Gadd45β is important for perpetuating cognate and inflammatory signals in T cells

[1]  Albert J Fornace,et al.  Gadd45a protects against UV irradiation-induced skin tumors, and promotes apoptosis and stress signaling via MAPK and p53. , 2002, Cancer research.

[2]  F. Itoh,et al.  Smad‐dependent GADD45β expression mediates delayed activation of p38 MAP kinase by TGF‐β , 2002 .

[3]  D. Chakravarty,et al.  Three GADD45 isoforms contribute to hypertonic stress phenotype of murine renal inner medullary cells. , 2002, American journal of physiology. Renal physiology.

[4]  A. Fornace,et al.  Genomic instability, centrosome amplification, cell cycle checkpoints and Gadd45a , 2002, Oncogene.

[5]  G. Feldman,et al.  STAT4 serine phosphorylation is critical for IL-12-induced IFN-γ production but not for cell proliferation , 2002, Proceedings of the National Academy of Sciences of the United States of America.

[6]  B. Hoffman,et al.  GADD45b and GADD45g are cdc2/cyclinB1 kinase inhibitors with a role in S and G2/M cell cycle checkpoints induced by genotoxic stress , 2002, Journal of cellular physiology.

[7]  Peter J. Murray,et al.  Shaping Gene Expression in Activated and Resting Primary Macrophages by IL-101 , 2002, The Journal of Immunology.

[8]  S. Kaufmann,et al.  Organ-Specific CD4+ T Cell Response During Listeria monocytogenes Infection1 , 2002, The Journal of Immunology.

[9]  B. Hoffman,et al.  Myeloid differentiation (MyD) primary response genes in hematopoiesis , 2002, Oncogene.

[10]  C. Gélinas,et al.  Regulation of the gadd45β promoter by NF-kB , 2002 .

[11]  Chen Dong,et al.  MAP kinases in the immune response. , 2002, Annual review of immunology.

[12]  A. Fornace,et al.  Gadd 45 a Protects against UV Irradiation-induced Skin Tumors , and Promotes Apoptosis and Stress Signaling via MAPK and p 531 , 2 , 2002 .

[13]  Francesca Zazzeroni,et al.  Induction of gadd45β by NF-κB downregulates pro-apoptotic JNK signalling , 2001, Nature.

[14]  Joshua M. Korn,et al.  The plasticity of dendritic cell responses to pathogens and their components. , 2001, Science.

[15]  A. Fornace,et al.  Oligomerization of Human Gadd45a Protein* , 2001, The Journal of Biological Chemistry.

[16]  R. Flavell,et al.  GADD45gamma mediates the activation of the p38 and JNK MAP kinase pathways and cytokine production in effector TH1 cells. , 2001, Immunity.

[17]  J. Ihle,et al.  Gadd45γ Is Dispensable for Normal Mouse Development and T-Cell Proliferation , 2001, Molecular and Cellular Biology.

[18]  W. Ouyang,et al.  IL-18–stimulated GADD45β required in cytokine-induced, but not TCR-induced, IFN-γ production , 2001, Nature Immunology.

[19]  Wei Zhang,et al.  Interaction of CR6 (GADD45γ) with Proliferating Cell Nuclear Antigen Impedes Negative Growth Control* , 2001, The Journal of Biological Chemistry.

[20]  A. Fornace,et al.  The central region of Gadd45 is required for its interaction with p21/WAF1. , 2000, Experimental cell research.

[21]  B. Hoffman,et al.  Characterization of MyD118, Gadd45, and Proliferating Cell Nuclear Antigen (PCNA) Interacting Domains , 2000, The Journal of Biological Chemistry.

[22]  I. Bae,et al.  Gadd45 family proteins are coactivators of nuclear hormone receptors. , 2000, Biochemical and biophysical research communications.

[23]  R. Flavell,et al.  Regulation of c-Jun NH2-terminal Kinase ( Jnk) Gene Expression during T Cell Activation , 2000, The Journal of experimental medicine.

[24]  Kendall A. Smith,et al.  Cytokine response gene 6 induces p21 and regulates both cell growth and arrest , 1999, Oncogene.

[25]  M. Gorospe,et al.  gadd45 Is Not Required for Activation of c-Jun N-terminal Kinase or p38 during Acute Stress* , 1999, The Journal of Biological Chemistry.

[26]  M. Karin,et al.  Stress-induced JNK Activation Is Independent of Gadd45 Induction* , 1999, The Journal of Biological Chemistry.

[27]  William F. Morgan,et al.  Genomic instability in Gadd45a-deficient mice , 1999, Nature Genetics.

[28]  Wei Zhang,et al.  CR6: A third member in the MyD118 and Gadd45 gene family which functions in negative growth control , 1999, Oncogene.

[29]  F. Christians,et al.  Induction of GADD45 and JNK/SAPK-Dependent Apoptosis following Inducible Expression of BRCA1 , 1999, Cell.

[30]  X. Wang,et al.  Association with Cdc2 and inhibition of Cdc2/Cyclin B1 kinase activity by the p53-regulated protein Gadd45 , 1999, Oncogene.

[31]  H. Saito,et al.  A Family of Stress-Inducible GADD45-like Proteins Mediate Activation of the Stress-Responsive MTK1/MEKK4 MAPKKK , 1998, Cell.

[32]  M. Su,et al.  Interferon‐γ expression by Th1 effector T cells mediated by the p38 MAP kinase signaling pathway , 1998, The EMBO journal.

[33]  F. Posas,et al.  A human homolog of the yeast Ssk2/Ssk22 MAP kinase kinase kinases, MTK1, mediates stress‐induced activation of the p38 and JNK pathways , 1997, The EMBO journal.

[34]  G. Johnson,et al.  Cloning of a Novel Mitogen-activated Protein Kinase Kinase Kinase, MEKK4, That Selectively Regulates the c-Jun Amino Terminal Kinase Pathway* , 1997, The Journal of Biological Chemistry.

[35]  A. Fornace,et al.  The differentiation primary response gene MyD118, related to GADD45, encodes for a nuclear protein which interacts with PCNA and p21WAF1/CIP1. , 1996, Oncogene.

[36]  A. Prescott,et al.  Gadd45 is a nuclear cell cycle regulated protein which interacts with p21Cip1. , 1995, Oncogene.

[37]  D. Pardoll,et al.  A recombinant Listeria monocytogenes vaccine expressing a model tumour antigen protects mice against lethal tumour cell challenge and causes regression of established tumours , 1995, Nature Medicine.

[38]  R. Ahmed,et al.  Recombinant Listeria monocytogenes as a live vaccine vehicle for the induction of protective anti-viral cell-mediated immunity. , 1995, Proceedings of the National Academy of Sciences of the United States of America.

[39]  P. O'Connor,et al.  Interaction of the p53-regulated protein Gadd45 with proliferating cell nuclear antigen. , 1994, Science.

[40]  S. Kaufmann,et al.  Studies with MHC-deficient knock-out mice reveal impact of both MHC I- and MHC II-dependent T cell responses on Listeria monocytogenes infection. , 1994, Journal of immunology.

[41]  C. Hsieh,et al.  Development of TH1 CD4+ T cells through IL-12 produced by Listeria-induced macrophages. , 1993, Science.

[42]  A. Fornace,et al.  Genotoxic‐Stress‐Response Genes and Growth‐Arrest Genes , 1992, Annals of the New York Academy of Sciences.

[43]  F. Itoh,et al.  Smad-dependent GADD 45 b expression mediates delayed activation of p 38 MAP kinase by TGF-b , 2022 .