Endothelium and the lipid metabolism: the current understanding.
暂无分享,去创建一个
[1] J. Liao,et al. Endothelial function and oxidative stress. , 2004, Endothelium : journal of endothelial cell research.
[2] C. Stefanadis,et al. Vasomotor effects of L- and D-arginine in stenotic atheromatous coronary plaque , 2001, Heart.
[3] C. Napoli,et al. Involvement of oxidation-sensitive mechanisms in the cardiovascular effects of hypercholesterolemia. , 2001, Mayo Clinic proceedings.
[4] C. Napoli,et al. Nitric oxide and atherosclerosis. , 2001, Nitric oxide : biology and chemistry.
[5] C. Stefanadis,et al. Therapeutic modification of the L-arginine-eNOS pathway in cardiovascular diseases. , 2001, Atherosclerosis.
[6] G. Schuler,et al. Exercise Training in Coronary Artery Disease and Coronary Vasomotion , 2001, Circulation.
[7] C. Stefanadis,et al. L-Arginine in coronary atherosclerosis. , 2000, International journal of cardiology.
[8] E. Feskens,et al. Arginine Intake and Risk of Coronary Heart Disease Mortality in Elderly Men , 2000, Arteriosclerosis, thrombosis, and vascular biology.
[9] G. Schuler,et al. Effect of exercise on coronary endothelial function in patients with coronary artery disease. , 2000, The New England journal of medicine.
[10] R. Vogel. Cholesterol lowering and endothelial function. , 1999, The American journal of medicine.
[11] G. Kojda,et al. Interactions between NO and reactive oxygen species: pathophysiological importance in atherosclerosis, hypertension, diabetes and heart failure. , 1999, Cardiovascular research.
[12] W. Graier,et al. Selective stimulation of L-arginine uptake contributes to shear stress-induced formation of nitric oxide. , 1999, Life sciences.
[13] M. Grigioni,et al. ET(A)/ET(B) receptor antagonist bosentan inhibits neointimal development in collared carotid arteries of rabbits. , 1998, Life sciences.
[14] M. Corretti,et al. Cholesterol, cholesterol lowering, and endothelial function. , 1998, Progress in cardiovascular diseases.
[15] R. Cannon. Role of nitric oxide in cardiovascular disease: focus on the endothelium. , 1998, Clinical chemistry.
[16] R. Schmieder,et al. Increased bioavailability of nitric oxide after lipid-lowering therapy in hypercholesterolemic patients: a randomized, placebo-controlled, double-blind study. , 1998, Circulation.
[17] H. Kosaka,et al. Role of endogenous endothelin in the development of graft arteriosclerosis in rat cardiac allografts: antiproliferative effects of bosentan, a nonselective endothelin receptor antagonist. , 1998, Circulation.
[18] G. Plotnick,et al. The mechanism of improvement in endothelial function by pravastatin: direct effect or through cholesterol lowering , 1998 .
[19] A. Baron,et al. Endothelial dysfunction is associated with cholesterol levels in the high normal range in humans. , 1997, Circulation.
[20] K. Schenck-Gustafsson,et al. Transient triglyceridemia decreases vascular reactivity in young, healthy men without risk factors for coronary heart disease. , 1997, Circulation.
[21] M. Creager,et al. When 'normal' cholesterol levels injure the endothelium. , 1997, Circulation.
[22] D. Harrison,et al. Cellular and molecular mechanisms of endothelial cell dysfunction. , 1997, The Journal of clinical investigation.
[23] W. Erl,et al. HMG-CoA reductase inhibitors decrease CD11b expression and CD11b-dependent adhesion of monocytes to endothelium and reduce increased adhesiveness of monocytes isolated from patients with hypercholesterolemia. , 1997, Journal of the American College of Cardiology.
[24] A. Quyyumi,et al. Effect of L-arginine on human coronary endothelium-dependent and physiologic vasodilation. , 1997, Journal of the American College of Cardiology.
[25] T. Lüscher,et al. Biology of the Endothelium , 1997, Clinical cardiology.
[26] E. Whitney,et al. Effect of gemfibrozil +/- niacin +/- cholestyramine on endothelial function in patients with serum low-density lipoprotein cholesterol levels <160 mg/dl and high-density lipoprotein cholesterol levels <40 mg/dl. , 1997, The American journal of cardiology.
