Prognostic impact of trisomies of chromosomes 10, 17, and 5 among children with acute lymphoblastic leukemia and high hyperdiploidy (> 50 chromosomes).
暂无分享,去创建一个
P S Gaynon | N. Heerema | H. Sather | B. Lange | G. Reaman | F. Uckun | M. Sensel | P. Steinherz | P. Gaynon | B. Bostrom | R. Hutchinson | J. Nachman | H N Sather | F. Uckun | R J Hutchinson | N A Heerema | G H Reaman | F M Uckun | P G Steinherz | M G Sensel | J B Nachman | B J Lange | T Zhang | B C Bostrom | T. Zhang | Tracy Zhang | M. G. Sensel
[1] S. Raimondi,et al. Reduced folate carrier expression in acute lymphoblastic leukemia: a mechanism for ploidy but not lineage differences in methotrexate accumulation. , 1999, Blood.
[2] N. Heerema,et al. Treatment outcome and prognostic factors for infants with acute lymphoblastic leukemia treated on two consecutive trials of the Children's Cancer Group. , 1999, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[3] Y. Ravindranath,et al. Reduced folate carrier gene expression in childhood acute lymphoblastic leukemia: relationship to immunophenotype and ploidy. , 1998, Clinical cancer research : an official journal of the American Association for Cancer Research.
[4] F. Behm,et al. Early intensification of intrathecal chemotherapy virtually eliminates central nervous system relapse in children with acute lymphoblastic leukemia. , 1998, Blood.
[5] H. Sather,et al. Augmented post-induction therapy for children with high-risk acute lymphoblastic leukemia and a slow response to initial therapy. , 1998, The New England journal of medicine.
[6] H. Sather,et al. Response of children with high-risk acute lymphoblastic leukemia treated with and without cranial irradiation: a report from the Children's Cancer Group. , 1998, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[7] H. Adams,et al. TEL-AML1 Fusion Transcript in Relapsed Childhood Acute Lymphoblastic Leukemia , 1998 .
[8] H. Sather,et al. Treatment of patients with acute lymphoblastic leukemia with bulky extramedullary disease and T‐cell phenotype or other poor prognostic features , 1998, Cancer.
[9] J. Downing,et al. 12p Abnormalities and the TEL Gene (ETV6) in Childhood Acute Lymphoblastic Leukemia , 1997 .
[10] H. Sather,et al. Early response to therapy and outcome in childhood acute lymphoblastic leukemia , 1997, Cancer.
[11] S. Richards,et al. Cytogenetics and prognosis in childhood lymphoblastic leukaemia: results of MRC UKALL X , 1997, British journal of haematology.
[12] H. Sather,et al. Improved clinical outcome for children with T-lineage acute lymphoblastic leukemia after contemporary chemotherapy: a Children's Cancer Group Study. , 1996, Leukemia & lymphoma.
[13] F. Behm,et al. Heterogeneity of hyperdiploid (51-67) childhood acute lymphoblastic leukemia. , 1996, Leukemia.
[14] R. Pieters,et al. Favorable prognosis of hyperdiploid common acute lymphoblastic leukemia may be explained by sensitivity to antimetabolites and other drugs: results of an in vitro study. , 1995, Blood.
[15] C. C. Bailey,et al. Intensification of treatment and survival in all children with lymphoblastic leukaemia: results of UK Medical Research Council trial UKALL X , 1995, The Lancet.
[16] M. Relling,et al. Blast cell methotrexate-polyglutamate accumulation in vivo differs by lineage, ploidy, and methotrexate dose in acute lymphoblastic leukemia. , 1994, The Journal of clinical investigation.
[17] S. Raimondi. Current Status of Cytogenetic Research in Childhood Acute Lymphoblastic Leukemia , 1993 .
