MicroRNA-223 controls susceptibility to tuberculosis by regulating lung neutrophil recruitment.
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J. Schreiber | H. Mollenkopf | S. Kaufmann | M. Farinacci | G. Nouailles | D. Goletti | A. Dorhoi | S. Jörg | R. Capparelli | M. Iannaccone | Pedro Moura-Alves | Delia Loewe | F. Del Nonno | K. Faé | Karin Hahnke | Dagmar Oberbeck-Mueller | E. Heinemann | Delia Löwe | Dagmar Oberbeck-Müller
[1] Mrutyunjay Suar,et al. Mycobacterium tuberculosis Controls MicroRNA-99b (miR-99b) Expression in Infected Murine Dendritic Cells to Modulate Host Immunity* , 2012, The Journal of Biological Chemistry.
[2] Anna K Rieger,et al. NLRP3 Inflammasome Activity Is Negatively Controlled by miR-223 , 2012, The Journal of Immunology.
[3] Xuetao Cao,et al. Inducible MicroRNA-223 Down-Regulation Promotes TLR-Triggered IL-6 and IL-1β Production in Macrophages by Targeting STAT3 , 2012, PloS one.
[4] Leon N. Schulte,et al. The mammalian microRNA response to bacterial infections , 2012, RNA biology.
[5] Stefan H. E. Kaufmann,et al. Common patterns and disease-related signatures in tuberculosis and sarcoidosis , 2012, Proceedings of the National Academy of Sciences.
[6] Ryan M. O’Connell,et al. microRNA regulation of inflammatory responses. , 2012, Annual review of immunology.
[7] J. Ernst,et al. Tuberculosis pathogenesis and immunity. , 2012, Annual review of pathology.
[8] A. Luster,et al. Lipid‐cytokine‐chemokine cascades orchestrate leukocyte recruitment in inflammation , 2012, Journal of leukocyte biology.
[9] J. Ernst,et al. Mycobacterium tuberculosis inhibits neutrophil apoptosis, leading to delayed activation of naive CD4 T cells. , 2012, Cell host & microbe.
[10] B. Nandi,et al. Regulation of neutrophils by interferon-γ limits lung inflammation during tuberculosis infection , 2011, The Journal of experimental medicine.
[11] Yurong Fu,et al. Circulating MicroRNAs in Patients with Active Pulmonary Tuberculosis , 2011, Journal of Clinical Microbiology.
[12] Qian Gao,et al. Comparative miRNA Expression Profiles in Individuals with Latent and Active Tuberculosis , 2011, PloS one.
[13] Murugesan V. S. Rajaram,et al. Mycobacterium tuberculosis lipomannan blocks TNF biosynthesis by regulating macrophage MAPK-activated protein kinase 2 (MK2) and microRNA miR-125b , 2011, Proceedings of the National Academy of Sciences.
[14] Ryan M. O’Connell,et al. MicroRNA function in myeloid biology. , 2011, Blood.
[15] Xiongfei Xu,et al. The microRNA miR-29 controls innate and adaptive immune responses to intracellular bacterial infection by targeting interferon-γ , 2011, Nature Immunology.
[16] A. Prince,et al. Innate immunity in the respiratory epithelium. , 2011, American journal of respiratory cell and molecular biology.
[17] J. Ernst,et al. Lung Neutrophils Facilitate Activation of Naive Antigen-Specific CD4+ T Cells during Mycobacterium tuberculosis Infection , 2011, The Journal of Immunology.
[18] Xinjing Wang,et al. Modulation of T cell cytokine production by miR-144* with elevated expression in patients with pulmonary tuberculosis. , 2011, Molecular immunology.
[19] G. Kaplan,et al. Specific T cell frequency and cytokine expression profile do not correlate with protection against tuberculosis after bacillus Calmette-Guérin vaccination of newborns. , 2010, American journal of respiratory and critical care medicine.
[20] Shyamasree Datta,et al. Diversity in post-transcriptional control of neutrophil chemoattractant cytokine gene expression. , 2010, Cytokine.
[21] T. Zhu,et al. miR-223 and miR-142 attenuate hematopoietic cell proliferation, and miR-223 positively regulates miR-142 through LMO2 isoforms and CEBP-β , 2010, Cell Research.
[22] Yan-Yan Zhang,et al. MicroRNAs modulate the noncanonical transcription factor NF-kappaB pathway by regulating expression of the kinase IKKalpha during macrophage differentiation. , 2010 .
[23] W. Filipowicz,et al. The widespread regulation of microRNA biogenesis, function and decay , 2010, Nature Reviews Genetics.
[24] Virginia Pascual,et al. An Interferon-Inducible Neutrophil-Driven Blood Transcriptional Signature in Human Tuberculosis , 2010, Nature.
[25] Chuan Yi Tang,et al. Computational modeling with forward and reverse engineering links signaling network and genomic regulatory responses: NF-κB signaling-induced gene expression responses in inflammation , 2010, BMC Bioinformatics.
[26] R. D. de Boer,et al. In Mice, Tuberculosis Progression Is Associated with Intensive Inflammatory Response and the Accumulation of Gr-1dim Cells in the Lungs , 2010, PloS one.
[27] H. Mollenkopf,et al. The adaptor molecule CARD9 is essential for tuberculosis control , 2010, The Journal of experimental medicine.
[28] Ryan M. O’Connell,et al. Physiological and pathological roles for microRNAs in the immune system , 2010, Nature Reviews Immunology.