[27] R. Schwartz,et al. Enhanced endothelin-mediated coronary vasoconstriction and attenuated basal nitric oxide activity in experimental hypercholesterolemia. , 1997, Circulation.
[28] B. Hornig,et al. Vitamin C improves endothelial dysfunction of epicardial coronary arteries in hypertensive patients. , 1997, Circulation.
[29] P. Marraccini,et al. Plasma cholesterol regulates soluble cell adhesion molecule expression in familial hypercholesterolemia. , 1997, Circulation.
[30] D. Celermajer,et al. Endothelial dysfunction: does it matter? Is it reversible? , 1997, Journal of the American College of Cardiology.
[31] T. Saldeen,et al. Preservation of endogenous antioxidant activity and inhibition of lipid peroxidation as common mechanisms of antiatherosclerotic effects of vitamin E, lovastatin and amlodipine. , 1997, Journal of the American College of Cardiology.
[32] J. Jukema,et al. Functional evaluation of lipid-lowering therapy by pravastatin in the Regression Growth Evaluation Statin Study (REGRESS) , 1997, Circulation.
[33] T. Lehtimäki,et al. In vivo low density lipoprotein oxidation relates to coronary reactivity in young men. , 1997, Journal of the American College of Cardiology.
[34] R. Vogel. Coronary risk factors, endothelial function, and atherosclerosis: A review , 1997, Clinical cardiology.
[35] G. Assmann,et al. Low-density lipoproteins inhibit the Na+/H+ antiport in human platelets. A novel mechanism enhancing platelet activity in hypercholesterolemia. , 1997, Circulation.
[36] D. Green,et al. Simvastatin, an HMG-coenzyme A reductase inhibitor, improves endothelial function within 1 month. , 1997, Circulation.
[37] G. Plotnick,et al. Effect of a single high-fat meal on endothelial function in healthy subjects. , 1997, The American journal of cardiology.
[38] P. Ganz,et al. Effect of cholesterol reduction on myocardial ischemia in patients with coronary disease. , 1997, Circulation.
[39] T. Imaizumi,et al. Single LDL apheresis improves endothelium-dependent vasodilatation in hypercholesterolemic humans. , 1997, Circulation.
[40] P. Vanhoutte,et al. Endothelium-derived hyperpolarizing factor. , 1996, Clinical and experimental pharmacology & physiology.
[41] M. Petch,et al. Aging-associated endothelial dysfunction in humans is reversed by L-arginine. , 1996, Journal of the American College of Cardiology.
[42] H. Superko,et al. Beyond LDL cholesterol reduction. , 1996, Circulation.
[43] H. Crijns,et al. Reduction of transient myocardial ischemia with pravastatin in addition to the conventional treatment in patients with angina pectoris. REGRESS Study Group. , 1996, Circulation.
[44] A. Yeung,et al. Endothelium-dependent coronary vasomotion relates to the susceptibility of LDL to oxidation in humans. , 1996, Circulation.
[45] J. Mehta,et al. Oxidized LDL decreases L-arginine uptake and nitric oxide synthase protein expression in human platelets: relevance of the effect of oxidized LDL on platelet function. , 1996, Circulation.
[46] R. Virmani,et al. Coronary plaque erosion without rupture into a lipid core. A frequent cause of coronary thrombosis in sudden coronary death. , 1996, Circulation.
[47] C. Ballantyne,et al. Levels of Soluble Cell Adhesion Molecules in Patients With Dyslipidemia , 1996 .
[48] P. Ganz,et al. Atherosclerosis: risk factors and the vascular endothelium. , 1996, American heart journal.
[49] M. Hori,et al. Endothelial dysfunction in the early stage of atherosclerosis precedes appearance of intimal lesions assessable with intravascular ultrasound. , 1996, American heart journal.
[50] D. Waters,et al. Hyperlipidemia and coronary disease. Correction of the increased thrombogenic potential with cholesterol reduction. , 1995, Circulation.
[51] A. Quyyumi,et al. Evidence that endothelial dysfunction in patients with hypercholesterolemia is not due to increased extracellular nitric oxide breakdown by superoxide anions. , 1995, The American journal of cardiology.
[52] O. Hess,et al. Exercise-induced vasomotion of angiographically normal and stenotic coronary arteries improves after cholesterol-lowering drug therapy with bezafibrate. , 1995, Journal of the American College of Cardiology.
