Differential MHC class II component expression in HPV‐positive cervical cancer cells: Implication for immune surveillance
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H. Pilch | M. Maeurer | I. Zehbe | H. Höhn | C. Neukirch | K. Freitag
[1] M. Toyota,et al. Epigenetic inactivation of class II transactivator (CIITA) is associated with the absence of interferon-γ-induced HLA-DR expression in colorectal and gastric cancer cells , 2004, Oncogene.
[2] G. Hämmerling,et al. Mini‐review: Overcoming tumor‐intrinsic resistance to immune effector function , 2004, European journal of immunology.
[3] M. Klemsz,et al. Cutting Edge: Induction of the Antigen-Processing Enzyme IFN-γ-Inducible Lysosomal Thiol Reductase in Melanoma Cells Is STAT1-Dependent but CIITA-Independent1 , 2004, The Journal of Immunology.
[4] F. Koning,et al. Dissection of the HLA-DR4 Peptide Repertoire in Endocrine Epithelial Cells: Strong Influence of Invariant Chain and HLA-DM Expression on the Nature of Ligands1 , 2004, The Journal of Immunology.
[5] R. Gutzmer,et al. A Tumor-Associated Glycoprotein That Blocks MHC Class II-Dependent Antigen Presentation by Dendritic Cells1 , 2004, The Journal of Immunology.
[6] G. Jakiel,et al. Alterations in the expression of selected MHC antigens in premalignant lesions and squamous carcinomas of the uterine cervix , 2003, Acta obstetricia et gynecologica Scandinavica.
[7] E. Unanue,et al. Cutting Edge: H-2DM Is Responsible for the Large Differences in Presentation Among Peptides Selected by I-Ak During Antigen Processing 1 , 2003, The Journal of Immunology.
[8] J. Elliott,et al. Editing of an Immunodominant Epitope of Glutamate Decarboxylase by HLA-DM1 , 2003, The Journal of Immunology.
[9] Soldano Ferrone,et al. Tumors as elusive targets of T-cell-based active immunotherapy. , 2003, Trends in immunology.
[10] Ed Schuuring,et al. Mutations in the HLA class II genes leading to loss of expression of HLA-DR and HLA-DQ in diffuse large B-cell lymphoma , 2003, Immunogenetics.
[11] L. Eisenlohr,et al. Presentation by Recycling MHC Class II Molecules of an Influenza Hemagglutinin-Derived Epitope That Is Revealed in the Early Endosome by Acidification1 , 2003, The Journal of Immunology.
[12] S. Ferrone,et al. HLA class I antigen loss, tumor immune escape and immune selection. , 2002, Vaccine.
[13] H. Hausen. Papillomaviruses and cancer: from basic studies to clinical application , 2002, Nature Reviews Cancer.
[14] J. Kim,et al. Alterations of HLA class I and II antigen expression in preinvasive, invasive and metastatic cervical cancers , 2001, Experimental & Molecular Medicine.
[15] T. Reichert,et al. H2-Mβ1 and H2-Mβ2 Heterodimers Equally Promote CLIP Removal in I-Aq Molecules from Autoimmune-prone DBA/1 Mice* , 2001, The Journal of Biological Chemistry.
[16] E. Schmitt,et al. MHC class II antigen presentation pathway in murine tumours: tumour evasion from immunosurveillance? , 2000, British Journal of Cancer.
[17] E. Schmitt,et al. H2-M, a facilitator of MHC class II peptide loading, and its negative modulator H2-O are differentially expressed in response to proinflammatory cytokines , 2000, Immunogenetics.
[18] H. Pilch,et al. Human Papillomavirus Type 33 E7 Peptides Presented by HLA-DR*0402 to Tumor-Infiltrating T Cells in Cervical Cancer , 2000, Journal of Virology.
[19] P. Magnusson,et al. Cervical cancer risk: is there a genetic component? , 2000, Molecular medicine today.
[20] B. Seliger,et al. CD4+ tumor-infiltrating lymphocytes in cervical cancer recognize HLA-DR-restricted peptides provided by human papillomavirus-E7. , 1999, Journal of immunology.
[21] M. Sospedra,et al. HLA-DM and invariant chain are expressed by thyroid follicular cells, enabling the expression of compact DR molecules. , 1999, International immunology.
