Phosphorylation of SET mediates apoptosis via P53 hyperactivation and NM23-H1 nuclear import
暂无分享,去创建一个
Jian-Zhi Wang | Xiaochuan Wang | Bin Zhang | Qun Wang | Mengjuan Wu | Guang Yu | Tonghai Yan | Dan Ke | Rong Liu | Dan Chen
[1] A-Microtubule , 2020, Definitions.
[2] B. Dai,et al. MTHFR c.677C>T Inhibits Cell Proliferation and Decreases Prostate Cancer Susceptibility in the Han Chinese Population in Shanghai , 2016, Scientific Reports.
[3] Yuan Wang,et al. Construction of SET overexpression vector and its effects on the proliferation and apoptosis of 293T cells. , 2016, Molecular medicine reports.
[4] Jian-Zhi Wang,et al. Stimulation of EphB2 attenuates tau phosphorylation through PI3K/Akt-mediated inactivation of glycogen synthase kinase-3β , 2015, Scientific Reports.
[5] Karan H. Patel,et al. The N-terminal Set-β Protein Isoform Induces Neuronal Death* , 2015, The Journal of Biological Chemistry.
[6] Yao-Cheng Lin,et al. Genome dynamics of the human embryonic kidney 293 lineage in response to cell biology manipulations , 2014, Nature Communications.
[7] Karan H. Patel,et al. Regulating Set-β's Subcellular Localization Toggles Its Function between Inhibiting and Promoting Axon Growth and Regeneration , 2014, The Journal of Neuroscience.
[8] L. Buée,et al. A major role for Tau in neuronal DNA and RNA protection in vivo under physiological and hyperthermic conditions , 2014, Front. Cell. Neurosci..
[9] H. Scrable,et al. P44, the ‘longevity-assurance’ isoform of P53, regulates tau phosphorylation and is activated in an age-dependent fashion , 2014, Aging cell.
[10] Bo Yang,et al. DNA-dependent protein kinase catalytic subunit (DNA-PKcs)-SIN1 association mediates ultraviolet B (UVB)-induced Akt Ser-473 phosphorylation and skin cell survival , 2013, Molecular Cancer.
[11] Yan-yan Xing,et al. Curcumin Protects Microglia and Primary Rat Cortical Neurons against HIV-1 gp120-Mediated Inflammation and Apoptosis , 2013, PloS one.
[12] Jian-Zhi Wang,et al. Ser9 phosphorylation causes cytoplasmic detention of I2 PP2A/SET in Alzheimer disease , 2013, Neurobiology of Aging.
[13] B. Kaina,et al. DNA damage-induced cell death: from specific DNA lesions to the DNA damage response and apoptosis. , 2013, Cancer letters.
[14] Hyun-A Seong,et al. Murine Protein Serine-threonine Kinase 38 Activates p53 Function through Ser15 Phosphorylation* , 2012, The Journal of Biological Chemistry.
[15] M. Odero,et al. Overexpression of SET is a recurrent event associated with poor outcome and contributes to protein phosphatase 2A inhibition in acute myeloid leukemia , 2012, Haematologica.
[16] X. Yao,et al. I2 PP2A regulates p53 and Akt correlatively and leads the neurons to abort apoptosis , 2012, Neurobiology of Aging.
[17] I. Grundke‐Iqbal,et al. Mechanism of inhibition of PP2A activity and abnormal hyperphosphorylation of tau by I2 PP2A/SET , 2011, FEBS letters.
[18] D. Wink,et al. Targeting SET/I2PP2A Oncoprotein Functions as a Multi-pathway Strategy for Cancer Therapy , 2010, Oncogene.
[19] L. Buée,et al. Nuclear Tau, a Key Player in Neuronal DNA Protection* , 2010, The Journal of Biological Chemistry.
[20] M. V. Vijayakumar,et al. Cdk5 phosphorylates non-genotoxically overexpressed p53 following inhibition of PP2A to induce cell cycle arrest/apoptosis and inhibits tumor progression , 2010, Molecular Cancer.
[21] Jianhua Shi,et al. Reduced O-GlcNAcylation links lower brain glucose metabolism and tau pathology in Alzheimer's disease. , 2009, Brain : a journal of neurology.
[22] Y. Liu,et al. Binding to the Minor Groove of the Double-Strand, Tau Protein Prevents DNA from Damage by Peroxidation , 2008, PloS one.
[23] Zhixue Liu,et al. Neuroprotective actions of PIKE-L by inhibition of SET proteolytic degradation by asparagine endopeptidase. , 2008, Molecular cell.
[24] Hyun-A Seong,et al. NM23-H1 Tumor Suppressor and Its Interacting Partner STRAP Activate p53 Function* , 2007, Journal of Biological Chemistry.
[25] Matthew J. Gamble,et al. SET and PARP1 remove DEK from chromatin to permit access by the transcription machinery , 2007, Nature Structural &Molecular Biology.
[26] Simon Lovestone,et al. p53 is upregulated in Alzheimer's disease and induces tau phosphorylation in HEK293a cells , 2007, Neuroscience Letters.
[27] Huaxi Xu,et al. Phosphorylation of tau antagonizes apoptosis by stabilizing β-catenin, a mechanism involved in Alzheimer's neurodegeneration , 2007, Proceedings of the National Academy of Sciences.
[28] P. Hordijk,et al. Rac1‐induced cell migration requires membrane recruitment of the nuclear oncogene SET , 2007, The EMBO journal.
