Muscle insulin sensitivity and glucose metabolism are controlled by the intrinsic muscle clock

[1]  H. Himmelbauer,et al.  Conventional knockout of Tbc1d1 in mice impairs insulin- and AICAR-stimulated glucose uptake in skeletal muscle. , 2013, Endocrinology.

[2]  C. Johnson,et al.  Circadian Disruption Leads to Insulin Resistance and Obesity , 2013, Current Biology.

[3]  G. FitzGerald,et al.  Obesity in mice with adipocyte-specific deletion of clock component Arntl , 2012, Nature Medicine.

[4]  J. Ferrières,et al.  Shiftwork and Higher Pancreatic Secretion: Early Detection of an Intermediate State of Insulin Resistance? , 2012, Chronobiology international.

[5]  A. Deshmukh,et al.  The Rab-GTPase-activating protein TBC1D1 regulates skeletal muscle glucose metabolism. , 2012, American journal of physiology. Endocrinology and metabolism.

[6]  Pierre Baldi,et al.  CircadiOmics: integrating circadian genomics, transcriptomics, proteomics and metabolomics , 2012, Nature Methods.

[7]  J. Bogan Regulation of glucose transporter translocation in health and diabetes. , 2012, Annual review of biochemistry.

[8]  P. Neufer,et al.  Lipid-induced mitochondrial stress and insulin action in muscle. , 2012, Cell metabolism.

[9]  E. Ravussin,et al.  Muscle-specific deletion of carnitine acetyltransferase compromises glucose tolerance and metabolic flexibility. , 2012, Cell metabolism.

[10]  G. Cartee,et al.  A Novel Method to Measure Glucose Uptake and Myosin Heavy Chain Isoform Expression of Single Fibers From Rat Skeletal Muscle , 2012, Diabetes.

[11]  S. Shea,et al.  Adverse Metabolic Consequences in Humans of Prolonged Sleep Restriction Combined with Circadian Disruption , 2012, Science Translational Medicine.

[12]  Pierre Baldi,et al.  Coordination of the transcriptome and metabolome by the circadian clock , 2012, Proceedings of the National Academy of Sciences.

[13]  R. Evans,et al.  Cryptochromes mediate rhythmic repression of the glucocorticoid receptor , 2011, Nature.

[14]  K. Petersen,et al.  Reversal of muscle insulin resistance with exercise reduces postprandial hepatic de novo lipogenesis in insulin resistant individuals , 2011, Proceedings of the National Academy of Sciences.

[15]  Kathryn Moynihan Ramsey,et al.  Circadian rhythms, sleep, and metabolism. , 2011, The Journal of clinical investigation.

[16]  Ueli Schibler,et al.  Crosstalk between components of circadian and metabolic cycles in mammals. , 2011, Cell metabolism.

[17]  Felix Naef,et al.  Genome-Wide and Phase-Specific DNA-Binding Rhythms of BMAL1 Control Circadian Output Functions in Mouse Liver , 2011, PLoS biology.

[18]  D. Hardie,et al.  PGC-1alpha increases PDH content but does not change acute PDH regulation in mouse skeletal muscle. , 2010, American journal of physiology. Regulatory, integrative and comparative physiology.

[19]  Kenneth S Campbell,et al.  CLOCK and BMAL1 regulate MyoD and are necessary for maintenance of skeletal muscle phenotype and function , 2010, Proceedings of the National Academy of Sciences.

[20]  H. Pilegaard,et al.  Lipid-Induced Insulin Resistance Affects Women Less Than Men and Is Not Accompanied by Inflammation or Impaired Proximal Insulin Signaling , 2010, Diabetes.

[21]  Joseph S. Takahashi,et al.  Temperature as a Universal Resetting Cue for Mammalian Circadian Oscillators , 2010, Science.

[22]  Karl Kornacker,et al.  JTK_CYCLE: An Efficient Nonparametric Algorithm for Detecting Rhythmic Components in Genome-Scale Data Sets , 2010, Journal of biological rhythms.

[23]  A. Kalsbeek,et al.  The hypothalamic clock and its control of glucose homeostasis , 2010, Trends in Endocrinology & Metabolism.

[24]  K. Petersen,et al.  Lipid-induced insulin resistance: unravelling the mechanism , 2010, The Lancet.

