Genome-wide appraisal of thyroid cancer progression.
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J. Shah | Bhuvanesh Singh | P. Rao | R. Ghossein | R. Tuttle | A. Shaha | V. Wreesmann | E. Schnaser | C. Harris | S. Patel | Jatin P. Shah
[1] G. Brabant,et al. Gene expression of differentiation- and dedifferentiation markers in normal and malignant human thyroid tissues. , 2009, Experimental and clinical endocrinology.
[2] G. Viglietto,et al. Isolation of a SIR-like gene, SIR-T8, that is overexpressed in thyroid carcinoma cell lines and tissues , 2002, British Journal of Cancer.
[3] D. Stoler,et al. Genomic instability measurement in the diagnosis of thyroid neoplasms , 2002, Head & neck.
[4] C. Larsson,et al. Gain of 1q and loss of 9q21.3‐q32 are associated with a less favorable prognosis in papillary thyroid carcinoma , 2001, Genes, chromosomes & cancer.
[5] E. Speel,et al. Genetic evidence for early divergence of small functioning and nonfunctioning endocrine pancreatic tumors: gain of 9Q34 is an early event in insulinomas. , 2001, Cancer research.
[6] E. Speel,et al. Putative tumor suppressor loci at 6q22 and 6q23-q24 are involved in the malignant progression of sporadic endocrine pancreatic tumors. , 2001, The American journal of pathology.
[7] J. Shah,et al. Molecular cytogenetic characterization of head and neck squamous cell carcinoma and refinement of 3q amplification. , 2001, Cancer research.
[8] H. Clevers,et al. Mutations in the APC tumour suppressor gene cause chromosomal instability , 2001, Nature Cell Biology.
[9] D. Rimm,et al. β-Catenin Dysregulation in Thyroid Neoplasms : Down-Regulation, Aberrant Nuclear Expression, and CTNNB1 Exon 3 Mutations Are Markers for Aggressive Tumor Phenotypes and Poor Prognosis , 2001 .
[10] T. Dwight,et al. Sporadic and familial pheochromocytomas are associated with loss of at least two discrete intervals on chromosome 1p. , 2000, Cancer research.
[11] F. de Nigris,et al. FRA-1 expression in hyperplastic and neoplastic thyroid diseases. , 2000, Clinical cancer research : an official journal of the American Association for Cancer Research.
[12] C. Larsson,et al. Patterns of chromosomal imbalances in parathyroid carcinomas. , 2000, The American journal of pathology.
[13] R. Lloyd,et al. The Role of Cell Cycle Regulatory Protein, Cyclin D1, in the Progression of Thyroid Cancer , 2000, Modern Pathology.
[14] S. Mccormick,et al. Improving degenerate oligonucleotide primed PCR‐comparative genomic hybridization for analysis of DNA copy number changes in tumors , 2000, Genes, chromosomes & cancer.
[15] H. Bonjer,et al. Losses of chromosomes 1p and 3q are early genetic events in the development of sporadic pheochromocytomas. , 2000, The American journal of pathology.
[16] C. Larsson,et al. Chromosomal alterations in 15 breast cancer cell lines by comparative genomic hybridization and spectral karyotyping , 2000, Genes, chromosomes & cancer.
[17] J. Tchinda,et al. Aberrations of chromosomes 5 and 8 as recurrent cytogenetic events in anaplastic carcinoma of the thyroid as detected by fluorescence in situ hybridisation and comparative genomic hybridisation , 2000, Virchows Archiv.
[18] M. Emi,et al. Allelotyping of anaplastic thyroid carcinoma: Frequent allelic losses on 1q, 9p, 11, 17, 19p, and 22q , 2000, Genes, chromosomes & cancer.
[19] K. Kinzler,et al. Genetic instability and darwinian selection in tumours. , 1999, Trends in cell biology.
[20] A. Fusco,et al. A KRAB zinc finger protein gene is the potential target of 19q13 translocation in benign thyroid tumors , 1999, Genes, chromosomes & cancer.
[21] H. Rabes,et al. The transcription coactivator HTIF1 and a related protein are fused to the RET receptor tyrosine kinase in childhood papillary thyroid carcinomas , 1999, Oncogene.
[22] K. Franssila,et al. DNA copy number changes in thyroid carcinoma. , 1999, The American journal of pathology.
[23] R. Heim,et al. Using GFP in FRET-based applications. , 1999, Trends in cell biology.
