BMP signaling controls muscle mass
暂无分享,去创建一个
Stefano Piccolo | Marco Sandri | Luana Toniolo | A. Goldberg | S. Dupont | L. Toniolo | M. Sandri | A. Ferry | E. Mouisel | S. Piccolo | E. Enzo | B. Blaauw | Helge Amthor | Arnaud Ferry | Sirio Dupont | Elena Enzo | Alfred L. Goldberg | S. Stricker | Sigmar Stricker | Roberta Sartori | E. Schirwis | Bert Blaauw | Sergia Bortolanza | Jinghui Zhao | Amalia Stantzou | Etienne Mouisel | H. Amthor | R. Sartori | Jinghui Zhao | S. Bortolanza | A. Stantzou | E. Schirwis
[1] Gerald C. Chu,et al. Smad4 is dispensable for normal pancreas development yet critical in progression and tumor biology of pancreas cancer. , 2006, Genes & development.
[2] A. Gomes,et al. Upregulation of proteasome activity in muscle RING finger 1‐null mice following denervation , 2012, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[3] A. Goldberg,et al. FoxO3 controls autophagy in skeletal muscle in vivo. , 2007, Cell metabolism.
[4] P. ten Dijke,et al. Identification and Functional Characterization of Distinct Critically Important Bone Morphogenetic Protein-specific Response Elements in the Id1 Promoter* , 2002, The Journal of Biological Chemistry.
[5] A. Weissman,et al. RINGs of good and evil: RING finger ubiquitin ligases at the crossroads of tumour suppression and oncogenesis , 2011, Nature Reviews Cancer.
[6] Naoya Yamamoto,et al. Constitutively active BMP type I receptors transduce BMP-2 signals without the ligand in C2C12 myoblasts. , 1997, Experimental cell research.
[7] V. Sartorelli,et al. Molecular and Cellular Determinants of Skeletal Muscle Atrophy and Hypertrophy , 2004, Science's STKE.
[8] Luca Scorrano,et al. Mitochondrial fission and remodelling contributes to muscle atrophy , 2010, The EMBO journal.
[9] Denis Vivien,et al. Direct binding of Smad3 and Smad4 to critical TGFβ‐inducible elements in the promoter of human plasminogen activator inhibitor‐type 1 gene , 1998, The EMBO journal.
[10] R. Harland,et al. The Spemann Organizer Signal noggin Binds and Inactivates Bone Morphogenetic Protein 4 , 1996, Cell.
[11] C. Reggiani,et al. Inducible activation of Akt increases skeletal muscle mass and force without satellite cell activation , 2009, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[12] M. Matzuk,et al. Regulation of muscle growth by multiple ligands signaling through activin type II receptors. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[13] L. Larsson,et al. The bone morphogenetic protein axis is a positive regulator of skeletal muscle mass , 2013, The Journal of cell biology.
[14] G. Butler-Browne,et al. Muscle weakness and atrophy are associated with decreased regenerative capacity and changes in mTOR signaling in skeletal muscles of venerable (18–24‐month‐old) dystrophic mdx mice , 2010, Muscle & nerve.
[15] J. Haspel,et al. Selective expression of Cre recombinase in skeletal muscle fibers , 2000, Genesis.
[16] Marco Sandri,et al. Signaling in muscle atrophy and hypertrophy. , 2008, Physiology.
[17] S. Webb,et al. Tied up in knots , 2004, Anaesthesia.
[18] G. Lanfranchi,et al. JunB transcription factor maintains skeletal muscle mass and promotes hypertrophy , 2010, The Journal of cell biology.
[19] Se-Jin Lee,et al. Regulation of skeletal muscle mass in mice by a new TGF-p superfamily member , 1997, nature.
[20] Se-Jin Lee,et al. Regulation of myostatin activity and muscle growth , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[21] D J Glass,et al. Identification of Ubiquitin Ligases Required for Skeletal Muscle Atrophy , 2001, Science.
[22] S. Witt,et al. MuRF1-dependent regulation of systemic carbohydrate metabolism as revealed from transgenic mouse studies. , 2008, Journal of molecular biology.
[23] D. Brazil,et al. Extracellular BMP-antagonist regulation in development and disease: tied up in knots. , 2010, Trends in cell biology.
[24] Tomokazu Fukuda,et al. BMP type I receptor inhibition reduces heterotopic ossification , 2008, Nature Medicine.
[25] C. Mammucari,et al. Smad2 and 3 transcription factors control muscle mass in adulthood. , 2009, American journal of physiology. Cell physiology.
[26] C. Reggiani,et al. Akt activation prevents the force drop induced by eccentric contractions in dystrophin-deficient skeletal muscle. , 2008, Human molecular genetics.
[27] N. Copeland,et al. Limb alterations in brachypodism mice due to mutations in a new member of the TGFβ-superfamily , 1994, Nature.
[28] K. Miyazono,et al. Id: A Target of BMP Signaling , 2002, Science's STKE.
[29] Marco Sandri,et al. Foxo Transcription Factors Induce the Atrophy-Related Ubiquitin Ligase Atrogin-1 and Cause Skeletal Muscle Atrophy , 2004, Cell.
[30] I. Conboy,et al. Imbalance between pSmad3 and Notch induces CDK inhibitors in old muscle stem cells , 2008, Nature.
[31] S. Hatakeyama,et al. Myostatin reduces Akt/TORC1/p70S6K signaling, inhibiting myoblast differentiation and myotube size. , 2009, American journal of physiology. Cell physiology.
[32] A. Goldberg,et al. Protein degradation by the ubiquitin-proteasome pathway in normal and disease states. , 2006, Journal of the American Society of Nephrology : JASN.
[33] A. Munnich,et al. Mutations at a single codon in Mad homology 2 domain of SMAD4 cause Myhre syndrome , 2011, Nature Genetics.
[34] A. Goldberg,et al. Rapid disuse and denervation atrophy involve transcriptional changes similar to those of muscle wasting during systemic diseases , 2007, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.