[22] L. Eisenlohr,et al. Antigen processing of two H2-IEd-restricted epitopes is differentially influenced by the structural changes in a viral glycoprotein. , 1998, Journal of immunology.
[23] I. Mellman,et al. Developmental Regulation of Invariant Chain Proteolysis Controls MHC Class II Trafficking in Mouse Dendritic Cells , 1998, Cell.
[24] Eric O Long,et al. Peptide loading onto recycling HLA‐DR molecules occurs in early endosomes , 1998, European journal of immunology.
[25] P. Delvenne,et al. Correlation of T‐helper secretory differentiation and types of antigen‐presenting cells in squamous intraepithelial lesions of the uterine cervix , 1998, The Journal of pathology.
[26] E. Wilander,et al. Human papillomavirus 16 E6 variants are more prevalent in invasive cervical carcinoma than the prototype. , 1998, Cancer research.
[27] A. Rudensky,et al. Major Histocompatibility Complex Class II Compartments in Human and Mouse B Lymphoblasts Represent Conventional Endocytic Compartments , 1997, The Journal of cell biology.
[28] C. Harding,et al. Early endosomes and a late endocytic compartment generate different peptide-class II MHC complexes via distinct processing mechanisms. , 1997, Journal of immunology.
[29] E. Unanue,et al. Distinct antigen MHC class II complexes generated by separate processing pathways. , 1996, The EMBO journal.
[30] E. Wilander,et al. Detection of human papillomavirus in cervical intra-epithelial neoplasia, using in situ hybridization and various polymerase chain reaction techniques , 1996, Virchows Archiv.
[31] S. Beaudenon,et al. Two novel genital human papillomavirus (HPV) types, HPV68 and HPV70, related to the potentially oncogenic HPV39 , 1996, Journal of clinical microbiology.
[32] B. Mach,et al. A novel antigen-processing-defective phenotype in major histocompatibility complex class II-positive CIITA transfectants is corrected by interferon-gamma , 1995, The Journal of experimental medicine.
[33] M. Stanley,et al. Immunological events in regressing genital warts. , 1994, American journal of clinical pathology.
[34] C. Viret,et al. Defective lymphokine production by most CD8+ and CD4+ tumor‐specific T cell clones derived from human melanoma‐infiltrating lymphocytes in response to autologous tumor cells in vitro , 1994, European journal of immunology.
[35] B. Mach,et al. Regulation of MHC class II expression by interferon-gamma mediated by the transactivator gene CIITA. , 1994, Science.
[36] Eric O Long,et al. Two processing pathways for the MHC class II-restricted presentation of exogenous influenza virus antigen. , 1994, Journal of immunology.
[37] M. Stanley,et al. Analysis of HLA‐DR expression on keratinocytes in cervical neoplasia , 1994, International journal of cancer.
[38] C. Meijer,et al. Loss of transporter protein, encoded by the TAP-1 gene, is highly correlated with loss of HLA expression in cervical carcinomas , 1994, The Journal of experimental medicine.
[39] C. Meijer,et al. Analysis of MHC class I and II expression in relation to presence of HPV genotypes in premalignant and malignant cervical lesions. , 1993, British Journal of Cancer.
[40] Barbara J. Reaves,et al. Epitope mapping of two isoforms of a trans Golgi network specific integral membrane protein TGN38/41 , 1992, FEBS letters.
[41] H. Delius,et al. Characterization of a novel human papillomavirus DNA in the cervical carcinoma cell line ME180 , 1991, Journal of virology.
[42] T. Reichert,et al. H2-Mbeta 1 and H2-Mbeta 2 heterodimers equally promote clip removal in I-A(q) molecules from autoimmune-prone DBA/1 mice. , 2001, The Journal of biological chemistry.
[43] R. Rappuoli,et al. The small GTP binding protein rab7 is essential for cellular vacuolation induced by Helicobacter pylori cytotoxin , 1997, The EMBO journal.
[44] G. Hämmerling,et al. Selection of the MHC Class II-associated peptide repertoire by HLA-DM , 1997, Immunologic research.
[45] G. Fleuren,et al. The expression of histocompatibility-related leukocyte antigens in the pathway to cervical carcinoma. , 1994, American journal of clinical pathology.
[46] C. Meijer,et al. HLA expression in pre-invasive cervical neoplasia in relation to human papilloma virus infection. , 1993, European journal of cancer.