Association between asthma-related phenotypes and the CC16 A38G polymorphism in an unselected population of young adult Danes

[1]  J. Goldblatt,et al.  Associations of the IL12B promoter polymorphism in longitudinal data from asthmatic patients 7 to 42 years of age. , 2004, The Journal of allergy and clinical immunology.

[2]  Eric Boerwinkle,et al.  Comparison of strategies for selecting single nucleotide polymorphisms for case/control association studies , 2003, Human Genetics.

[3]  T. Beaty,et al.  Evidence for asthma susceptibility genes on chromosome 11 in an African-American population , 2003, Human Genetics.

[4]  K. Deichmann,et al.  Clara cell protein 16 (CC16) gene polymorphism influences the degree of airway responsiveness in asthmatic children. , 2003, The Journal of allergy and clinical immunology.

[5]  C. Porsbjerg,et al.  Factors associated with asthma in young Danish adults. , 2002, Annals of allergy, asthma & immunology : official publication of the American College of Allergy, Asthma, & Immunology.

[6]  R. Strange,et al.  An association study between the Clara cell secretory protein CC16 A38G polymorphism and asthma phenotypes , 2002, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.

[7]  J. S. Lee,et al.  Uteroglobin gene polymorphisms affect the progression of immunoglobulin A nephropathy by modulating the level of uteroglobin expression. , 2001, Pharmacogenetics.

[8]  M. Landi,et al.  [The natural history of asthma]. , 1998, Recenti progressi in medicina.

[9]  C. Hermans,et al.  Clearance of Clara cell secretory protein 16 (CC16) and surfactant proteins A and B from blood in acute respiratory failure. , 1998, American journal of respiratory and critical care medicine.

[10]  B. Stripp,et al.  Clara cell secretory protein decreases lung inflammation after acute virus infection. , 1998, American journal of physiology. Lung cellular and molecular physiology.

[11]  B. Stripp,et al.  Clara cell secretory protein deficiency increases oxidant stress response in conducting airways. , 1998, American journal of physiology. Lung cellular and molecular physiology.

[12]  T. Shirakawa,et al.  Negative association between asthma and variants of CC16(CC10) on chromosome 11q13 in British and Japanese populations , 1998, Human Genetics.

[13]  P. Burton,et al.  A polymorphism of the CC16 gene is associated with an increased risk of asthma. , 1998, Journal of medical genetics.

[14]  G. Michalopoulos,et al.  Antagonistic Effects of Dexamethasone and Retinoic Acid on Rat Lung Morphogenesis , 1998, Pediatric Research.

[15]  G. Michalopoulos,et al.  Glucocorticoid-Induced Effects on Pattern Formation and Epithelial Cell Differentiation in Early Embryonic Rat Lungs , 1998, Pediatric Research.

[16]  T. Shirakawa,et al.  Association between asthma and an intragenic variant of CC16 on chromosome 11q13 , 1998, Clinical genetics.

[17]  B. Stripp,et al.  Altered pulmonary response to hyperoxia in Clara cell secretory protein deficient mice. , 1997, American journal of respiratory cell and molecular biology.

[18]  A. Dirksen,et al.  Risk factors for development of asthma in children and adolescents: findings from a longitudinal population study. , 1996, Respiratory medicine.

[19]  L. Lim,et al.  Thyroid transcription factor-1, hepatocyte nuclear factor-3beta, surfactant protein B, C, and Clara cell secretory protein in developing mouse lung. , 1996, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.

[20]  G. Lathrop,et al.  A genome-wide search for quantitative trait loci underlying asthma , 1996, Nature.

[21]  R. Crystal,et al.  Human CC10 gene expression in airway epithelium and subchromosomal locus suggest linkage to airway disease. , 1995, The American journal of physiology.

[22]  M. De Ley,et al.  Potent inhibition of both human interferon-gamma production and biologic activity by the Clara cell protein CC16. , 1995, American journal of respiratory cell and molecular biology.

[23]  R. Lauwerys,et al.  Early decrease of serum Clara cell protein in silica-exposed workers. , 1994, The European respiratory journal.

[24]  E. M. Laursen,et al.  Atopy and bronchial responsiveness in random population sample of 527 children and adolescents. , 1992, Annals of allergy.

[25]  J. Faux,et al.  LINKAGE BETWEEN IMMUNOGLOBULIN E RESPONSES UNDERLYING ASTHMA AND RHINITIS AND CHROMOSOME 11q , 1989, The Lancet.

[26]  Shirley A. Miller,et al.  A simple salting out procedure for extracting DNA from human nucleated cells. , 1988, Nucleic acids research.

[27]  A. Mukherjee,et al.  Uteroglobin: structure, molecular biology, and new perspectives on its function as a phospholipase A2 inhibitor. , 1987, Endocrine reviews.

[28]  A. Mukherjee,et al.  Uteroglobin inhibits phospholipase A2 activity. , 1986, Life sciences.

[29]  R. Townley,et al.  Specificity and sensitivity of methacholine inhalation challenge in normal and asthmatic children. , 1984, The Journal of allergy and clinical immunology.

[30]  B. Ferris,et al.  Epidemiology Standardization Project (American Thoracic Society). , 1978, The American review of respiratory disease.

[31]  F. Hargreave,et al.  Bronchial reactivity to inhaled histamine: a method and clinical survey , 1977, Clinical allergy.

[32]  J. Turner,et al.  DOES T4 TOXICOSIS EXIST ? , 1975, The Lancet.

[33]  P. Burton,et al.  Association between plasma CC16 levels, the A38G polymorphism, and asthma. , 2000, American journal of respiratory and critical care medicine.

[34]  S. Hashimoto,et al.  Monkey Clara cell 10 kDa protein (CC10): a characterization of the amino acid sequence with an evolutional comparison with humans, rabbits, rats, and mice. , 1996, American journal of respiratory cell and molecular biology.

[35]  C. Ulrik Comparison of stayers, dropouts, and newcomers in a longitudinal population study of asthma and bronchial hyperresponsiveness: introduction of bias? , 1995, The Journal of asthma : official journal of the Association for the Care of Asthma.

[36]  R. Townley,et al.  The presence of airway reactivity before the development of asthma. , 1990, The American review of respiratory disease.