The emerging field of osteoimmunology
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[1] Y. Toyama,et al. The role of DC-STAMP in maintenance of immune tolerance through regulation of dendritic cell function. , 2008, International immunology.
[2] A. Mandelin,et al. TH-17 cells in rheumatoid arthritis , 2008, Arthritis research & therapy.
[3] Chen Dong,et al. TH17 cells in development: an updated view of their molecular identity and genetic programming , 2008, Nature Reviews Immunology.
[4] Jay K Kolls,et al. The Biological Functions of T Helper 17 Cell Effector Cytokines in Inflammation , 2022 .
[5] J. Smolen,et al. CTLA-4 directly inhibits osteoclast formation , 2008, Annals of the rheumatic diseases.
[6] R. Matthews. The B Cell Slayer , 2007, Science.
[7] A. Paradowska,et al. The function of interleukin 17 in the pathogenesis of rheumatoid arthritis , 2007, Archivum Immunologiae et Therapiae Experimentalis.
[8] D. Mosser,et al. Monocyte subpopulations and their differentiation patterns during infection , 2007, Journal of leukocyte biology.
[9] D. Gladman,et al. Infliximab inhibits progression of radiographic damage in patients with active psoriatic arthritis through one year of treatment: Results from the induction and maintenance psoriatic arthritis clinical trial 2. , 2007, Arthritis and rheumatism.
[10] E. Schwarz,et al. Clinical development of anti-RANKL therapy , 2007, Arthritis research & therapy.
[11] Elizabeth H. Chen,et al. Cell–cell fusion , 2007, FEBS letters.
[12] Wei-Ping Qian,et al. B cells and T cells are critical for the preservation of bone homeostasis and attainment of peak bone mass in vivo. , 2007, Blood.
[13] H. Takayanagi. Osteoimmunology: shared mechanisms and crosstalk between the immune and bone systems , 2007, Nature Reviews Immunology.
[14] L. Ziegler‐Heitbrock,et al. The CD14+ CD16+ blood monocytes: their role in infection and inflammation , 2007, Journal of leukocyte biology.
[15] S. Teitelbaum. Osteoclasts: what do they do and how do they do it? , 2007, The American journal of pathology.
[16] Georg Schett,et al. Dickkopf-1 is a master regulator of joint remodeling , 2007, Nature Medicine.
[17] T. Miyamoto. The dendritic cell-specific transmembrane protein DC-STAMP is essential for osteoclast fusion and osteoclast bone-resorbing activity , 2006, Modern rheumatology.
[18] Stanley B. Cohen,et al. Rituximab for rheumatoid arthritis refractory to anti-tumor necrosis factor therapy: Results of a multicenter, randomized, double-blind, placebo-controlled, phase III trial evaluating primary efficacy and safety at twenty-four weeks. , 2006, Arthritis and rheumatism.
[19] Y. Kadono,et al. Osteoimmunology: interplay between the immune system and bone metabolism. , 2006, Annual review of immunology.
[20] P. Miller,et al. Denosumab in Postmenopausal Women with Low Bone Mineral Density , 2007 .
[21] S. Gordon,et al. Monocyte and macrophage heterogeneity , 2005, Nature Reviews Immunology.
[22] R. D. Hatton,et al. Interleukin 17–producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages , 2005, Nature Immunology.
[23] D. Gladman,et al. Adalimumab for the treatment of patients with moderately to severely active psoriatic arthritis: results of a double-blind, randomized, placebo-controlled trial. , 2005, Arthritis and rheumatism.
[24] Y. Toyama,et al. DC-STAMP is essential for cell–cell fusion in osteoclasts and foreign body giant cells , 2005, The Journal of experimental medicine.
[25] F. Luyten,et al. Modulation of bone morphogenetic protein signaling inhibits the onset and progression of ankylosing enthesitis. , 2005, The Journal of clinical investigation.
[26] C. Rabourdin-Combe,et al. Immature dendritic cell transdifferentiation into osteoclasts: a novel pathway sustained by the rheumatoid arthritis microenvironment. , 2004, Blood.
[27] K. Toh,et al. RANKL-induced DC-STAMP Is Essential for Osteoclastogenesis , 2004, The Journal of experimental medicine.
[28] J. Smolen,et al. Arthritis Induces Lymphocytic Bone Marrow Inflammation and Endosteal Bone Formation , 2004 .
[29] M. Duddy,et al. Distinct Profiles of Human B Cell Effector Cytokines: A Role in Immune Regulation?1 , 2004, The Journal of Immunology.
[30] M. Takami,et al. Osteoimmunology: interactions of the immune and skeletal systems. , 2004, Molecules and Cells.
