Complement cascade gene expression defines novel prognostic subgroups of acute myeloid leukemia.
暂无分享,去创建一个
W. Hiddemann | K. Eppert | S. Bohlander | K. Spiekermann | J. Rak | W. Berdel | B. Wörmann | T. Herold | I. Laverdière | Meaghan Boileau | Kolja Eppert | Isabelle Laverdière
[1] E. Gottlin,et al. Complement Factor H Antibodies from Lung Cancer Patients Induce Complement-Dependent Lysis of Tumor Cells, Suggesting a Novel Immunotherapeutic Strategy , 2015, Cancer Immunology Research.
[2] S. Mamidi,et al. Neutralization of membrane complement regulators improves complement-dependent effector functions of therapeutic anticancer antibodies targeting leukemic cells , 2015, Oncoimmunology.
[3] Ulrich Mansmann,et al. Isolated trisomy 13 defines a homogeneous AML subgroup with high frequency of mutations in spliceosome genes and poor prognosis. , 2014, Blood.
[4] B. Wollenberg,et al. The complement receptors CD46, CD55 and CD59 are regulated by the tumour microenvironment of head and neck cancer to facilitate escape of complement attack. , 2014, European journal of cancer.
[5] K. Baggerly,et al. Autocrine Effects of Tumor-Derived Complement , 2014, Cell reports.
[6] Susumu Goto,et al. Data, information, knowledge and principle: back to metabolism in KEGG , 2013, Nucleic Acids Res..
[7] Ulrich Mansmann,et al. Identification of a 24-gene prognostic signature that improves the European LeukemiaNet risk classification of acute myeloid leukemia: an international collaborative study. , 2013, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[8] Angelo J. Canty,et al. Stem cell gene expression programs influence clinical outcome in human leukemia , 2011, Nature Medicine.
[9] John D Lambris,et al. Complement: a key system for immune surveillance and homeostasis , 2010, Nature Immunology.
[10] Bob Löwenberg,et al. Review Articles (434 articles) , 2008 .
[11] John D Lambris,et al. Is complement good or bad for cancer patients? A new perspective on an old dilemma. , 2009, Trends in immunology.
[12] M. Ratajczak,et al. DEFECTIVE ENGRAFTMENT OF C3aR-/- HEMATOPOIETIC STEM PROGENITOR CELLS REVEALS A NOVEL ROLE OF THE C3a-C3aR AXIS IN BONE MARROW HOMING , 2009, Leukemia.
[13] Bas J. Wouters,et al. Prediction of molecular subtypes in acute myeloid leukemia based on gene expression profiling , 2009, Haematologica.
[14] J. Itskovitz‐Eldor,et al. CD133‐Positive Hematopoietic Stem Cell “Stemness” Genes Contain Many Genes Mutated or Abnormally Expressed in Leukemia , 2005, Stem cells.
[15] Gerard Brady,et al. Routine expression profiling of microarray gene signatures in acute leukaemia by real‐time PCR of human bone marrow * , 2005, British journal of haematology.
[16] John D Lambris,et al. Functional receptor for C3a anaphylatoxin is expressed by normal hematopoietic stem/progenitor cells, and C3a enhances their homing-related responses to SDF-1. , 2003, Blood.
[17] S. Meri,et al. Secretion of soluble complement inhibitors factor H and factor H-like protein (FHL-1) by ovarian tumour cells , 2002, British Journal of Cancer.
[18] E. Kitano,et al. Synthesis of factor D by gastric cancer-derived cell lines. , 2002, International immunopharmacology.