[18] H. Sather,et al. Improved outcome with delayed intensification for children with acute lymphoblastic leukemia and intermediate presenting features: a Childrens Cancer Group phase III trial. , 1993, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[19] A. Look,et al. Accumulation of high levels of methotrexate polyglutamates in lymphoblasts from children with hyperdiploid (greater than 50 chromosomes) B-lineage acute lymphoblastic leukemia: a Pediatric Oncology Group study. , 1992, Blood.
[20] A. Look,et al. Trisomy of leukemic cell chromosomes 4 and 10 identifies children with B-progenitor cell acute lymphoblastic leukemia with a very low risk of treatment failure: a Pediatric Oncology Group study. , 1992, Blood.
[21] F. Behm,et al. Hyperdiploid (47-50) acute lymphoblastic leukemia in children. , 1992, Blood.
[22] L. Frankel,et al. Ploidy of lymphoblasts is the strongest predictor of treatment outcome in B-progenitor cell acute lymphoblastic leukemia of childhood: a Pediatric Oncology Group study. , 1992, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[23] J. Peto,et al. Results of Medical Research Council Childhood Leukaemia Trial UKALL VIII (Report to the Medical Research Council on behalf of the Working Party on Leukaemia in Childhood) , 1991, British journal of haematology.
[24] M. Borowitz,et al. Favorable prognosis associated with hyperdiploidy in children with acute lymphocytic leukemia correlates with extra chromosome 6. A pediatric oncology group study , 1990, Cancer.
[25] C. Bloomfield,et al. Six-year follow-up of the clinical significance of karyotype in acute lymphoblastic leukemia. , 1989, Cancer genetics and cytogenetics.
[26] S. Richards,et al. Chromosomes and other prognostic factors in acute lymphoblastic leukaemia: a long‐term follow‐up , 1989, British journal of haematology.
[27] B. Koller,et al. Biphenotypic leukemic lymphocyte precursors in CD2+CD19+ acute lymphoblastic leukemia and their putative normal counterparts in human fetal hematopoietic tissues. , 1989, Blood.
[28] C. Song,et al. Heterogeneity of cultured leukemic lymphoid progenitor cells from B cell precursor acute lymphoblastic leukemia (ALL) patients. , 1987, The Journal of clinical investigation.
[29] R. Gelber,et al. Four-agent induction and intensive asparaginase therapy for treatment of childhood acute lymphoblastic leukemia. , 1986, The New England journal of medicine.
[30] N. Heerema,et al. Karyotypic and clinical findings in a consecutive series of children with acute lymphocytic leukemia. , 1985, Cancer genetics and cytogenetics.
[31] C. Pui,et al. Prognostic importance of blast cell DNA content in childhood acute lymphoblastic leukemia. , 1985, Blood.
[32] M. Pike,et al. Design and analysis of randomized clinical trials requiring prolonged observation of each patient. II. analysis and examples. , 1977, British Journal of Cancer.
[33] N. Mantel. Evaluation of survival data and two new rank order statistics arising in its consideration. , 1966, Cancer chemotherapy reports.
[34] E. Kaplan,et al. Nonparametric Estimation from Incomplete Observations , 1958 .
[35] F. Behm,et al. Hyperdiploid acute lymphoblastic leukemia with 51 to 65 chromosomes: a distinct biological entity with a marked propensity to undergo apoptosis. , 1999, Blood.
[36] J. Downing,et al. Low frequency of TEL-AML1 in relapsed acute lymphoblastic leukemia supports a favorable prognosis for this genetic subgroup , 1999, Leukemia.
[37] C. Pui,et al. Uniform approach to risk classification and treatment assignment for children with acute lymphoblastic leukemia. , 1996, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[38] G. Henze,et al. Results and significance of six randomized trials in four consecutive ALL-BFM studies. , 1990, Haematology and blood transfusion.
[39] J. Whang‐Peng. Clinical significance of chromosomal abnormalities in acute lymphoblastic leukemia , 1981 .
[40] Iscn. International System for Human Cytogenetic Nomenclature , 1978 .
[41] D. Cox. Regression Models and Life-Tables , 1972 .