Association between TP53 and p21 genetic polymorphisms and acute side effects of radiotherapy in breast cancer patients

[1]  J. Chang-Claude,et al.  Association between Polymorphisms in the DNA Repair Genes, XRCC1, APE1, and XPD and Acute Side Effects of Radiotherapy in Breast Cancer Patients , 2005, Clinical Cancer Research.

[2]  M. O’Reilly,et al.  p53 Modulates Radiation Sensitivity Independent of p21 Transcriptional Activation , 2005, American journal of clinical oncology.

[3]  Ming-Tsang Wu,et al.  p53 Codon 72 and p21 codon 31 polymorphisms in prostate cancer. , 2004, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.

[4]  N. Park,et al.  p53 and p21 genetic polymorphisms and susceptibility to endometrial cancer. , 2004, Gynecologic oncology.

[5]  T. Crook,et al.  Polymorphism in wild-type p53 modulates response to chemotherapy in vitro and in vivo , 2004, Oncogene.

[6]  J. Goldblum,et al.  Dual effect of p53 on radiation sensitivity in vivo: p53 promotes hematopoietic injury, but protects from gastro-intestinal syndrome in mice , 2004, Oncogene.

[7]  E. Guinó,et al.  A TP53 polymorphism is associated with increased risk of colorectal cancer and with reduced levels of TP53 mRNA , 2004, Oncogene.

[8]  D. Pim,et al.  p53 polymorphic variants at codon 72 exert different effects on cell cycle progression , 2004, International journal of cancer.

[9]  Jang-Ming Lee,et al.  Association between p21 codon 31 polymorphism and esophageal cancer risk in a Taiwanese population. , 2003, Cancer letters.

[10]  J. Chang-Claude,et al.  Personal characteristics, therapy modalities and individual DNA repair capacity as predictive factors of acute skin toxicity in an unselected cohort of breast cancer patients receiving radiotherapy. , 2003, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.

[11]  Peter Donnelly,et al.  A comparison of bayesian methods for haplotype reconstruction from population genotype data. , 2003, American journal of human genetics.

[12]  J. Hopewell,et al.  Time factor for acute tissue reactions following fractionated irradiation: a balance between repopulation and enhanced radiosensitivity , 2003, International journal of radiation biology.

[13]  W. Dörr Modulation of repopulation processes in oral mucosa: experimental results , 2003, International journal of radiation biology.

[14]  J. Chang-Claude,et al.  Radiation-induced DNA damage and repair in lymphocytes from breast cancer patients and their correlation with acute skin reactions to radiotherapy. , 2003, International journal of radiation oncology, biology, physics.

[15]  M. Murphy,et al.  The codon 72 polymorphic variants of p53 have markedly different apoptotic potential , 2003, Nature Genetics.

[16]  R. Schneider-Stock,et al.  Homozygous proline at codon 72 of p53 as a potential risk factor favoring the development of undifferentiated thyroid carcinoma. , 2002, International journal of oncology.

[17]  M. Spitz,et al.  p53 Genotypes and Haplotypes Associated With Lung Cancer Susceptibility and Ethnicity. , 2002, Journal of the National Cancer Institute.

[18]  Shan Wang-Gohrke,et al.  Intron 3 16 bp duplication polymorphism of p53 is associated with an increased risk for breast cancer by the age of 50 years. , 2002, Pharmacogenetics.

[19]  E. Berns,et al.  Associations between common polymorphisms in TP53 and p21WAF1/Cip1 and phenotypic features of breast cancer. , 2002, Carcinogenesis.

[20]  M. Wolff,et al.  Waf-1 (p21) and p53 polymorphisms in breast cancer. , 2002, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.

[21]  N. Park,et al.  Polymorphisms in codon 31 of p21 and cervical cancer susceptibility in Korean women. , 2001, Cancer letters.

[22]  Miranda Thomas,et al.  Two Polymorphic Variants of Wild-Type p53 Differ Biochemically and Biologically , 1999, Molecular and Cellular Biology.

[23]  T. Hideshima,et al.  p53 mutations predict non-small cell lung carcinoma response to radiotherapy. , 1999, Cancer letters.

[24]  T. Rebbeck,et al.  p53 germline polymorphisms are associated with an increased risk for breast cancer in German women. , 1998, Anticancer research.

[25]  Y Taya,et al.  Enhanced phosphorylation of p53 by ATM in response to DNA damage. , 1998, Science.

[26]  C. Bellamy,et al.  p53 and apoptosis. , 1997, British medical bulletin.

[27]  A. Levine,et al.  Identification of a novel p53 functional domain that is necessary for efficient growth suppression. , 1996, Proceedings of the National Academy of Sciences of the United States of America.

[28]  M. Oren,et al.  p53 in growth control and neoplasia. , 1996, Biochimica et biophysica acta.

[29]  C C Ling,et al.  Radiation-induced apoptosis: relevance to radiotherapy. , 1995, International journal of radiation oncology, biology, physics.

[30]  James Brugarolas,et al.  Radiation-induced cell cycle arrest compromised by p21 deficiency , 1995, Nature.

[31]  D. Givol,et al.  A single nucleotide substitution at codon 31 (Ser/Arg) defines a polymorphism in a highly conserved region of the p53-inducible gene WAF1/CIP1. , 1994, Oncogene.

[32]  Jurg Ott,et al.  Handbook of Human Genetic Linkage , 1994 .

[33]  Yi-Song Wang,et al.  WAF1/CIP1 is induced in p53-mediated G1 arrest and apoptosis. , 1994, Cancer research.

[34]  B S Weir,et al.  Independence tests for VNTR alleles defined as quantile bins. , 1993, American journal of human genetics.

[35]  V. Lazar,et al.  Simple sequence repeat polymorphism within the p53 gene. , 1993, Oncogene.

[36]  S. Bentzen,et al.  Relationship between early and late normal-tissue injury after postmastectomy radiotherapy. , 1991, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.