[25]  Joseph S. Takahashi,et al.  Disruption of the Clock Components CLOCK and BMAL1 Leads to Hypoinsulinemia and Diabetes , 2010, Nature.

[26]  N. Fujii,et al.  TBC1D1 Regulates Insulin- and Contraction-Induced Glucose Transport in Mouse Skeletal Muscle , 2010, Diabetes.

[27]  Akiko Hida,et al.  Circadian Clock Gene Bmal1 Is Not Essential; Functional Replacement with its Paralog, Bmal2 , 2010, Current Biology.

[28]  U. Schibler,et al.  The mammalian circadian timing system: organization and coordination of central and peripheral clocks. , 2010, Annual review of physiology.

[29]  C. Reggiani,et al.  Inducible activation of Akt increases skeletal muscle mass and force without satellite cell activation , 2009, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[30]  A. Bonen,et al.  Restoring AS160 phosphorylation rescues skeletal muscle insulin resistance and fatty acid oxidation while not reducing intramuscular lipids. , 2009, American journal of physiology. Endocrinology and metabolism.

[31]  Louis Hue,et al.  The Randle cycle revisited: a new head for an old hat. , 2009, American journal of physiology. Endocrinology and metabolism.

[32]  N. Turner,et al.  Lipid and insulin infusion-induced skeletal muscle insulin resistance is likely due to metabolic feedback and not changes in IRS-1, Akt, or AS160 phosphorylation. , 2009, American journal of physiology. Endocrinology and metabolism.

[33]  Rob C Hoeben,et al.  Tbc1d1 mutation in lean mouse strain confers leanness and protects from diet-induced obesity , 2008, Nature Genetics.

[34]  Yuting Liu,et al.  Analysis of Gene Regulatory Networks in the Mammalian Circadian Rhythm , 2008, PLoS Comput. Biol..

[35]  Hiroki R Ueda,et al.  Analysis and synthesis of high-amplitude Cis-elements in the mammalian circadian clock , 2008, Proceedings of the National Academy of Sciences.

[36]  Kai-Florian Storch,et al.  Physiological significance of a peripheral tissue circadian clock , 2008, Proceedings of the National Academy of Sciences.

[37]  Joseph S. Takahashi,et al.  The Meter of Metabolism , 2008, Cell.

[38]  F. Fleury-Olela,et al.  Differential display of DNA-binding proteins reveals heat-shock factor 1 as a circadian transcription factor. , 2008, Genes & development.

[39]  Erin L. McDearmon,et al.  Identification of the circadian transcriptome in adult mouse skeletal muscle. , 2007, Physiological genomics.

[40]  Kai-Florian Storch,et al.  Intrinsic Circadian Clock of the Mammalian Retina: Importance for Retinal Processing of Visual Information , 2007, Cell.

[41]  Ueli Schibler,et al.  System-Driven and Oscillator-Dependent Circadian Transcription in Mice with a Conditionally Active Liver Clock , 2007, PLoS biology.

[42]  Erin L. McDearmon,et al.  Dissecting the Functions of the Mammalian Clock Protein BMAL1 by Tissue-Specific Rescue in Mice , 2006, Science.

[43]  P. Neufer,et al.  PDH-E1alpha dephosphorylation and activation in human skeletal muscle during exercise: effect of intralipid infusion. , 2006, Diabetes.

[44]  N. Fujii,et al.  AS160 Regulates Insulin- and Contraction-stimulated Glucose Uptake in Mouse Skeletal Muscle* , 2006, Journal of Biological Chemistry.

[45]  Robert A. Harris,et al.  Role of pyruvate dehydrogenase kinase isoenzyme 4 (PDHK4) in glucose homoeostasis during starvation. , 2006, The Biochemical journal.

[46]  M. Antoch,et al.  Early aging and age-related pathologies in mice deficient in BMAL1, the core componentof the circadian clock. , 2006, Genes & development.

[47]  Sanda Šulić,et al.  Inactivation of S6 ribosomal protein gene in T lymphocytes activates a p53-dependent checkpoint response. , 2005, Genes & development.

[48]  R. Myers,et al.  Evolving gene/transcript definitions significantly alter the interpretation of GeneChip data , 2005, Nucleic acids research.

[49]  Pablo Tamayo,et al.  Gene set enrichment analysis: A knowledge-based approach for interpreting genome-wide expression profiles , 2005, Proceedings of the National Academy of Sciences of the United States of America.

