydrocannabinol protects hippocampal neurons from excitotoxicity , 2007, Brain Research.
[13] J. Crow,et al. The CB2 cannabinoid agonist AM‐1241 prolongs survival in a transgenic mouse model of amyotrophic lateral sclerosis when initiated at symptom onset , 2006, Journal of neurochemistry.
[14] Tiziana Bisogno,et al. Depolarization‐induced retrograde synaptic inhibition in the mouse cerebellar cortex is mediated by 2‐arachidonoylglycerol , 2006, The Journal of physiology.
[15] D. Cleveland,et al. ALS: A Disease of Motor Neurons and Their Nonneuronal Neighbors , 2006, Neuron.
[16] L. Greensmith,et al. Increasing cannabinoid levels by pharmacological and genetic manipulation delays disease progression in SOD1 mice , 2006, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[17] B. Seilheimer,et al. Glutamate AMPA receptors change in motor neurons of SOD1G93A transgenic mice and their inhibition by a noncompetitive antagonist ameliorates the progression of amytrophic lateral sclerosis‐like disease , 2006, Journal of neuroscience research.
[18] Linda Greensmith,et al. Amyotrophic lateral sclerosis: recent advances and future therapies , 2005, Current opinion in neurology.
[19] P. Greengard,et al. Cannabinoid Action Depends on Phosphorylation of Dopamine- and cAMP-Regulated Phosphoprotein of 32 kDa at the Protein Kinase A Site in Striatal Projection Neurons , 2005, The Journal of Neuroscience.
[20] Mauro Maccarrone,et al. Abnormal Sensitivity to Cannabinoid Receptor Stimulation Might Contribute to Altered Gamma-Aminobutyric Acid Transmission in the Striatum of R6/2 Huntington’s Disease Mice , 2005, Biological Psychiatry.
[21] T. Möller,et al. Cannabinol delays symptom onset in SOD1 (G93A) transgenic mice without affecting survival , 2005, Amyotrophic lateral sclerosis and other motor neuron disorders : official publication of the World Federation of Neurology, Research Group on Motor Neuron Diseases.
[22] Y. Kawahara,et al. Excitotoxicity and ALS: What is unique about the AMPA receptors expressed on spinal motor neurons? , 2005, Amyotrophic lateral sclerosis and other motor neuron disorders : official publication of the World Federation of Neurology, Research Group on Motor Neuron Diseases.
[23] P. Calabresi,et al. A Critical Interaction between Dopamine D2 Receptors and Endocannabinoids Mediates the Effects of Cocaine on Striatal GABAergic Transmission , 2004, Neuropsychopharmacology.
[24] L. Bruijn,et al. Unraveling the mechanisms involved in motor neuron degeneration in ALS. , 2004, Annual review of neuroscience.
[25] M. Kliot,et al. Endocannabinoids accumulate in spinal cord of SOD1G93A transgenic mice , 2004, Journal of neurochemistry.
[26] S. McAllister,et al. Amyotrophic lateral sclerosis: delayed disease progression in mice by treatment with a cannabinoid , 2004, Amyotrophic lateral sclerosis and other motor neuron disorders : official publication of the World Federation of Neurology, Research Group on Motor Neuron Diseases.
[27] G. Bernardi,et al. α-amino-3-hydroxy-5-methyl-isoxazole-4-propionate receptors in spinal cord motor neurons are altered in transgenic mice overexpressing human Cu,Zn superoxide dismutase (Gly93→Ala) mutation , 2003, Neuroscience.
[28] D. Piomelli. The molecular logic of endocannabinoid signalling , 2003, Nature Reviews Neuroscience.
[29] M. Eder,et al. CB1 Cannabinoid Receptors and On-Demand Defense Against Excitotoxicity , 2003, Science.
[30] A. Thompson,et al. Cannabinoids inhibit neurodegeneration in models of multiple sclerosis. , 2003, Brain : a journal of neurology.
