Acute Myocardial Infarction, Cardioprotection, and Muse Cells.

[1]  R. Califf,et al.  Relationship between delay in performing direct coronary angioplasty and early clinical outcome in patients with acute myocardial infarction: results from the global use of strategies to open occluded arteries in Acute Coronary Syndromes (GUSTO-IIb) trial. , 1999, Circulation.

[2]  R. Kloner,et al.  α‐Adrenoceptor Stimulation With Exogenous Norepinephrine or Release of Endogenous Catecholamines Mimics Ischemic Preconditioning , 1994, Circulation.

[3]  H. Okano,et al.  Nonhematopoietic mesenchymal stem cells can be mobilized and differentiate into cardiomyocytes after myocardial infarction. , 2004, Blood.

[4]  M. Pfeffer,et al.  Ventricular Remodeling After Myocardial Infarction: Experimental Observations and Clinical Implications , 1990, Circulation.

[5]  M. Arai,et al.  Acceleration of the Healing Process and Myocardial Regeneration May Be Important as a Mechanism of Improvement of Cardiac Function and Remodeling by Postinfarction Granulocyte Colony–Stimulating Factor Treatment , 2004, Circulation.

[6]  D. Yellon,et al.  Ischaemic preconditioning limits infarct size in the rat heart. , 1992, Cardiovascular research.

[7]  C. Thiemermann,et al.  Limitation of myocardial infarct size in the rabbit by ischaemic preconditioning is abolished by sodium 5-hydroxydecanoate. , 1996, Cardiovascular research.

[8]  C. Nienaber,et al.  Preservation From Left Ventricular Remodeling by Front-Integrated Revascularization and Stem Cell Liberation in Evolving Acute Myocardial Infarction by Use of Granulocyte-Colony–Stimulating Factor (FIRSTLINE-AMI) , 2005, Circulation.

[9]  M. Arai,et al.  Effect of granulocyte colony-stimulating factor treatment at a low dose but for a long duration in patients with coronary heart disease. , 2006, Circulation journal : official journal of the Japanese Circulation Society.

[10]  T. Aoyama,et al.  Endogenous Adenosine May Be Related to Left Ventricular Dysfunction, Dilation, and Wall Thinning in Patients With Heart Disease. , 2018, Circulation journal : official journal of the Japanese Circulation Society.

[11]  H. Ohgushi,et al.  Intravenous administration of mesenchymal stem cells improves cardiac function in rats with acute myocardial infarction through angiogenesis and myogenesis. , 2004, American journal of physiology. Heart and circulatory physiology.

[12]  J. Downey,et al.  Protection Against Infarction Afforded by Preconditioning is Mediated by A1 Adenosine Receptors in Rabbit Heart , 1991, Circulation.

[13]  C. Muramatsu,et al.  S1P–S1PR2 Axis Mediates Homing of Muse Cells Into Damaged Heart for Long-Lasting Tissue Repair and Functional Recovery After Acute Myocardial Infarction , 2018, Circulation research.

[14]  J. Downey,et al.  Role of bradykinin in protection of ischemic preconditioning in rabbit hearts. , 1995, Circulation research.

[15]  S. Kuroda,et al.  Mobilization of Pluripotent Multilineage-Differentiating Stress-Enduring Cells in Ischemic Stroke. , 2016, Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association.

[16]  W. Schaper,et al.  Ischemic preconditioning reduces infarct size in swine myocardium. , 1990, Circulation research.

[17]  Federica Limana,et al.  Mobilized bone marrow cells repair the infarcted heart, improving function and survival , 2001, Proceedings of the National Academy of Sciences of the United States of America.

[18]  J. George Stem cell therapy in acute myocardial infarction: a review of clinical trials. , 2010, Translational research : the journal of laboratory and clinical medicine.

[19]  J. Downey,et al.  alpha 1-adrenergic agonists precondition rabbit ischemic myocardium independent of adenosine by direct activation of protein kinase C. , 1994, Circulation research.

[20]  S. Sasayama,et al.  Superoxide dismutase and N-2-mercaptopropionyl glycine attenuate infarct size limitation effect of ischaemic preconditioning in the rabbit. , 1994, Cardiovascular research.

[21]  H. Figulla,et al.  Treatment with granulocyte colony-stimulating factor for mobilization of bone marrow cells in patients with acute myocardial infarction. , 2005, American heart journal.

[22]  C. Nienaber,et al.  Prevention of Left Ventricular Remodeling With Granulocyte Colony-Stimulating Factor After Acute Myocardial Infarction: Final 1-year Results of the Front-Integrated Revascularization and Stem Cell Liberation in Evolving Acute Myocardial Infarction by Granulocyte Colony-Stimulating Factor (FIRSTLINE- , 2005, Circulation.

[23]  J. Downey,et al.  Preconditioning protects ischemic rabbit heart by protein kinase C activation. , 1994, The American journal of physiology.

