Contribution of complement‐stimulated hepatic macrophages and neutrophils to endotoxin‐induced liver injury in rats
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[1] C. Smith,et al. Functional inactivation of neutrophils with a Mac‐1 (CD11b/CD18) monoclonal antibody protects against ischemia‐reperfusion injury in rat liver , 1993, Hepatology.
[2] H. Jaeschke,et al. Superoxide generation by neutrophils and Kupffer cells during in vivo reperfusion after hepatic ischemia in rats , 1992, Journal of leukocyte biology.
[3] J. Harlan,et al. Adhesion : its role in inflammatory disease , 1992 .
[4] H. Jaeschke,et al. NADH-dependent reductive stress and ferritin-bound iron in allyl alcohol-induced lipid peroxidation in vivo: the protective effect of vitamin E. , 1992, Chemico-biological interactions.
[5] R. Roth,et al. Neutrophil depletion protects against liver injury from bacterial endotoxin. , 1992, Laboratory investigation; a journal of technical methods and pathology.
[6] J. Horwitz,et al. Evaluation of the electroinjection method for introducing proteins into living cells. , 1991, The American journal of physiology.
[7] G. R. Carson,et al. Recombinant soluble human complement receptor type 1 inhibits inflammation in the reversed passive arthus reaction in rats. , 1991, Journal of immunology.
[8] H. Jaeschke,et al. Vascular oxidant stress and hepatic ischemia/reperfusion injury. , 1991, Free radical research communications.
[9] H. Jaeschke,et al. Superoxide generation by Kupffer cells and priming of neutrophils during reperfusion after hepatic ischemia. , 1991, Free radical research communications.
[10] C. Smith,et al. Neutrophils contribute to ischemia/reperfusion injury in rat liver in vivo , 1990, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[11] D. Remick,et al. Endotoxin-induced cytokine gene expression in vivo. II. Regulation of tumor necrosis factor and interleukin-1 alpha/beta expression and suppression. , 1990, The American journal of pathology.
[12] K. Decker,et al. Biologically active products of stimulated liver macrophages (Kupffer cells). , 1990, European journal of biochemistry.
[13] J. Nagakawa,et al. Involvement of tumor necrosis factor-alpha in the pathogenesis of activated macrophage-mediated hepatitis in mice. , 1990, Gastroenterology.
[14] J. Spitzer,et al. Superoxide Anion Generation in the Liver During the Early Stage of Endotoxemia in Rats , 1990, Journal of leukocyte biology.
[15] G. R. Carson,et al. Soluble human complement receptor type 1: in vivo inhibitor of complement suppressing post-ischemic myocardial inflammation and necrosis. , 1990, Science.
[16] M. Entman,et al. Mac-1 (CD11b/CD18) mediates adherence-dependent hydrogen peroxide production by human and canine neutrophils. , 1990, Journal of immunology.
[17] H. Coxson,et al. CD18-dependent and -independent mechanisms of neutrophil emigration in the pulmonary and systemic microcirculation of rabbits. , 1990, Journal of immunology.
[18] G. Bagby,et al. Pentoxifylline inhibits lipopolysaccharide-induced serum tumor necrosis factor and mortality. , 1990, Life sciences.
[19] H. Jaeschke,et al. Use of isolated perfused organs in hypoxia and ischemia/reperfusion oxidant stress. , 1990, Methods in enzymology.
[20] T. Hugli,et al. Endotoxin-induced shock in the rat. A role for C5a. , 1989, The American journal of pathology.
[21] K. Fujiwara,et al. In situ evaluation of the stimulatory state of hepatic macrophages based on their ability to produce superoxide anions in rats , 1989, The Journal of pathology.
[22] S. Weiss. Tissue destruction by neutrophils. , 1989, The New England journal of medicine.
[23] D. Dhumeaux,et al. In vitro toxicity of polymorphonuclear neutrophils to rat hepatocytes: Evidence for a proteinase‐mediated mechanism , 1988, Hepatology.
[24] B. Beutler,et al. Cachectin, cachexia, and shock. , 1988, Annual review of medicine.
[25] H. Jaeschke,et al. The role of acrolein in allyl alcohol-induced lipid peroxidation and liver cell damage in mice. , 1987, Biochemical pharmacology.
[26] M. Arthur,et al. Corynebacterium parvum-elicited hepatic macrophages demonstrate enhanced respiratory burst activity compared with resident Kupffer cells in the rat. , 1986, Gastroenterology.
[27] D. Laskin,et al. Accumulation of Activated Mononuclear Phagocytes in the Liver Following Lipopolysaccharide Treatment of Rats , 1986, Journal of leukocyte biology.
[28] M. Arthur,et al. Oxygen-derived free radicals promote hepatic injury in the rat. , 1985, Gastroenterology.
[29] P. Ward,et al. Evidence for role of hydroxyl radical in complement and neutrophil-dependent tissue injury. , 1983, The Journal of clinical investigation.
[30] G. Holdstock,et al. Proteolytic enzymes released by liver macrophages may promote hepatic injury in a rat model of hepatic damage. , 1981, Gastroenterology.
[31] J. Ferluga,et al. ROLE OF MONONUCLEAR INFILTRATING CELLS IN PATHOGENESIS OF HEPATITIS , 1978, The Lancet.
[32] J. Yates,et al. PNEUMOCYSTIS CARINII IN A HUSBAND AND WIFE , 1975, The Lancet.
[33] W. Moloney,et al. ESTERASE ACTIVITY IN LEUKOCYTES DEMONSTRATED BY THE USE OF NAPHTHOL AS-D CHLOROACETATE SUBSTRATE , 1960, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.