Eosinophils recruited to the lung by segmental antigen challenge show a reduced chemotactic response to leukotriene B4.

[1]  S. Peters,et al.  Neutrophils recruited to the lungs of humans by segmental antigen challenge display a reduced chemotactic response to leukotriene B4. , 1993, American journal of respiratory cell and molecular biology.

[2]  P. Weller,et al.  Mechanisms of eosinophil recruitment. , 1993, American journal of respiratory cell and molecular biology.

[3]  S. Peters,et al.  Immunoglobulin E-mediated increase in vascular permeability correlates with eosinophilic inflammation. , 1993, The American review of respiratory disease.

[4]  A. Wardlaw,et al.  Interleukin-5 selectively enhances the chemotactic response of eosinophils obtained from normal but not eosinophilic subjects. , 1992, Blood.

[5]  S. Peters,et al.  Effect of antigen dose on the recruitment of inflammatory cells to the lung by segmental antigen challenge. , 1992, The Journal of allergy and clinical immunology.

[6]  K. Blaser,et al.  An improved immunomagnetic procedure for the isolation of highly purified human blood eosinophils. , 1991, Journal of immunological methods.

[7]  G. Gleich.  The eosinophil and bronchial asthma: current understanding. , 1990, The Journal of allergy and clinical immunology.

[8]  J. Oostveen,et al.  Effect of the selective leukotriene B4 antagonist U-75302 on antigen-induced bronchopulmonary eosinophilia in sensitized guinea pigs. , 1989, The American review of respiratory disease.

[9]  R. Naclerio,et al.  Leukotriene B4 as a mediator of early and late reactions to antigen in humans: the effect of systemic glucocorticoid treatment in vivo. , 1989, The Journal of allergy and clinical immunology.

[10]  F. Sun,et al.  Disappearance and metabolism of leukotriene B4 during carrageenan-induced pleurisy. , 1985, Biochemical pharmacology.

[11]  A. Wardlaw,et al.  Allergen‐Induced Release of Sulphidopeptide Leukotrienes (SRS‐A) and LTB4 in Allergic Rhinitis , 1985, Allergy.

[12]  S. Shak,et al.  Omega-oxidation is the major pathway for the catabolism of leukotriene B4 in human polymorphonuclear leukocytes. , 1984, The Journal of biological chemistry.

[13]  L. Lichtenstein,et al.  The transition from specific to nonspecific desensitization in human basophils. , 1981, Journal of immunology.

[14]  I. Gärtner.  Separation of human eosinophils in density gradients of polyvinylpyrrolidone-coated silica gel (Percoll). , 1980, Immunology.

[15]  P. Henson,et al.  Intracellular control of human neutrophil secretion. I. C5a-induced stimulus-specific desensitization and the effects of cytochalasin B. , 1978, Journal of immunology.

[16]  R. Simmons,et al.  Chemotaxis under agarose: a new and simple method for measuring chemotaxis and spontaneous migration of human polymorphonuclear leukocytes and monocytes. , 1975, Journal of immunology.