Vascular effects of a low-carbohydrate high-protein diet
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Robert E. Gerszten | K. Moore | J. Wykrzykowska | R. Gerszten | S. Foo | A. Rosenzweig | C. Sullivan | Kathryn J. Moore | Anthony Rosenzweig | E. Heller | Jennifer J Manning-Tobin | Joanna Wykrzykowska | Shi Yin Foo | Eric R. Heller | Christopher J. Sullivan | Jennifer J. Manning-Tobin | K. Moore
[1] J. Isner,et al. Bone marrow origin of endothelial progenitor cells responsible for postnatal vasculogenesis in physiological and pathological neovascularization. , 1999, Circulation research.
[2] G. Dagenais,et al. Plasma free fatty acid levels and the risk of ischemic heart disease in men: prospective results from the Québec Cardiovascular Study. , 2002, Atherosclerosis.
[3] P. Libby,et al. Absence of monocyte chemoattractant protein-1 reduces atherosclerosis in low density lipoprotein receptor-deficient mice. , 1998, Molecular cell.
[4] Leslie A. Smith,et al. Transplantation of Circulating Endothelial Progenitor Cells Restores Endothelial Function of Denuded Rabbit Carotid Arteries , 2004, Stroke.
[5] Qingbo Xu,et al. Rapid Endothelial Turnover in Atherosclerosis-Prone Areas Coincides With Stem Cell Repair in Apolipoprotein E–Deficient Mice , 2008, Circulation.
[6] N. Maeda,et al. Generation of mice carrying a mutant apolipoprotein E gene inactivated by gene targeting in embryonic stem cells. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[7] E. Gracely,et al. A low-carbohydrate as compared with a low-fat diet in severe obesity. , 2003, The New England journal of medicine.
[8] Y. Ni,et al. Inhibited atherosclerotic plaque formation by local administration of magnetically labeled endothelial progenitor cells (EPCs) in a rabbit model. , 2009, Atherosclerosis.
[9] J. Flier,et al. Hepatic fibroblast growth factor 21 is regulated by PPARalpha and is a key mediator of hepatic lipid metabolism in ketotic states. , 2007, Cell metabolism.
[10] G. Hansson,et al. Effects of sex and age on atherosclerosis and autoimmunity in apoE-deficient mice. , 1999, Atherosclerosis.
[11] A M Zeiher,et al. HMG-CoA reductase inhibitors (statins) increase endothelial progenitor cells via the PI 3-kinase/Akt pathway. , 2001, The Journal of clinical investigation.
[12] C. Heeschen,et al. Essential role of endothelial nitric oxide synthase for mobilization of stem and progenitor cells , 2003, Nature Medicine.
[13] T. Dai,et al. SDF-1alpha/CXCR4 decreases endothelial progenitor cells apoptosis under serum deprivation by PI3K/Akt/eNOS pathway. , 2008, Atherosclerosis.
[14] J. Isner,et al. Estrogen-Mediated, Endothelial Nitric Oxide Synthase–Dependent Mobilization of Bone Marrow–Derived Endothelial Progenitor Cells Contributes to Reendothelialization After Arterial Injury , 2003, Circulation.
[15] Pascal J. Goldschmidt-Clermont,et al. Aging, Progenitor Cell Exhaustion, and Atherosclerosis , 2003, Circulation.
[16] R. Hoffmann,et al. Benefit of Low-Fat Over Low-Carbohydrate Diet on Endothelial Health in Obesity , 2008, Hypertension.
[17] G. Nickenig,et al. Improvement of Endothelial Function by Systemic Transfusion of Vascular Progenitor Cells , 2006, Circulation research.
[18] A. Aljada,et al. Elevation of free fatty acids induces inflammation and impairs vascular reactivity in healthy subjects. , 2003, Diabetes.
[19] Takayuki Asahara,et al. Isolation of Putative Progenitor Endothelial Cells for Angiogenesis , 1997, Science.
[20] T. Murohara,et al. Transplanted cord blood-derived endothelial precursor cells augment postnatal neovascularization. , 2000, The Journal of clinical investigation.
[21] D. Rader,et al. A randomized trial of a low-carbohydrate diet for obesity. , 2003, The New England journal of medicine.
[22] E. Rubin,et al. Severe hypercholesterolemia and atherosclerosis in apolipoprotein E-deficient mice created by homologous recombination in ES cells , 1992, Cell.
[23] A. Quyyumi,et al. Circulating endothelial progenitor cells, vascular function, and cardiovascular risk. , 2003, The New England journal of medicine.
[24] Hong Yu,et al. Statin and Stromal Cell‐Derived Factor‐1 Additively Promote Angiogenesis by Enhancement of Progenitor Cells Incorporation into New Vessels , 2008, Stem cells.
[25] R. Ross. Cellular and molecular studies of atherogenesis. , 1997, Atherosclerosis.
[26] Haruchika Masuda,et al. Ischemia- and cytokine-induced mobilization of bone marrow-derived endothelial progenitor cells for neovascularization , 1999, Nature Medicine.
[27] R. Vile,et al. Autologous Culture-Modified Mononuclear Cells Confer Vascular Protection After Arterial Injury , 2003, Circulation.
[28] T. Kooistra,et al. Mouse models for atherosclerosis and pharmaceutical modifiers. , 2007, Arteriosclerosis, thrombosis, and vascular biology.
[29] Y. Fujio,et al. Akt Mediates Cytoprotection of Endothelial Cells by Vascular Endothelial Growth Factor in an Anchorage-dependent Manner* , 1999, The Journal of Biological Chemistry.
[30] S. Coughlin,et al. Monocyte chemoattractant protein-1 in human atheromatous plaques. , 1991, The Journal of clinical investigation.
[31] W. März,et al. Free fatty acids are independently associated with all-cause and cardiovascular mortality in subjects with coronary artery disease. , 2006, The Journal of clinical endocrinology and metabolism.
[32] J. D. Smith,et al. Mouse models of atherosclerosis. , 1998, Laboratory animal science.
[33] E. Gunsilius. Evidence from a leukemia model for maintenance of vascular endothelium by bone-marrow-derived endothelial cells. , 2003, Advances in experimental medicine and biology.
[34] B. Rollins,et al. MCP-1 deficiency reduces susceptibility to atherosclerosis in mice that overexpress human apolipoprotein B. , 1999, The Journal of clinical investigation.
[35] G. Nickenig,et al. Circulating endothelial progenitor cells and cardiovascular outcomes. , 2005, The New England journal of medicine.