[24] P. Meltzer,et al. Specific chromosomal aberrations and amplification of the AIB1 nuclear receptor coactivator gene in pancreatic carcinomas. , 1999, The American journal of pathology.
[25] O. Ozaki,et al. Anaplastic transformation of papillary thyroid carcinoma in recurrent disease in regional lymph nodes: A histologic and immunohistochemical study , 1999, Journal of surgical oncology.
[26] K. Kinzler,et al. Genetic instabilities in human cancers , 1998, Nature.
[27] I. Fleming,et al. A National Cancer Data Base report on 53,856 cases of thyroid carcinoma treated in the U.S., 1985‐1995 , 1998, Cancer.
[28] S. Piantadosi,et al. Polymerase chain reaction-based microsatellite polymorphism analysis of follicular and Hürthle cell neoplasms of the thyroid. , 1998, The Journal of clinical endocrinology and metabolism.
[29] L. Meisner,et al. Use of nonbreakpoint DNA probes to detect the t(X;18) in interphase cells from synovial sarcoma: implications for detection of diagnostic tumor translocations. , 1998, The American journal of pathology.
[30] K. Hoang-Xuan,et al. Determination of the replication error phenotype in human tumors without the requirement for matching normal DNA by analysis of mononucleotide repeat microsatellites , 1998, Genes, chromosomes & cancer.
[31] J. Soares,et al. Cytogenetic investigations of 340 thyroid hyperplasias and adenomas revealing correlations between cytogenetic findings and histology. , 1998, Cancer genetics and cytogenetics.
[32] P. Goodfellow,et al. Allelotype of follicular thyroid carcinomas reveals genetic instability consistent with frequent nondisjunctional chromosomal loss , 1997, Genes, chromosomes & cancer.
[33] J. Califano,et al. 11:40 AM: A Genetic Progression Model for Head and Neck Cancer: Implications for Field Cancerization , 1996 .
[34] M. Borrello,et al. Cytogenetics and molecular genetics of carcinomas arising from thyroid epithelial follicular cells , 1996, Genes, chromosomes & cancer.
[35] B. Scheithauer,et al. Frequent loss of heterozygosity at the retinoblastoma susceptibility gene (RB) locus in aggressive pituitary tumors: evidence for a chromosome 13 tumor suppressor gene other than RB. , 1995, Cancer research.
[36] D. Pinkel,et al. Deficiency of p53 accelerates mammary tumorigenesis in Wnt-1 transgenic mice and promotes chromosomal instability. , 1995, Genes & development.
[37] S. Devries,et al. Analysis of changes in DNA sequence copy number by comparative genomic hybridization in archival paraffin-embedded tumor samples. , 1994, The American journal of pathology.
[38] M. Pierotti,et al. Gene p53 mutations are restricted to poorly differentiated and undifferentiated carcinomas of the thyroid gland. , 1993, The Journal of clinical investigation.
[39] D. Pinkel,et al. Comparative Genomic Hybridization for Molecular Cytogenetic Analysis of Solid Tumors , 2022 .
[40] R. Berger,et al. Cytogenetic studies on 19 papillary thyroid carcinomas , 1992, Genes, chromosomes & cancer.
[41] T. Mikkelsen,et al. Clonal expansion of p53 mutant cells is associated with brain tumour progression , 1992, Nature.
[42] N. Ordóñez,et al. Anaplastic carcinoma of the thyroid: A clinicopathologic study of 121 cases , 1990, Cancer.
[43] B. Vogelstein,et al. A genetic model for colorectal tumorigenesis , 1990, Cell.
[44] Y. Nakamura,et al. Genetic alterations during colorectal-tumor development. , 1988, The New England journal of medicine.
[45] L. Woolner,et al. Undifferentiated and poorly differentiated carcinoma. , 1985, Seminars in diagnostic pathology.
[46] J. Rosai,et al. Anaplastic thyroid carcinoma. A study of 70 cases. , 1985, American journal of clinical pathology.
[47] J. Rosai,et al. Poorly differentiated (“insular”) thyroid carcinoma: A reinterpretation of Langhans' “wuchernde Struma” , 1984, The American journal of surgical pathology.
[48] A. Sakamoto,et al. Poorly differentiated carcinoma of the thyroid. A clinicopathologic entity for a high‐risk group of papillary and follicular carcinomas , 1983, Cancer.
[49] J. Rosai,et al. Tumors of the thyroid gland , 1992 .
[50] R. Miller,et al. Anaplastic Thyroid Carcinoma: Association With Differentiated Thyroid Cancer , 1988 .