[24]  Richard B. Thompson,et al.  A novel protective effect of erythropoietin in the infarcted heart. , 2003, The Journal of clinical investigation.

[25]  M. Hori,et al.  Effect of angina pectoris on myocardial protection in patients with reperfused anterior wall myocardial infarction: retrospective clinical evidence of "preconditioning". , 1995, Journal of the American College of Cardiology.

[26]  M. Hori,et al.  Evidence for the delayed effect in human ischemic preconditioning: prospective multicenter study for preconditioning in acute myocardial infarction. , 1999, Journal of the American College of Cardiology.

[27]  X. Niu,et al.  Erythropoietin treatment in patients with acute myocardial infarction: a meta-analysis of randomized controlled trials. , 2012, American heart journal.

[28]  R. Ferrari,et al.  Use of granulocyte-colony stimulating factor during acute myocardial infarction to enhance bone marrow stem cell mobilization in humans: clinical and angiographic safety profile. , 2005, European heart journal.

[29]  M. Galvani,et al.  Prodromal angina limits infarct size. A role for ischemic preconditioning. , 1995, Circulation.

[30]  Guanying Wang,et al.  κ- but not δ-opioid receptors mediate effects of ischemic preconditioning on both infarct and arrhythmia in rats , 2001 .

[31]  M. Arai,et al.  Infarct Size-Reducing Effect of Ischemic Preconditioning Is Related to α1b-Adrenoceptors But Not to α1a-Adrenoceptors in Rabbits , 1997 .

[32]  R. Jennings,et al.  Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium. , 1986, Circulation.

[33]  Robert L Wilensky,et al.  A quantitative, randomized study evaluating three methods of mesenchymal stem cell delivery following myocardial infarction. , 2006, European heart journal.

[34]  M. Schwaiger,et al.  Stem cell mobilization by granulocyte colony-stimulating factor in patients with acute myocardial infarction: a randomized controlled trial. , 2006, JAMA.

[35]  M. Dezawa Muse Cells Provide the Pluripotency of Mesenchymal Stem Cells: Direct Contribution of Muse Cells to Tissue Regeneration , 2016, Cell transplantation.

[36]  H. Johnsen,et al.  Stem Cell Mobilization Induced by Subcutaneous Granulocyte-Colony Stimulating Factor to Improve Cardiac Regeneration After Acute ST-Elevation Myocardial Infarction: Result of the Double-Blind, Randomized, Placebo-Controlled Stem Cells in Myocardial Infarction (STEMMI) Trial , 2006, Circulation.

[37]  R. Guyton,et al.  Inhibition of myocardial injury by ischemic postconditioning during reperfusion: comparison with ischemic preconditioning. , 2003, American journal of physiology. Heart and circulatory physiology.

[38]  M. Mocanu,et al.  Postconditioning: A Form of “Modified Reperfusion” Protects the Myocardium by Activating the Phosphatidylinositol 3-Kinase–Akt Pathway , 2004, Circulation research.

[39]  D. Glogar,et al.  Management of acute myocardial infarction: evaluating the past, practicing in the present, elaborating the future. , 1996, American heart journal.

[40]  Pietro Ghezzi,et al.  Recombinant human erythropoietin protects the myocardium from ischemia-reperfusion injury and promotes beneficial remodeling , 2003, Proceedings of the National Academy of Sciences of the United States of America.

[41]  Y. Fujiyoshi,et al.  Multilineage-differentiating stress-enduring (Muse) cells are a primary source of induced pluripotent stem cells in human fibroblasts , 2011, Proceedings of the National Academy of Sciences.

[42]  J. Adamson,et al.  The anemia of chronic renal failure: pathophysiology and effects of recombinant erythropoietin. , 1990, Contributions to nephrology.

[43]  J. Downey,et al.  Intravenous Pretreatment With A1‐Selective Adenosine Analogues Protects the Heart Against Infarction , 1992, Circulation.

[44]  U. Galderisi,et al.  The secretome of MUSE cells contains factors that may play a role in regulation of stemness, apoptosis and immunomodulation , 2017, Cell cycle.

[45]  M. Dezawa,et al.  Isolation, culture and evaluation of multilineage-differentiating stress-enduring (Muse) cells , 2013, Nature Protocols.

[46]  A. Inutsuka,et al.  Unique multipotent cells in adult human mesenchymal cell populations , 2010, Proceedings of the National Academy of Sciences.

[47]  I. Komuro,et al.  G-CSF prevents cardiac remodeling after myocardial infarction by activating the Jak-Stat pathway in cardiomyocytes , 2005, Nature Medicine.

[48]  G. Gross,et al.  Blockade of ATP-sensitive potassium channels prevents myocardial preconditioning in dogs. , 1992, Circulation research.

[49]  K. Nishigaki,et al.  Mobilized Muse Cells After Acute Myocardial Infarction Predict Cardiac Function and Remodeling in the Chronic Phase. , 2017, Circulation journal : official journal of the Japanese Circulation Society.