Parietal cortex activation predicts memory decline in apolipoprotein E-ϵϵ4 carriers
暂无分享,去创建一个
Jonas Persson | Lars-Göran Nilsson | Lars Nyberg | Martin Ingvar | Johanna Lind | Christine Van Broeckhoven | Kristel Sleegers | Rolf Adolfsson | L. Nyberg | M. Ingvar | K. Sleegers | C. van Broeckhoven | L. Nilsson | J. Lind | J. Persson | R. Adolfsson
[1] L. Nyberg,et al. Betula: A Prospective Cohort Study on Memory, Health and Aging , 2004 .
[2] Jonas Persson,et al. Reduced functional brain activity response in cognitively intact apolipoprotein E ε4 carriers , 2006 .
[3] D. Perani,et al. MCI conversion to dementia and the APOE genotype , 2004, Neurology.
[4] J C Mazziotta,et al. Apolipoprotein E type 4 allele and cerebral glucose metabolism in relatives at risk for familial Alzheimer disease. , 1995, JAMA.
[5] J. Mazziotta,et al. Cerebral metabolic and cognitive decline in persons at genetic risk for Alzheimer's disease. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[6] L. Fratiglioni,et al. Stability of the preclinical episodic memory deficit in Alzheimer's disease. , 2001, Brain : a journal of neurology.
[7] R. S. J. Frackowiak,et al. REGIONAL CEREBRAL OXYGEN SUPPLY AND UTILIZATION IN DEMENTIAA CLINICAL AND PHYSIOLOGICAL STUDY WITH OXYGEN-15 AND POSITRON TOMOGRAPHY A CLINICAL AND PHYSIOLOGICAL STUDY WITH OXYGEN - 15 AND POSITRON TOMOHRAPHY , 1981 .
[8] M. Pericak-Vance,et al. Apolipoprotein E: high-avidity binding to beta-amyloid and increased frequency of type 4 allele in late-onset familial Alzheimer disease. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[9] Jonas Persson,et al. Reduced functional brain activity response in cognitively intact apolipoprotein E epsilon4 carriers. , 2006, Brain : a journal of neurology.
[10] J C Mazziotta,et al. Mapping biochemistry to metabolism: FDG-PET and amyloid burden in Alzheimer's disease. , 1999, Neuroreport.
[11] Jonas Persson,et al. Reduced hippocampal volume in non-demented carriers of the apolipoprotein E ɛ4: Relation to chronological age and recognition memory , 2006, Neuroscience Letters.
[12] P. Bartenstein,et al. Association between Cognitive Performance and Cortical Glucose Metabolism in Patients with Mild Alzheimer’s Disease , 2005, Dementia and Geriatric Cognitive Disorders.
[13] M J de Leon,et al. Topography of cross-sectional and longitudinal glucose metabolic deficits in Alzheimer's disease. Pathophysiologic implications. , 1992, Archives of neurology.
[14] S. Thibodeau,et al. Preclinical evidence of Alzheimer's disease in persons homozygous for the epsilon 4 allele for apolipoprotein E. , 1996, The New England journal of medicine.
[15] R. Cabeza,et al. Imaging Cognition II: An Empirical Review of 275 PET and fMRI Studies , 2000, Journal of Cognitive Neuroscience.
[16] J. Baron,et al. Relations between hypometabolism in the posterior association neocortex and hippocampal atrophy in Alzheimer's disease: a PET/MRI correlative study , 2001, Journal of neurology, neurosurgery, and psychiatry.
[17] R. Buckner,et al. THE COGNITIVE NEUROSCIENCE OF REMEMBERING , 2001 .
[18] Lars Nyberg,et al. Memory functions and rCBF 99mTc-HMPAO SPET: developing diagnostics in Alzheimer's disease , 2002, European Journal of Nuclear Medicine and Molecular Imaging.
[19] T Jones,et al. Regional cerebral oxygen supply and utilization in dementia. A clinical and physiological study with oxygen-15 and positron tomography. , 1981, Brain : a journal of neurology.
[20] F. Fazio,et al. Impairment of Neocortical Metabolism Predicts Progression in Alzheimer’s Disease , 1999, Dementia and Geriatric Cognitive Disorders.