[53] N. Mullani,et al. Changes in myocardial perfusion abnormalities by positron emission tomography after long-term, intense risk factor modification. , 1995, JAMA.
[54] C. Wanner,et al. Impairment of endothelium-dependent dilation in rabbit renal arteries by oxidized lipoprotein(a). Role of oxygen-derived radicals. , 1995, Circulation.
[55] T. Rabelink,et al. Vascular function in the forearm of hypercholesterolaemic patients off and on lipid-lowering medication , 1995, The Lancet.
[56] V. Fuster,et al. Coronary plaque disruption. , 1995, Circulation.
[57] A. Heagerty,et al. In vitro responses of human peripheral small arteries in hypercholesterolemia and effects of therapy. , 1995, Circulation.
[58] P. Libby. Molecular bases of the acute coronary syndromes. , 1995, Circulation.
[59] W. Weintraub,et al. Beneficial effects of cholesterol-lowering therapy on the coronary endothelium in patients with coronary artery disease. , 1995, The New England journal of medicine.
[60] G. Levine,et al. Cholesterol reduction in cardiovascular disease. Clinical benefits and possible mechanisms. , 1995, The New England journal of medicine.
[61] A. Yeung,et al. The effect of cholesterol-lowering and antioxidant therapy on endothelium-dependent coronary vasomotion. , 1995, The New England journal of medicine.
[62] A. Quyyumi,et al. Effect of antioxidant vitamins on low density lipoprotein oxidation and impaired endothelium-dependent vasodilation in patients with hypercholesterolemia. , 1994, Journal of the American College of Cardiology.
[63] D. Celermajer,et al. Endothelium-dependent dilation in the systemic arteries of asymptomatic subjects relates to coronary risk factors and their interaction. , 1994, Journal of the American College of Cardiology.
[64] S. Nilsson,et al. The effect of probucol on femoral atherosclerosis: the Probucol Quantitative Regression Swedish Trial (PQRST). , 1994, The American journal of cardiology.
[65] V. Maher,et al. Reversal of coronary heart disease by lipid-lowering therapy. Observations and pathological mechanisms. , 1994, Circulation.
[66] M. Sugimachi,et al. Reduction in serum cholesterol with pravastatin improves endothelium-dependent coronary vasomotion in patients with hypercholesterolemia. , 1994, Circulation.
[67] G. Gibbons,et al. The emerging concept of vascular remodeling. , 1994, The New England journal of medicine.
[68] A. Quyyumi,et al. Investigation of decreased availability of nitric oxide precursor as the mechanism responsible for impaired endothelium-dependent vasodilation in hypercholesterolemic patients. , 1994, Journal of the American College of Cardiology.
[69] S. Moncada,et al. The L-arginine-nitric oxide pathway. , 1993, The New England journal of medicine.
[70] A. Quyyumi,et al. The Role of Nitric Oxide in Endothelium‐Dependent Vasodilation of Hypercholesterolemic Patients , 1993, Circulation.
[71] D. Waters,et al. Plasma Lipoproteins and Progression of Coronary Artery Disease Evaluated by Angiography and Clinical Events , 1993, Circulation.
[72] O. Hess,et al. Influence of Serum Cholesterol and Other Coronary Risk Factors on Vasomotion of Angiographically Normal Coronary Arteries , 1993, Circulation.
[73] C. Lau,et al. Beneficial effect of cholesterol-lowering therapy on coronary endothelium-dependent relaxation in hypercholesterolaemic patients , 1993, The Lancet.
[74] D. Harrison,et al. Hypercholesterolemia increases endothelial superoxide anion production. , 1993, The Journal of clinical investigation.
[75] J. Albers,et al. Lipid Lowering and Plaque Regression New Insights Into Prevention of Plaque Disruption and Clinical Events in Coronary Disease , 1993, Circulation.
[76] K. Hirata,et al. High density lipoprotein reverses inhibitory effect of oxidized low density lipoprotein on endothelium-dependent arterial relaxation. , 1993, Circulation research.
[77] A. Quyyumi,et al. Role of Endothelium‐Derived Nitric Oxide in the Abnormal Endothelium‐Dependent Vascular Relaxation of Patients With Essential Hypertension , 1993, Circulation.
[78] J. Cooke,et al. Vascular Biology and Medicine in the 1990s: Scope, Concepts, Potentials, and Perspectives , 1993, Circulation.