[50]  Kei Sakamoto,et al.  Deficiency of LKB1 in skeletal muscle prevents AMPK activation and glucose uptake during contraction , 2005, The EMBO journal.

[51]  Fred W. Turek,et al.  Obesity and Metabolic Syndrome in Circadian Clock Mutant Mice , 2005, Science.

[52]  P. Chambon,et al.  Temporally controlled targeted somatic mutagenesis in skeletal muscles of the mouse , 2005, Genesis.

[53]  R. Colman,et al.  Progressive arthropathy in mice with a targeted disruption of the Mop3/Bmal‐1 locus , 2005, Genesis.

[54]  J. Holloszy,et al.  A potential link between muscle peroxisome proliferator- activated receptor-alpha signaling and obesity-related diabetes. , 2005, Cell metabolism.

[55]  Kristian Gundersen,et al.  Three myosin heavy chain isoforms in type 2 skeletal muscle fibres , 1989, Journal of Muscle Research & Cell Motility.

[56]  Ana Conesa,et al.  maSigPro: a Method to Identify Significantly Differential Expression Profiles in Time-Course Microarray Experiments , 2006, Spanish Bioinformatics Conference.

[57]  Satchidananda Panda,et al.  BMAL1 and CLOCK, Two Essential Components of the Circadian Clock, Are Involved in Glucose Homeostasis , 2004, PLoS biology.

[58]  R. Feneberg,et al.  Circadian Rhythm of Glucose Uptake in Cultures of Skeletal Muscle Cells and Adipocytes in Wistar-Kyoto, Wistar, Goto-Kakizaki, and Spontaneously Hypertensive Rats , 2004, Chronobiology international.

[59]  Rafael A Irizarry,et al.  Exploration, normalization, and summaries of high density oligonucleotide array probe level data. , 2003, Biostatistics.

[60]  A. Knutsson,et al.  Is there an association between shift work and having a metabolic syndrome? Results from a population based study of 27 485 people , 2001, Occupational and environmental medicine.

[61]  John B. Hogenesch,et al.  Mop3 Is an Essential Component of the Master Circadian Pacemaker in Mammals , 2000, Cell.

[62]  C. Kahn,et al.  Targeted disruption of the glucose transporter 4 selectively in muscle causes insulin resistance and glucose intolerance , 2000, Nature Medicine.

[63]  C. Kahn,et al.  Redistribution of substrates to adipose tissue promotes obesity in mice with selective insulin resistance in muscle. , 2000, The Journal of clinical investigation.

[64]  J. Haspel,et al.  Selective expression of Cre recombinase in skeletal muscle fibers , 2000, Genesis.

[65]  Rena R Wing,et al.  Skeletal muscle fatty acid metabolism in association with insulin resistance, obesity, and weight loss. , 1999, American journal of physiology. Endocrinology and metabolism.

[66]  J. Pessin,et al.  American Journal of Physiology - Endocrinology and Metabolism: Prologue , 1999 .

[67]  C. Kahn,et al.  A muscle-specific insulin receptor knockout exhibits features of the metabolic syndrome of NIDDM without altering glucose tolerance. , 1998, Molecular cell.

[68]  R. McKelvie,et al.  Pyruvate dehydrogenase activity and acetyl group accumulation during exercise after different diets. , 1993, The American journal of physiology.

[69]  G. Cooney,et al.  Diurnal patterns of cardiac and hepatic pyruvate dehydrogenase complex activity in gold-thioglucose-obese mice. , 1993, The Biochemical journal.

[70]  D. Constantin-Teodosiu,et al.  A sensitive radioisotopic assay of pyruvate dehydrogenase complex in human muscle tissue. , 1991, Analytical biochemistry.

[71]  D. Constantin-Teodosiu,et al.  Radioisotopic assays of CoASH and carnitine and their acetylated forms in human skeletal muscle. , 1990, Analytical biochemistry.

[72]  C. Wang Insulin-stimulated glucose uptake in rat diaphragm during postnatal development: lack of correlation with the number of insulin receptors and of intracellular glucose transporters. , 1985, Proceedings of the National Academy of Sciences of the United States of America.

[73]  R. DeFronzo,et al.  The Effect of Insulin on the Disposal of Intravenous Glucose: Results from Indirect Calorimetry and Hepatic and Femoral Venous Catheterization , 1981, Diabetes.