[31] V. Campbell,et al. Tetrahydrocannabinol‐induced neurotoxicity depends on CB1 receptor‐mediated c‐Jun N‐terminal kinase activation in cultured cortical neurons , 2003, British journal of pharmacology.
[32] G. Bernardi,et al. Altered long-term corticostriatal synaptic plasticity in transgenic mice overexpressing human CU/ZN superoxide dismutase (GLY93→ALA) mutation , 2003, Neuroscience.
[33] E. Del Giudice,et al. Cannabinoids ablate release of TNFα in rat microglial cells stimulated with lypopolysaccharide , 2003, Glia.
[34] P. Mcgeer,et al. Inflammatory processes in amyotrophic lateral sclerosis , 2002, Muscle & nerve.
[35] P. Calabresi,et al. Experimental Parkinsonism Alters Endocannabinoid Degradation: Implications for Striatal Glutamatergic Transmission , 2002, The Journal of Neuroscience.
[36] L. Garcia-Segura,et al. Blockade of cannabinoid CB1 receptor function protects against in vivo disseminating brain damage following NMDA‐induced excitotoxicity , 2002, Journal of neurochemistry.
[37] K. Nicolay,et al. Exogenous Anandamide Protects Rat Brain against Acute Neuronal Injury In Vivo , 2001, The Journal of Neuroscience.
[38] D. Rokicki,et al. Locomotor activity and a conditioned fear response: correlation with cortical and subcortical binding of the dopamine D1 receptor antagonist [3H]SCH 23390 , 2000, Neuroreport.
[39] W. Robberecht,et al. Ca2+-permeable AMPA receptors and selective vulnerability of motor neurons , 2000, Journal of the Neurological Sciences.
[40] M. Swash,et al. Extrapyramidal involvement in amyotrophic lateral sclerosis: backward falls and retropulsion , 1999, Journal of neurology, neurosurgery, and psychiatry.
[41] K. Jin,et al. Cannabinoids and Neuroprotection in Global and Focal Cerebral Ischemia and in Neuronal Cultures , 1999, The Journal of Neuroscience.
[42] D. Figlewicz,et al. Glutamate Potentiates the Toxicity of Mutant Cu/Zn-Superoxide Dismutase in Motor Neurons by Postsynaptic Calcium-Dependent Mechanisms , 1998, The Journal of Neuroscience.
[43] M. Shen,et al. Cannabinoid receptor agonists protect cultured rat hippocampal neurons from excitotoxicity. , 1998, Molecular pharmacology.
[44] J. Powell,et al. Mutations in all five exons of SOD‐1 may cause ALS , 1998, Annals of neurology.
[45] J. Trojanowski,et al. Neurofilaments and Orthograde Transport Are Reduced in Ventral Root Axons of Transgenic Mice that Express Human SOD1 with a G93A Mutation , 1997, The Journal of cell biology.
[46] A. Levey,et al. Selective loss of glial glutamate transporter GLT‐1 in amyotrophic lateral sclerosis , 1995, Annals of neurology.
[47] D. Borchelt,et al. Superoxide dismutase is an abundant component in cell bodies, dendrites, and axons of motor neurons and in a subset of other neurons. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[48] M. Gurney,et al. Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation. , 1994, Science.
[49] B. Snow,et al. Evidence for a dopaminergic deficit in sporadic amyotrophic lateral sclerosis on positron emission scanning , 1993, The Lancet.
[50] J. Haines,et al. Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis , 1993, Nature.
[51] S. Thayer,et al. Delta9-tetrahydrocannabinol protects hippocampal neurons from excitotoxicity. , 2007, Brain research.
[52] D. Lovinger,et al. CB1 cannabinoid receptor inhibits synaptic release of glutamate in rat dorsolateral striatum. , 2001, Journal of neurophysiology.
[53] G. Cabral,et al. Cannabinoids inhibit LPS‐inducible cytokine mRNA expression in rat microglial cells , 2000, Glia.
[54] A. F. Soleng,et al. A thorny question: how does activity maintain dendritic spines? , 1999, Nature Neuroscience.