[29] Fengqin Gao,et al. Protein phosphatase 2A inactivates Bcl2's antiapoptotic function by dephosphorylation and up-regulation of Bcl2-p53 binding. , 2006, Blood.
[30] A. Boynton,et al. Inhibition of PP2A, but not PP5, mediates p53 activation by low levels of okadaic acid in rat liver epithelial cells , 2006, Journal of cellular biochemistry.
[31] J. Lieberman,et al. The exonuclease TREX1 is in the SET complex and acts in concert with NM23-H1 to degrade DNA during granzyme A-mediated cell death. , 2006, Molecular cell.
[32] A. Prochiantz,et al. SET protein (TAF1β, I2PP2A) is involved in neuronal apoptosis induced by an amyloid precursor protein cytoplasmic subdomain , 2005, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[33] D. Jans,et al. Nuclear import of the respiratory syncytial virus matrix protein is mediated by importin beta1 independent of importin alpha. , 2005, Biochemistry.
[34] I. Grundke‐Iqbal,et al. Up-regulation of inhibitors of protein phosphatase-2A in Alzheimer's disease. , 2005, The American journal of pathology.
[35] I. Grundke‐Iqbal,et al. Inhibitors of protein phosphatase‐2A from human brain structures, immunocytological localization and activities towards dephosphorylation of the Alzheimer type hyperphosphorylated tau , 2005, FEBS letters.
[36] Y. Tamura,et al. Dephosphorylation of Bcl‐2 by protein phosphatase 2A results in apoptosis resistance , 2004, Cancer science.
[37] Robert M Friedlander,et al. Apoptosis and caspases in neurodegenerative diseases. , 2003, The New England journal of medicine.
[38] J. Lieberman,et al. Tumor Suppressor NM23-H1 Is a Granzyme A-Activated DNase during CTL-Mediated Apoptosis, and the Nucleosome Assembly Protein SET Is Its Inhibitor , 2003, Cell.
[39] Y. Taya,et al. Osmotic Shock Induces G1 Arrest through p53 Phosphorylation at Ser33 by Activated p38MAPK without Phosphorylation at Ser15 and Ser20 * , 2001, The Journal of Biological Chemistry.
[40] V. Janssens,et al. Protein phosphatase 2A: a highly regulated family of serine/threonine phosphatases implicated in cell growth and signalling. , 2001, The Biochemical journal.
[41] P. McNamara,et al. Regulation of Histone Acetylation and Transcription by INHAT, a Human Cellular Complex Containing the Set Oncoprotein , 2001, Cell.
[42] Yusuke Nakamura,et al. p53AIP1, a Potential Mediator of p53-Dependent Apoptosis, and Its Regulation by Ser-46-Phosphorylated p53 , 2000, Cell.
[43] G. Johnson,et al. The Microtubule Binding of Tau and High Molecular Weight Tau in Apoptotic PC12 Cells Is Impaired because of Altered Phosphorylation* , 1999, The Journal of Biological Chemistry.
[44] David S. Park,et al. Bax-Dependent Caspase-3 Activation Is a Key Determinant in p53-Induced Apoptosis in Neurons , 1999, The Journal of Neuroscience.
[45] I. Grundke‐Iqbal,et al. Regulation of expression, phosphorylation and biological activity of tau during differentiation in SY5Y cells , 1999, Brain Research.
[46] J. León,et al. Apoptosis and mitotic arrest are two independent effects of the protein phosphatases inhibitor okadaic acid in K562 leukemia cells. , 1999, Biochemical and biophysical research communications.
[47] Z. Damuni,et al. The Myeloid Leukemia-associated Protein SET Is a Potent Inhibitor of Protein Phosphatase 2A (*) , 1996, The Journal of Biological Chemistry.
[48] Hong Guo,et al. Purification and characterization of two potent heat-stable protein inhibitors of protein phosphatase 2A from bovine kidney. , 1995, Biochemistry.
[49] R. Zinkowski,et al. A novel tau transcript in cultured human neuroblastoma cells expressing nuclear tau , 1993, The Journal of cell biology.
[50] L. Binder,et al. Identification of nuclear tau isoforms in human neuroblastoma cells. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[51] J E Talmadge,et al. Evidence for a novel gene associated with low tumor metastatic potential. , 1988, Journal of the National Cancer Institute.
[52] H. Wiśniewski,et al. Abnormal phosphorylation of the microtubule-associated protein tau (tau) in Alzheimer cytoskeletal pathology. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[53] M. Dichter,et al. The use of specific AAV serotypes to stably transduce primary CNS neuron cultures. , 2012, Methods in molecular biology.
[54] David M. Conrad,et al. Apoptosis induced by protein phosphatase 2A (PP2A) inhibition in T leukemia cells is negatively regulated by PP2A-associated p38 mitogen-activated protein kinase. , 2007, Cellular signalling.
[55] John M. Walker,et al. Myeloid Leukemia , 2020, Methods In Molecular Medicine™.
[56] 織田 克利. p53AIP1,a potential mediator of p53-dependent apoptosis,and its regulation by Ser46-phosphorylated p53 , 2001 .
[57] G. Muszyńska,et al. Protein phosphatase 2A: variety of forms and diversity of functions. , 2001, Acta biochimica Polonica.
[58] Z. Damuni,et al. I1PP2A and I2PP2A. Two potent protein phosphatase 2A-specific inhibitor proteins. , 1998, Methods in molecular biology.