Amino acid determinants conferring stable sialidase activity at low pH for H5N1 influenza A virus neuraminidase

[1]  Y. Kawaoka,et al.  The Low-pH Stability Discovered in Neuraminidase of 1918 Pandemic Influenza A Virus Enhances Virus Replication , 2010, PloS one.

[2]  Jack Gauldie,et al.  Transforming Growth Factor-b: Activation by Neuraminidase and Role in Highly Pathogenic H5N1 Influenza Pathogenesis , 2010 .

[3]  Jiping Zhu,et al.  The Special Neuraminidase Stalk-Motif Responsible for Increased Virulence and Pathogenesis of H5N1 Influenza A Virus , 2009, PloS one.

[4]  M. Okamatsu,et al.  Molecular epidemiological analysis of highly pathogenic avian influenza H5N1 subtype isolated from poultry and wild bird in Thailand. , 2008, Virus research.

[5]  C. Basler,et al.  Progress in identifying virulence determinants of the 1918 H1N1 and the Southeast Asian H5N1 influenza A viruses. , 2008, Antiviral research.

[6]  J. Fu,et al.  Molecular characterization of the surface glycoprotein genes of an H5N1 influenza virus isolated from a human in Guangdong, China , 2007, Archives of Virology.

[7]  David J. Stevens,et al.  The structure of H5N1 avian influenza neuraminidase suggests new opportunities for drug design , 2006, Nature.

[8]  Samson S. Y. Wong,et al.  Avian Influenza Virus Infections in Humans , 2006, Chest.

[9]  B. Johansson,et al.  Variation in the divalent cation requirements of influenza A virus N1 neuraminidases. , 2006, Journal of biochemistry.

[10]  H. Goto,et al.  Sialidase Activity of Influenza A Virus in an Endocytic Pathway Enhances Viral Replication , 2005, Journal of Virology.

[11]  D. Miyamoto,et al.  Evolutional analysis of human influenza A virus N2 neuraminidase genes based on the transition of the low‐pH stability of sialidase activity1 , 2004, FEBS letters.

[12]  D. Miyamoto,et al.  A molecular mechanism for the low pH stability of sialidase activity of influenza A virus N2 neuraminidases , 2004 .

[13]  B. Johansson,et al.  Variation in the divalent cation requirements of influenza a virus N2 neuraminidases. , 2003, Journal of biochemistry.

[14]  D. Miyamoto,et al.  A molecular mechanism for the low‐pH stability of sialidase activity of influenza A virus N2 neuraminidases 1 , 2003, FEBS letters.

[15]  D. Miyamoto,et al.  Duck and human pandemic influenza A viruses retain sialidase activity under low pH conditions. , 2001, Journal of biochemistry.