Corrigendum to “Long-term valproic acid exposure increases the number of neocortical neurons in the developing rat brain” [Neurosci. Lett. 580 (2014) 12–16] A possible new animal model of autism
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[1] H. Markram,et al. General developmental health in the VPA-rat model of autism , 2013, Front. Behav. Neurosci..
[2] I. Chiu,et al. The mood stabilizer valproate activates human FGF1 gene promoter through inhibiting HDAC and GSK‐3 activities , 2013, Journal of neurochemistry.
[3] Mogens Vestergaard,et al. Prenatal valproate exposure and risk of autism spectrum disorders and childhood autism. , 2013, JAMA.
[4] J. Foster,et al. In utero exposure to valproic acid and autism--a current review of clinical and animal studies. , 2013, Neurotoxicology and teratology.
[5] K. Meador,et al. Fetal antiepileptic drug exposure and cognitive outcomes at age 6 years (NEAD study): a prospective observational study , 2013, The Lancet Neurology.
[6] T. Chomiak,et al. Alterations of neocortical development and maturation in autism: insight from valproic acid exposure and animal models of autism. , 2013, Neurotoxicology and teratology.
[7] Marta García-Fiñana,et al. The prevalence of neurodevelopmental disorders in children prenatally exposed to antiepileptic drugs , 2013, Journal of Neurology, Neurosurgery & Psychiatry.
[8] Se Jin Jeon,et al. Prenatal exposure to valproic acid increases the neural progenitor cell pool and induces macrocephaly in rat brain via a mechanism involving the GSK-3β/β-catenin pathway , 2012, Neuropharmacology.
[9] W. Hauser,et al. Comparative safety of antiepileptic drugs during pregnancy , 2012, Neurology.
[10] Kristopher T Kahle,et al. The GABA Excitatory/Inhibitory Shift in Brain Maturation and Neurological Disorders , 2012, The Neuroscientist : a review journal bringing neurobiology, neurology and psychiatry.
[11] T. Tomson,et al. Dose-dependent risk of malformations with antiepileptic drugs: an analysis of data from the EURAP epilepsy and pregnancy registry , 2011, The Lancet Neurology.
[12] J. Piven,et al. Early brain overgrowth in autism associated with an increase in cortical surface area before age 2 years. , 2011, Archives of general psychiatry.
[13] D. Guilloteau,et al. Fetal exposure to teratogens: Evidence of genes involved in autism , 2011, Neuroscience & Biobehavioral Reviews.
[14] Kwang Ho Ko,et al. The critical period of valproate exposure to induce autistic symptoms in Sprague-Dawley rats. , 2011, Toxicology letters.
[15] S. Essock,et al. Valproate prescription prevalence among women of childbearing age. , 2011, Psychiatric services.
[16] B. Hu,et al. Altering the trajectory of early postnatal cortical development can lead to structural and behavioural features of autism , 2010, BMC Neuroscience.
[17] Yoshio Imura,et al. Nonexploratory movement and behavioral alterations in a thalidomide or valproic acid-induced autism model rat , 2010, Neuroscience Research.
[18] A. Ornoy. Valproic acid in pregnancy: how much are we endangering the embryo and fetus? , 2009, Reproductive toxicology.
[19] H. Markram,et al. Hyper-Connectivity and Hyper-Plasticity in the Medial Prefrontal Cortex in the Valproic Acid Animal Model of Autism , 2008, Frontiers in neural circuits.
[20] Henry Markram,et al. The Intense World Syndrome – an Alternative Hypothesis for Autism , 2007, Front. Neurosci..
[21] Yehezkel Ben-Ari,et al. Fetal Exposure to GABA‐Acting Antiepileptic Drugs Generates Hippocampal and Cortical Dysplasias , 2007, Epilepsia.
[22] Ben S. Wittner,et al. Association of valproate‐induced teratogenesis with histone deacetylase inhibition in vivo , 2005, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[23] M. Narita,et al. Maternal administration of thalidomide or valproic acid causes abnormal serotonergic neurons in the offspring: implication for pathogenesis of autism , 2005, International Journal of Developmental Neuroscience.