[29] F. Tacchini-Cottier,et al. Neutrophil-Derived CCL3 Is Essential for the Rapid Recruitment of Dendritic Cells to the Site of Leishmania major Inoculation in Resistant Mice , 2010, PLoS pathogens.
[30] Xinxia Peng,et al. MicroRNA Expression and Virulence in Pandemic Influenza Virus-Infected Mice , 2010, Journal of Virology.
[31] C. Croce. Causes and consequences of microRNA dysregulation in cancer , 2009, Nature Reviews Genetics.
[32] M. Biffoni,et al. MicroRNA 223-dependent expression of LMO2 regulates normal erythropoiesis , 2009, Haematologica.
[33] Tongbin Li,et al. miRecords: an integrated resource for microRNA–target interactions , 2008, Nucleic Acids Res..
[34] Heidi J. Peltier,et al. Normalization of microRNA expression levels in quantitative RT-PCR assays: identification of suitable reference RNA targets in normal and cancerous human solid tissues. , 2008, RNA.
[35] O. Kirak,et al. Regulation of progenitor cell proliferation and granulocyte function by microRNA-223 , 2008, Nature.
[36] S. Kaufmann,et al. Poor correlation between BCG vaccination-induced T cell responses and protection against tuberculosis , 2007, Proceedings of the National Academy of Sciences.
[37] R. Hunter,et al. Pathology of postprimary tuberculosis in humans and mice: contradiction of long-held beliefs. , 2007, Tuberculosis.
[38] Yoko Fukuda,et al. An Evolutionarily Conserved Mechanism for MicroRNA-223 Expression Revealed by MicroRNA Gene Profiling , 2007, Cell.
[39] P. V. van Helden,et al. Immune markers measured before treatment predict outcome of intensive phase tuberculosis therapy , 2006, Clinical and experimental immunology.
[40] P. V. van Helden,et al. Changes in leucocyte and lymphocyte subsets during tuberculosis treatment; prominence of CD3dimCD56+ natural killer T cells in fast treatment responders , 2006, Clinical and experimental immunology.
[41] S. Ehlers,et al. Genetically Determined Susceptibility to Tuberculosis in Mice Causally Involves Accelerated and Enhanced Recruitment of Granulocytes , 2006, Infection and Immunity.
[42] S. Kaufmann. Tuberculosis: back on the immunologists' agenda. , 2006, Immunity.
[43] R. Ransohoff,et al. The many roles of chemokines and chemokine receptors in inflammation. , 2006, The New England journal of medicine.
[44] Alessandro Fatica,et al. A Minicircuitry Comprised of MicroRNA-223 and Transcription Factors NFI-A and C/EBPα Regulates Human Granulopoiesis , 2005, Cell.
[45] A. Apt,et al. Neutrophil Responses to Mycobacterium tuberculosis Infection in Genetically Susceptible and Resistant Mice , 2005, Infection and Immunity.
[46] R. Strieter,et al. CXC chemokines in angiogenesis of cancer. , 2004, Seminars in cancer biology.
[47] J. Major,et al. Neutrophil chemoattractant genes KC and MIP‐2 are expressed in different cell populations at sites of surgical injury , 2004, Journal of leukocyte biology.
[48] Thomas D. Schmittgen,et al. Analysis of relative gene expression data using real-time quantitative PCR and the 2(-Delta Delta C(T)) Method. , 2001, Methods.
[49] I. Orme,et al. Interleukin-6 Induces Early Gamma Interferon Production in the Infected Lung but Is Not Required for Generation of Specific Immunity to Mycobacterium tuberculosisInfection , 2000, Infection and Immunity.
[50] S. McColl,et al. Regulation of chemokine gene expression in human peripheral blood neutrophils phagocytosing microbial pathogens. , 1998, Journal of immunology.
[51] D. Link,et al. Interleukin-6 and the granulocyte colony-stimulating factor receptor are major independent regulators of granulopoiesis in vivo but are not required for lineage commitment or terminal differentiation. , 1997, Blood.
[52] J. Farber. A macrophage mRNA selectively induced by gamma-interferon encodes a member of the platelet factor 4 family of cytokines. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[53] R. Mertelsmann,et al. Inducible production of interleukin-6 by human polymorphonuclear neutrophils: role of granulocyte-macrophage colony-stimulating factor and tumor necrosis factor-alpha. , 1990, Blood.
[54] S. Jhanwar,et al. Interferon-inducible gene maps to a chromosomal band associated with a (4;11) translocation in acute leukemia cells. , 1987, Proceedings of the National Academy of Sciences of the United States of America.
[55] R. Wilkinson,et al. Neutrophils in tuberculosis: friend or foe? , 2012, Trends in immunology.
[56] W. Hammerschmidt,et al. This information is current as Production β Inflammasome and IL-1 miR-BART 15 Regulate the NLRP 3 Cutting Edge : miR-223 and EBV , 2012 .
[57] Sang-Nae Cho,et al. Neutrophils are the predominant infected phagocytic cells in the airways of patients with active pulmonary TB. , 2010, Chest.
[58] Carl Nathan,et al. Nonresolving Inflammation , 2010, Cell.
[59] Rob Knight,et al. BMC Bioinformatics BioMed Central , 2008 .
[60] S. Kaufmann,et al. Lethal tuberculosis in interleukin-6-deficient mutant mice , 1997, Infection and immunity.
[61] A. Cerami,et al. Identification and characterization of macrophage inflammatory protein 2. , 1989, Proceedings of the National Academy of Sciences of the United States of America.