[31] D. Scadden,et al. Osteoblastic cells regulate the haematopoietic stem cell niche , 2003, Nature.
[32] A. Keegan,et al. IL-4 suppresses osteoclast development and mature osteoclast function by a STAT6-dependent mechanism: irreversible inhibition of the differentiation program activated by RANKL. , 2003, Blood.
[33] David L. Lacey,et al. Osteoclast differentiation and activation , 2003, Nature.
[34] E. Lubberts. The role of IL-17 and family members in the pathogenesis of arthritis. , 2003, Current opinion in investigational drugs.
[35] J. Bijlsma,et al. Fcγ receptor expression levels on monocytes are elevated in rheumatoid arthritis patients with high erythrocyte sedimentation rate who do not use anti‐rheumatic drugs , 2003 .
[36] E. Schwarz,et al. Mechanisms of TNF-alpha- and RANKL-mediated osteoclastogenesis and bone resorption in psoriatic arthritis. , 2003, The Journal of clinical investigation.
[37] W. Dougall,et al. A RANK/TRAF6-dependent Signal Transduction Pathway Is Essential for Osteoclast Cytoskeletal Organization and Resorptive Function* , 2002, The Journal of Biological Chemistry.
[38] J. Penninger,et al. Role of RANKL and RANK in bone loss and arthritis , 2002, Annals of the rheumatic diseases.
[39] J. Edwards. Importance of T cells in rheumatoid synovitis: comment on the review by Firestein and Zvaifler. , 2002, Arthritis and rheumatism.
[40] H. Makino,et al. CD14+,CD16+ blood monocytes and joint inflammation in rheumatoid arthritis. , 2002, Arthritis and rheumatism.
[41] Hao Wu,et al. Distinct molecular mechanism for initiating TRAF6 signalling , 2002, Nature.
[42] S. Nishikawa,et al. TRAF6-deficient mice display hypohidrotic ectodermal dysplasia , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[43] J. Smolen,et al. Autoantibodies in rheumatoid arthritis and their clinical significance. , 2002, Arthritis research.
[44] B. Kirkham,et al. Reduction of joint inflammation and bone erosion in rat adjuvant arthritis by treatment with interleukin-17 receptor IgG1 Fc fusion protein. , 2002, Arthritis and rheumatism.
[45] T. Suda,et al. Bifurcation of osteoclasts and dendritic cells from common progenitors. , 2001, Blood.
[46] Y. Abu-Amer. IL-4 abrogates osteoclastogenesis through STAT6-dependent inhibition of NF-κB , 2001 .
[47] J. Reginster,et al. Effect of parathyroid hormone (1-34) on fractures and bone mineral density in postmenopausal women with osteoporosis. , 2001, The New England journal of medicine.
[48] Y. Kadono,et al. Segregation of TRAF6‐mediated signaling pathways clarifies its role in osteoclastogenesis , 2001, The EMBO journal.
[49] H. Staege,et al. Two novel genes FIND and LIND differentially expressed in deactivated and Listeria-infected human macrophages , 2001, Immunogenetics.
[50] A. Nakanishi,et al. The Effect of a Single Dose of Osteoprotegerin in Postmenopausal Women , 2001, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[51] R. Baron,et al. Activated parathyroid hormone/parathyroid hormone-related protein receptor in osteoblastic cells differentially affects cortical and trabecular bone. , 2001, The Journal of clinical investigation.
[52] C. Figdor,et al. DC‐STAMP, a novel multimembrane‐spanning molecule preferentially expressed by dendritic cells , 2000, European journal of immunology.
[53] T. Martin,et al. Therapeutic approaches to bone diseases. , 2000, Science.
[54] S. Takeshita,et al. Identification and Characterization of the New Osteoclast Progenitor with Macrophage Phenotypes Being Able to Differentiate into Mature Osteoclasts , 2000, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[55] T. Komori,et al. Regulation of osteoblast differentiation mediated by bone morphogenetic proteins, hedgehogs, and Cbfa1. , 2000, Endocrine reviews.
[56] T. Miyata,et al. Commitment and Differentiation of Osteoclast Precursor Cells by the Sequential Expression of C-Fms and Receptor Activator of Nuclear Factor κb (Rank) Receptors , 1999, The Journal of experimental medicine.
[57] H. Hanafusa,et al. TRANCE, a TNF family member, activates Akt/PKB through a signaling complex involving TRAF6 and c-Src. , 1999, Molecular cell.
[58] Josef M. Penninger,et al. Activated T cells regulate bone loss and joint destruction in adjuvant arthritis through osteoprotegerin ligand , 1999, Nature.
