Association between 9p21.3 genomic markers and coronary artery disease in East Asians: a meta-analysis involving 9,813 cases and 10,710 controls
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Wei Li | Yang Wang | Tao Chen | Jin Guo | Zhen-qiang Wu | Xiao-ru Cheng
[1] X. Chu,et al. Replication of Putative Susceptibility Loci from Genome-Wide Association Studies Associated with Coronary Atherosclerosis in Chinese Han Population , 2011, PloS one.
[2] D. Absher,et al. Design of the Coronary ARtery DIsease Genome-Wide Replication And Meta-Analysis (CARDIoGRAM) Study: A Genome-Wide Association Meta-analysis Involving More Than 22 000 Cases and 60 000 Controls , 2010, Circulation. Cardiovascular genetics.
[3] G. Palomaki,et al. Association between 9p21 genomic markers and heart disease: a meta-analysis. , 2010, JAMA.
[4] Xin Yang,et al. Three SNPs on chromosome 9p21 confer increased risk of myocardial infarction in Chinese subjects. , 2009, Atherosclerosis.
[5] R. Hui,et al. 9p21 is a Shared Susceptibility Locus Strongly for Coronary Artery Disease and Weakly for Ischemic Stroke in Chinese Han Population , 2009, Circulation. Cardiovascular genetics.
[6] E. Boerwinkle,et al. Impact of Adding a Single Allele in the 9p21 Locus to Traditional Risk Factors on Reclassification of Coronary Heart Disease Risk and Implications for Lipid-Modifying Therapy in the Atherosclerosis Risk in Communities Study , 2009, Circulation. Cardiovascular genetics.
[7] S. Yusuf,et al. Potentially modifiable risk factors associated with myocardial infarction in China: the INTERHEART China study , 2009, Heart.
[8] Yu-yu Yao,et al. A common variant on chromosome 9p21 affects the risk of early-onset coronary artery disease , 2009, Molecular Biology Reports.
[9] L. Hedges,et al. Introduction to Meta‐Analysis , 2009, International Coaching Psychology Review.
[10] B. Horne,et al. Genetic variation at the 9p21 locus predicts angiographic coronary artery disease prevalence but not extent and has clinical utility. , 2008, American heart journal.
[11] F. Hu,et al. Associations Between Single Nucleotide Polymorphisms on Chromosome 9p21 and Risk of Coronary Heart Disease in Chinese Han Population , 2008, Arteriosclerosis, thrombosis, and vascular biology.
[12] Y. Kokubo,et al. Validation of the association of genetic variants on chromosome 9p21 and 1q41 with myocardial infarction in a Japanese population. , 2008, Circulation journal : official journal of the Japanese Circulation Society.
[13] Ludwig A Hothorn,et al. Data Supplement (unedited) at: , 2007 .
[14] T. Nakajima,et al. Replication of the association between a chromosome 9p21 polymorphism and coronary artery disease in Japanese and Korean populations , 2008, Journal of Human Genetics.
[15] Shaoqi Rao,et al. Four SNPs on Chromosome 9p21 in a South Korean Population Implicate a Genetic Locus That Confers High Cross-Race Risk for Development of Coronary Artery Disease , 2007, Arteriosclerosis, thrombosis, and vascular biology.
[16] A. Gylfason,et al. A Common Variant on Chromosome 9p21 Affects the Risk of Myocardial Infarction , 2007, Science.
[17] G. Abecasis,et al. A Genome-Wide Association Study of Type 2 Diabetes in Finns Detects Multiple Susceptibility Variants , 2007, Science.
[18] M. McCarthy,et al. Replication of Genome-Wide Association Signals in UK Samples Reveals Risk Loci for Type 2 Diabetes , 2007, Science.
[19] Y. Kokubo,et al. Validation of the association between the gene encoding proteasome subunit alpha type 6 and myocardial infarction in a Japanese population. , 2007, Circulation journal : official journal of the Japanese Circulation Society.
[20] Jonathan C. Cohen,et al. A Common Allele on Chromosome 9 Associated with Coronary Heart Disease , 2007, Science.
[21] D. Levy,et al. Multiple biomarkers for the prediction of first major cardiovascular events and death. , 2006, The New England journal of medicine.
[22] J. van der Lei,et al. Identification of the four conventional cardiovascular disease risk factors by Dutch general practitioners. , 2005, Chest.
[23] F. Hobbs. Cardiovascular disease: different strategies for primary and secondary prevention? , 2004, Heart.
[24] N. Kalinina,et al. Smad Expression in Human Atherosclerotic Lesions Evidence for Impaired TGF-β/Smad Signaling in Smooth Muscle Cells of Fibrofatty Lesions , 2004, Arteriosclerosis, thrombosis, and vascular biology.
[25] S. Lowe,et al. Tumor suppression by Ink4a-Arf: progress and puzzles. , 2003, Current opinion in genetics & development.
[26] Gregory J. Hannon,et al. pl5INK4B is a potentia| effector of TGF-β-induced cell cycle arrest , 1994, Nature.
[27] Qi Zhang,et al. Polymorphism on chromosome 9p21.3 contributes to early-onset and severity of coronary artery disease in non-diabetic and type 2 diabetic patients. , 2011, Chinese medical journal.
[28] Qi Zhang,et al. Chromosome 9p21 polymorphism is associated with myocardial infarction but not with clinical outcome in Han Chinese , 2009, Clinical chemistry and laboratory medicine.