Autoantibody production and cytokine profiles of MHC class I (beta2-microglobulin) gene deleted New Zealand black (NZB) mice.
暂无分享,去创建一个
[1] M. Gershwin,et al. The natural history of disease expression in CD4 and CD8 gene-deleted New Zealand black (NZB) mice. , 1996, Journal of immunology.
[2] H. Ljunggren,et al. Reactivity and Specificity of CD8+ T Cells in Mice with Defects in the MHC Class I Antigen‐Presenting Pathway , 1996, Immunological reviews.
[3] H. Yamamoto,et al. Development of CD8 alpha alpha+ intestinal intraepithelial T cells in beta 2-microglobulin- and/or TAP1-deficient mice. , 1996, Journal of immunology.
[4] H. Macdonald. NK1.1+ T cell receptor-alpha/beta+ cells: new clues to their origin, specificity, and function , 1995, The Journal of experimental medicine.
[5] C. Mohan,et al. Mechanisms of the pathogenic autoimmune response in lupus: Prospects for specific immunotherapy , 1995, Immunologic research.
[6] Eric O Long,et al. Peptide specificity in the recognition of MHC class I by natural killer cell clones , 1995, Science.
[7] J. Levine,et al. Cytokine dysregulation and the initiation of systemic autoimmunity. , 1994, Immunology letters.
[8] P. Walden,et al. CD4+ and CD8+ alpha beta, and gamma delta T cells are cytotoxic effector cells of beta 2-microglobulin-deficient mice against cells having normal MHC class I expression. , 1994, Journal of immunology.
[9] G. Anderson,et al. Thymic epithelial cells provide unique signals for positive selection of CD4+CD8+ thymocytes in vitro , 1994, The Journal of experimental medicine.
[10] A. Singer,et al. Crosstalk in the mouse thymus. , 1994, Immunology today.
[11] W. Paul,et al. CD4pos, NK1.1pos T cells promptly produce interleukin 4 in response to in vivo challenge with anti-CD3 , 1994, The Journal of experimental medicine.
[12] M. Sitkovsky,et al. Development and antigen specificity of CD8+ cytotoxic T lymphocytes in beta 2-microglobulin-negative, MHC class I-deficient mice in response to immunization with tumor cells. , 1994, Journal of immunology.
[13] R. Glas,et al. The CD8+ T cell repertoire in beta 2-microglobulin-deficient mice is biased towards reactivity against self-major histocompatibility class I , 1994, The Journal of experimental medicine.
[14] I. Weissman. Developmental switches in the immune system , 1994, Cell.
[15] P. Walden,et al. Increased number of cytotoxic T cells within CD4+8− T cells in β2‐microglobulin, major histocompatibility complex class I‐deficient mice , 1993, European journal of immunology.
[16] S. Ostrand-Rosenberg,et al. Beta 2M-/- knockout mice contain low levels of CD8+ cytotoxic T lymphocyte that mediate specific tumor rejection. , 1993, Journal of immunology.
[17] D. Singer,et al. Resistance of MHC class I-deficient mice to experimental systemic lupus erythematosus. , 1993, Science.
[18] T. Wilson,et al. Thymic microenvironmental abnormalities and thymic selection in NZB.H-2bm12 mice. , 1993, Journal of immunology.
[19] M. Sitkovsky,et al. Highly lytic CD8+, alpha beta T-cell receptor cytotoxic T cells with major histocompatibility complex (MHC) class I antigen-directed cytotoxicity in beta 2-microglobulin, MHC class I-deficient mice. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[20] M. Bix,et al. Functionally conformed free class I heavy chains exist on the surface of beta 2 microglobulin negative cells , 1992, The Journal of experimental medicine.
[21] P. Morrissey,et al. Beta 2-microglobulin-, CD8+ T-cell-deficient mice survive inoculation with high doses of vaccinia virus and exhibit altered IgG responses. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[22] R. Jaenisch,et al. Skin graft rejection by beta 2-microglobulin-deficient mice , 1992, The Journal of experimental medicine.
[23] A. Singer,et al. Disorganization and restoration of thymic medullary epithelial cells in T cell receptor‐negative scid mice: Evidence that receptor‐bearing lymphocytes influence maturation of the thymic microenvironment , 1991, European journal of immunology.
[24] P. Marrack,et al. Normal development of mice deficient in beta 2M, MHC class I proteins, and CD8+ T cells. , 1990, Science.
[25] T. Wilson,et al. The phenotypic heterogeneity of mouse thymic stromal cells. , 1990, Immunology.
[26] R. Jaenisch,et al. β2-Microglobulin deficient mice lack CD4−8+ cytolytic T cells , 1990, Nature.
[27] H. Boehmer,et al. Positive selection of antigen-specific T cells in thymus by restricting MHC molecules , 1988, Nature.
[28] Y. Uematsu,et al. Thymic major histocompatibility complex antigens and the αβ T-cell receptor determine the CD4/CD8 phenotype of T cells , 1988, Nature.
[29] J. Portanova,et al. The contribution of L3T4+ T cells to lymphoproliferation and autoantibody production in MRL-lpr/lpr mice , 1988, The Journal of experimental medicine.
[30] D. Wofsy. Administration of monoclonal anti-T cell antibodies retards murine lupus in BXSB mice. , 1986, Journal of immunology.
[31] W. Seaman,et al. Successful treatment of autoimmunity in NZB/NZW F1 mice with monoclonal antibody to L3T4 , 1985, The Journal of experimental medicine.
[32] H. Mcdevitt,et al. Treatment of (NZB x NZW)F1 disease with anti-I-A monoclonal antibodies , 1983, The Journal of experimental medicine.
[33] R. Warnke,et al. The immunologic phenotyping of bone marrow biopsies and aspirates: frozen section techniques. , 1982, Blood.
[34] A. Ahmed,et al. Studies of congenitally immunologic mutant New Zealand mice. II. Absence of T cell progenitor populations and B cell defects of congenitally athymic (nude) New Zealand Black (NZB) mice. , 1979, Journal of immunology.
[35] A. Ahmed,et al. Studies of congenitally immunologic mutant New Zealand mice. VI. Spontaneous and induced autoantibodies to red cells and DNA occur in New Zealand X-linked immunodeficient (Xid) mice without phenotypic alternations of the Xid gene or generalized polyclonal B cell activation. , 1979, Journal of immunology.
[36] M. Gershwin,et al. Studies of congenitally immunologic mutant New Zealand mice. I. Autoimmune features of hereditarily asplenic (Dh/+) NZB mice; reduction of naturally occurring thymocytotoxic antibody and normal suppressor function. , 1979, Journal of immunology.
[37] F. Burnet. A reassessment of the forbidden clone hypothesis of autoimmune disease. , 1972, The Australian journal of experimental biology and medical science.
[38] A. Bendelac. Mouse NK1+ T cells. , 1995, Current opinion in immunology.
[39] J. Yewdell,et al. Cell biology of antigen processing and presentation to major histocompatibility complex class I molecule-restricted T lymphocytes. , 1992, Advances in immunology.
[40] W. Ewijk. T-cell differentiation is influenced by thymic microenvironments. , 1991 .
[41] W. vanEwijk,et al. Phenotypic characterization of murine thymic microenvironments. , 1990 .