[59] Yongwon Choi,et al. TRANCE is a TNF family member that regulates dendritic cell and osteoclast function , 1999, Journal of leukocyte biology.
[60] P. Miossec,et al. Human interleukin-17: A T cell-derived proinflammatory cytokine produced by the rheumatoid synovium. , 1999, Arthritis and rheumatism.
[61] T. Martin,et al. IL-17 in synovial fluids from patients with rheumatoid arthritis is a potent stimulator of osteoclastogenesis. , 1999, The Journal of clinical investigation.
[62] S. Morony,et al. TRAF6 deficiency results in osteopetrosis and defective interleukin-1, CD40, and LPS signaling. , 1999, Genes & development.
[63] B. Kwon,et al. Receptor activator of NF‐κB recruits multiple TRAF family adaptors and activates c‐Jun N‐terminal kinase , 1999, FEBS letters.
[64] G. Kollias,et al. Mice lacking mature T and B lymphocytes develop arthritic lesions after immunization with type II collagen. , 1999, Journal of immunology.
[65] A. Livolsi,et al. Tumor Necrosis Factor-α Activation of Nuclear Transcription Factor-κB in Marrow Macrophages Is Mediated by c-Src Tyrosine Phosphorylation of IκBα* , 1998, The Journal of Biological Chemistry.
[66] D. Lacey,et al. Osteoprotegerin Ligand Is a Cytokine that Regulates Osteoclast Differentiation and Activation , 1998, Cell.
[67] K Yano,et al. Osteoclast differentiation factor is a ligand for osteoprotegerin/osteoclastogenesis-inhibitory factor and is identical to TRANCE/RANKL. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[68] R. Steinman,et al. TRANCE (Tumor Necrosis Factor [TNF]-related Activation-induced Cytokine), a New TNF Family Member Predominantly Expressed in T cells, Is a Dendritic Cell–specific Survival Factor , 1997, The Journal of experimental medicine.
[69] R. Dubose,et al. A homologue of the TNF receptor and its ligand enhance T-cell growth and dendritic-cell function , 1997, Nature.
[70] G Shimamoto,et al. Osteoprotegerin: A Novel Secreted Protein Involved in the Regulation of Bone Density , 1997, Cell.
[71] D. Kioussis,et al. Transgenic mice expressing human tumour necrosis factor: a predictive genetic model of arthritis. , 1991, The EMBO journal.
[72] A. Cope,et al. Regulatory T cells suppress osteoclast formation: A new link between the immune system and bone , 2008 .
[73] J. Block,et al. Rheumatoid arthritis: beyond tumor necrosis factor-alpha antagonists, B cell depletion, and T cell blockade. , 2008, American journal of therapeutics.
[74] R. Matthews. Autoimmune diseases. The B cell slayer. , 2007, Science.
[75] Y. Toyama,et al. Role of DC-STAMP in cellular fusion of osteoclasts and macrophage giant cells , 2006, Journal of Bone and Mineral Metabolism.
[76] Y. Yazici,et al. Denosumab in postmenopausal women with low bone mineral density. , 2006, The New England journal of medicine.
[77] E. Schwarz,et al. Systemic tumor necrosis factor α mediates an increase in peripheral CD11bhigh osteoclast precursors in tumor necrosis factor α–transgenic mice , 2004 .
[78] E. Schwarz,et al. Systemic tumor necrosis factor alpha mediates an increase in peripheral CD11bhigh osteoclast precursors in tumor necrosis factor alpha-transgenic mice. , 2004, Arthritis and rheumatism.
[79] J. Bijlsma,et al. Fcgamma receptor expression levels on monocytes are elevated in rheumatoid arthritis patients with high erythrocyte sedimentation rate who do not use anti-rheumatic drugs. , 2003, Rheumatology.
[80] E. Schwarz,et al. The TNF-alpha transgenic mouse model of inflammatory arthritis. , 2003, Springer seminars in immunopathology.
[81] S. Takemura. Lymphoid neogenesis in rheumatoid synovitis , 2003 .
[82] Y. Abu-Amer. IL-4 abrogates osteoclastogenesis through STAT6-dependent inhibition of NF-kappaB. , 2001, The Journal of clinical investigation.
[83] Josef M. Penninger,et al. Activated T cells regulate bone loss and joint destruction in adjuvant arthritis through osteoprotegerin ligand , 1999, Nature.
[84] A. Livolsi,et al. Tumor necrosis factor-alpha activation of nuclear transcription factor-kappaB in marrow macrophages is mediated by c-Src tyrosine phosphorylation of Ikappa Balpha. , 1998, The Journal of biological chemistry.