[79] J. Cooke,et al. L-arginine improves endothelium-dependent vasodilation in hypercholesterolemic humans. , 1992, The Journal of clinical investigation.
[80] H. Drexler,et al. Correction of endothelial dysfunction in coronary microcirculation of hypercholesterolaemic patients by L-arginine , 1991, The Lancet.
[81] E. Mohler,et al. Effects of high-density lipoprotein on acetylcholine-induced coronary vasoreactivity. , 1991, The American journal of cardiology.
[82] A. Lerman,et al. Circulating and tissue endothelin immunoreactivity in advanced atherosclerosis. , 1991, The New England journal of medicine.
[83] R. Lawn,et al. Lipoprotein(a) and atherosclerosis. , 1991, Annals of internal medicine.
[84] K. Margulies,et al. Endothelin: a new cardiovascular regulatory peptide. , 1990, Mayo Clinic proceedings.
[85] M J Davies,et al. A macro and micro view of coronary vascular insult in ischemic heart disease. , 1990, Circulation.
[86] M. Cybulsky,et al. Endothelial‐Dependent Mechanisms of Leukocyte Adhesion in Inflammation and Atherosclerosis a , 1990, Annals of the New York Academy of Sciences.
[87] R. Cohen,et al. Oxidized low density lipoproteins cause contraction and inhibit endothelium-dependent relaxation in the pig coronary artery. , 1990, The Journal of clinical investigation.
[88] K. Kugiyama,et al. Impairment of endothelium-dependent arterial relaxation by lysolecithin in modified low-density lipoproteins , 1990, Nature.
[89] A. Yeung,et al. Coronary vasomotor response to acetylcholine relates to risk factors for coronary artery disease. , 1990, Circulation.
[90] J. Breslow,et al. Lipoprotein(a) modulation of endothelial cell surface fibrinolysis and its potential role in atherosclerosis , 1989, Nature.
[91] H. Shimokawa,et al. Hypercholesterolemia causes generalized impairment of endothelium-dependent relaxation to aggregating platelets in porcine arteries. , 1989, Journal of the American College of Cardiology.
[92] J L Witztum,et al. Beyond cholesterol. Modifications of low-density lipoprotein that increase its atherogenicity. , 1989, The New England journal of medicine.
[93] E. Nabel,et al. Evidence of endothelial dysfunction in angiographically normal coronary arteries of patients with coronary artery disease. , 1989, Circulation.
[94] A. M. Lefer,et al. Cardiovascular effects of acute hypercholesterolemia in rabbits. Reversal with lovastatin treatment. , 1989, The Journal of clinical investigation.
[95] P. Ganz,et al. Paradoxical vasoconstriction induced by acetylcholine in atherosclerotic coronary arteries. , 1986, The New England journal of medicine.
[96] M. Gimbrone,et al. Influence of hemodynamic forces on vascular endothelial function. In vitro studies of shear stress and pinocytosis in bovine aortic cells. , 1984, The Journal of clinical investigation.
[97] R. Furchgott,et al. The obligatory role of endothelial cells in the relaxation of arterial smooth muscle by acetylcholine , 1980, Nature.
[98] W. Sterling Edwards,et al. Blood Vessels , 1959 .
[99] T. Lüscher,et al. Endothelin in atherosclerosis: importance of risk factors and therapeutic implications. , 2000, Journal of cardiovascular pharmacology.
[100] H. Drexler,et al. Endothelial Dysfunction in Hypercholesterolemia: Mechanisms, Pathophysiological Importance, and Therapeutic Interventions , 2000, Seminars in thrombosis and hemostasis.
[101] G. Plotnick,et al. Changes in flow-mediated brachial artery vasoactivity with lowering of desirable cholesterol levels in healthy middle-aged men. , 1996, The American journal of cardiology.
[102] T. Lüscher,et al. Endothelium-Derived Nitric Oxide, Endothelin, and Platelet Vessel Wall Interaction: Alterations in Hypercholesterolemia and Atherosclerosis , 1993, Seminars in thrombosis and hemostasis.
[103] B. Hoffman,et al. Inactivation of endothelial derived relaxing factor by oxidized lipoproteins. , 1992, The Journal of clinical investigation.
[104] S. Mocanda. THE L-ARGININE-NITRIC OXIDE PATHWAY , 1992 .