[24] Eric Courchesne,et al. Brain overgrowth in autism during a critical time in development: implications for frontal pyramidal neuron and interneuron development and connectivity , 2005, International Journal of Developmental Neuroscience.
[25] P. Rodier,et al. The teratology of autism , 2005, International Journal of Developmental Neuroscience.
[26] D. O’Carroll,et al. Essential function of histone deacetylase 1 in proliferation control and CDK inhibitor repression , 2002, The EMBO journal.
[27] Ping Zhu,et al. Valproic acid defines a novel class of HDAC inhibitors inducing differentiation of transformed cells , 2001, The EMBO journal.
[28] J R Nyengaard,et al. Tissue shrinkage and unbiased stereological estimation of particle number and size * , 2001, Journal of microscopy.
[29] M. Guenther,et al. Histone Deacetylase Is a Direct Target of Valproic Acid, a Potent Anticonvulsant, Mood Stabilizer, and Teratogen* , 2001, The Journal of Biological Chemistry.
[30] R. Przewłocki,et al. Nociceptive changes in rats after prenatal exposure to valproic acid. , 2001, Polish journal of pharmacology.
[31] Ruth A. Carper,et al. Unusual brain growth patterns in early life in patients with autistic disorder , 2001, Neurology.
[32] B. Kerr,et al. Fetal valproate syndrome and autism: additional evidence of an association , 2001, Developmental medicine and child neurology.
[33] P. Rodier,et al. Prenatal exposure of rats to valproic acid reproduces the cerebellar anomalies associated with autism. , 2000, Neurotoxicology and teratology.
[34] P. Rodier,et al. Linking etiologies in humans and animal models: studies of autism. , 1997, Reproductive toxicology.
[35] P. Rodier,et al. Embryological origin for autism: Developmental anomalies of the cranial nerve motor nuclei , 1996, The Journal of comparative neurology.
[36] J. Kromberg,et al. Fetal Valproate Syndrome: Clinical and Neuro‐developmental Features in Two Sibling Pairs , 1994 .
[37] C. Gillberg,et al. AUTISM IN THALIDOMIDE EMBRYOPATHY: A POPULATION STUDY , 1994, Developmental medicine and child neurology.
[38] C. Vorhees,et al. Teratogenicity and developmental toxicity of valproic acid in rats. , 1987, Teratology.
[39] C. Curry,et al. The fetal valproate syndrome. , 1984, American journal of medical genetics.
[40] J. Petrere,et al. Teratogenesis of calcium valproate in rats. , 1983, Fundamental and applied toxicology : official journal of the Society of Toxicology.
[41] A. Czeizel,et al. VALPROIC ACID AND SPINA BIFIDA , 1982, The Lancet.
[42] H. Nau,et al. A new model for embryotoxicity testing: teratogenicity and pharmacokinetics of valproic acid following constant-rate administration in the mouse using human therapeutic drug and metabolite concentrations. , 1981, Life sciences.
[43] Evans Da. Letter: Coeliac disease and HL-A8. , 1973 .
[44] Ryszard Przewłocki,et al. Behavioral Alterations in Rats Prenatally Exposed to Valproic Acid: Animal Model of Autism , 2005, Neuropsychopharmacology.
[45] C. Vorhees,et al. Behavioral teratogenicity of valproic acid: selective effects on behavior after prenatal exposure to rats , 2004, Psychopharmacology.
[46] B. Kerr,et al. Fetal valproate syndrome and autism , 2001 .
[47] O. Ladefoged,et al. Total number and mean cell volume of neocortical neurons in rats exposed to 2,5-hexanedione with and without acetone. , 1991, Neurotoxicology and teratology.
[48] J. Piven,et al. Magnetic resonance imaging and head circumference study of brain size in autism: birth through age 2 years. , 2005, Archives of general psychiatry.