Manganese inhibits NMDA receptor channel function: implications to psychiatric and cognitive effects.

[1]  Laurence Mir.  Dose-effect relationship between manganese exposure and neurological, neuropsychological and pulmonary function in confined space bridge welders. , 2007 .

[2]  M. Panisset,et al.  Neuropsychiatric symptoms and past manganese exposure in a ferro-alloy plant. , 2007, Neurotoxicology.

[3]  J. Coyle,et al.  Endogenous N‐acetylaspartylglutamate reduced NMDA receptor‐dependent current neurotransmission in the CA1 area of the hippocampus 1 , 2007, Journal of neurochemistry.

[4]  T. Guilarte,et al.  Nigrostriatal dopamine system dysfunction and subtle motor deficits in manganese-exposed non-human primates , 2006, Experimental Neurology.

[5]  T. Guilarte,et al.  Evidence for cortical dysfunction and widespread manganese accumulation in the nonhuman primate brain following chronic manganese exposure: a 1H-MRS and MRI study. , 2006, Toxicological sciences : an official journal of the Society of Toxicology.

[6]  T. Guilarte,et al.  Effects of chronic manganese exposure on cognitive and motor functioning in non-human primates , 2006, Brain Research.

[7]  R. Bowler,et al.  Dose–effect relationships between manganese exposure and neurological, neuropsychological and pulmonary function in confined space bridge welders , 2006, Occupational and Environmental Medicine.

[8]  N. Kumar,et al.  Neuropsychological profiles of manganese neurotoxicity , 2006, European journal of neurology.

[9]  D. Dorman,et al.  Tissue manganese concentrations in young male rhesus monkeys following subchronic manganese sulfate inhalation. , 2006, Toxicological sciences : an official journal of the Society of Toxicology.

[10]  M. Trenerry,et al.  Neurologic manifestations in welders with pallidal MRI T1 hyperintensity , 2005, Neurology.

[11]  Zhongqi Cheng,et al.  Water Manganese Exposure and Children’s Intellectual Function in Araihazar, Bangladesh , 2004, Environmental health perspectives.

[12]  N. Sotogaku,et al.  Manganese influences the levels of neurotransmitters in synapses in rat brain , 2002, Neuroscience.

[13]  H. Hudnell,et al.  Manganese accentuates adverse mental health effects associated with alcohol use disorders , 2002, Biological Psychiatry.

[14]  A. Woolf,et al.  A child with chronic manganese exposure from drinking water. , 2002, Environmental health perspectives.

[15]  R. V. Van Heertum,et al.  Prefrontal Dopamine D1 Receptors and Working Memory in Schizophrenia , 2002, The Journal of Neuroscience.

[16]  J. Krystal,et al.  NMDA receptor regulation of memory and behavior in humans , 2001, Hippocampus.

[17]  J. Texcalac-Sangrador,et al.  Exposure to manganese: health effects on the general population, a pilot study in central Mexico. , 2001, Environmental research.

[18]  P. Goldman-Rakic,et al.  The physiological approach: functional architecture of working memory and disordered cognition in schizophrenia , 1999, Biological Psychiatry.

[19]  T. Guilarte,et al.  Divalent cations modulate N-methyl-D-aspartate receptor function at the glycine site. , 1999, The Journal of pharmacology and experimental therapeutics.

[20]  A Beuter,et al.  Manganese neurotoxicity, a continuum of dysfunction: results from a community based study. , 1999, Neurotoxicology.

[21]  J. Coyle.  The Nagging Question of the Function ofN-Acetylaspartylglutamate , 1997, Neurobiology of Disease.

[22]  D Mergler,et al.  Early manifestations of manganese neurotoxicity in humans: an update. , 1997, Environmental research.

[23]  L. Vyklický,et al.  Copper Modulation of NMDA Responses in Mouse and Rat Cultured Hippocampal Neurons , 1996, The European journal of neuroscience.

[24]  G. Shepherd,et al.  Differential modulation by zinc and copper of amino acid receptors from rat olfactory bulb neurons. , 1996, Journal of neurophysiology.

[25]  B. Sakmann,et al.  Developmental and regional expression in the rat brain and functional properties of four NMDA receptors , 1994, Neuron.

[26]  B Sjögren,et al.  Neuropsychiatric symptoms among welders exposed to neurotoxic metals. , 1990, British journal of industrial medicine.

[27]  E. Albuquerque,et al.  Selective blockade of NMDA‐activated channel currents may be implicated in learning deficits caused by lead , 1990, FEBS letters.

[28]  D. Choi,et al.  Effect of zinc on NMDA receptor-mediated channel currents in cortical neurons , 1990, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[29]  F. Hajós,et al.  Nerve endings from rat brain tissue release copper upon depolarization. A possible role in regulating neuronal excitability , 1989, Neuroscience Letters.

[30]  E. Wong,et al.  [3H]MK‐801 Labels a Site on the N‐Methyl‐D‐Aspartate Receptor Channel Complex in Rat Brain Membranes , 1988, Journal of neurochemistry.

[31]  M. Mayer,et al.  Permeation and block of N‐methyl‐D‐aspartic acid receptor channels by divalent cations in mouse cultured central neurones. , 1987, The Journal of physiology.

[32]  J. Tata.  Where's the action in endocrinology? , 1987, Nature.

[33]  Shin-Ho Chung,et al.  Release of endogenous Zn2+ from brain tissue during activity , 1984, Nature.

[34]  J. Wehner,et al.  Effects of N-methyl-d-aspartate antagonism on spatial learning in mice , 2005, Psychopharmacology.

[35]  Christopher Hartney,et al.  Neuropsychological sequelae of exposure to welding fumes in a group of occupationally exposed men. , 2003, International journal of hygiene and environmental health.

[36]  T. Guilarte.  Glutamatergic system and developmental lead neurotoxicity. , 1997, Neurotoxicology.

[37]  H. Haas,et al.  Suppression of long‐term potentiation in hippocampal slices by copper , 1997, Hippocampus.

[38]  T. Guilarte,et al.  Biochemical evidence of an interaction of lead at the zinc allosteric sites of the NMDA receptor complex: effects of neuronal development. , 1995, Neurotoxicology.

[39]  J. Donaldson,et al.  The physiopathologic significance of manganese in brain: its relation to schizophrenia and neurodegenerative disorders. , 1987, Neurotoxicology.

[40]  G. Lynch,et al.  Selective impairment of learning and blockade of long-term potentiation by an N-methyl-D-aspartate receptor antagonist, AP5 , 1986, Nature.