Oxidant, mitochondria and calcium: an overview.

[1]  D. H. Burgess,et al.  Involvement of cellular proteolytic machinery in apoptosis. , 1997, Biochemical and biophysical research communications.

[2]  S. Chakraborti,et al.  H2O2-induced lipid peroxidation in mitochondria of pulmonary vascular smooth muscle tissue and its modification by DFO, DMTU and DIDS. , 1996, Indian journal of experimental biology.

[3]  S. Chakraborti,et al.  Redox state of pyridine nucleotides, but not glutathione, regulate Ca2+ release by H2O2 from mitochondria of pulmonary smooth muscle. , 1996, Indian journal of biochemistry & biophysics.

[4]  J. Hsuan,et al.  Involvement of cyclophilin D in the activation of a mitochondrial pore by Ca2+ and oxidant stress. , 1996, European journal of biochemistry.

[5]  C. Richter,et al.  Peroxynitrite Stimulates the Pyridine Nucleotide-Linked Ca2+ Release from Intact Rat Liver Mitochondria† , 1996 .

[6]  B. Yu,et al.  4-Hydroxyhexenal Is a Potent Inducer of the Mitochondrial Permeability Transition (*) , 1996, The Journal of Biological Chemistry.

[7]  B. Herman,et al.  Mitochondrial free calcium transients during excitation‐contraction coupling in rabbit cardiac myocytes , 1996, FEBS letters.

[8]  G. Gores,et al.  Induction of the mitochondrial permeability transition by protease activity in rats: a mechanism of hepatocyte necrosis. , 1996, Gastroenterology.

[9]  A. Cossarizza,et al.  Control of apoptosis by the cellular ATP level , 1996, FEBS letters.

[10]  A. Vercesi,et al.  Opening of the mitochondrial permeability transition pore by uncoupling or inorganic phosphate in the presence of Ca2+ is dependent on mitochondrial‐generated reactive oxygen species , 1996, FEBS letters.

[11]  H. Klocker,et al.  Identification and purification of a bovine liver mitochondrial NAD+‐glycohydrolase , 1995, FEBS letters.

[12]  Shaoying Zhang,et al.  TNF‐α inhibits glucose‐induced insulin secretion in a pancreatic β‐cell line (INS‐1) , 1995 .

[13]  B. Lucas-Héron,et al.  Does calmitine, a protein specific for the mitochondrial matrix of skeletal muscle, play a key role in mitochondrial function? , 1995, FEBS letters.

[14]  Guido Kroemer,et al.  The biochemistry of programmed cell death , 1995, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[15]  N E Saris,et al.  Mitochondrial dysfunction in ischaemia‐reperfusion , 1995, Acta anaesthesiologica Scandinavica. Supplementum.

[16]  Guy C. Brown.  Nitric oxide regulates mitochondrial respiration and cell functions by inhibiting cytochrome oxidase , 1995, FEBS letters.

[17]  D. Miller,et al.  Intracellular effects of free radicals and reactive oxygen species in cardiac muscle. , 1995, Journal of human hypertension.

[18]  R. Schlapbach,et al.  Oxidants in mitochondria: from physiology to diseases. , 1995, Biochimica et biophysica acta.

[19]  Z. Oltvai,et al.  Reactive oxygen species and the regulation of cell death by the Bcl-2 gene family. , 1995, Biochimica et biophysica acta.

[20]  S. Orrenius,et al.  The role of intracellular oxidants in apoptosis. , 1995, Biochimica et biophysica acta.

[21]  B. Chernyak,et al.  Selective inhibition of the mitochondrial permeability transition pore at the oxidation‐reduction sensitive dithiol by monobromobimane , 1995, FEBS letters.

[22]  Packer Cs.  Changes in Arterial Smooth Muscle Contractility, Contractile Proteins, and Arterial Wall Structure in Spontaneous Hypertension , 1994 .

[23]  P. Bernardi,et al.  Recent progress on regulation of the mitochondrial permeability transition pore; a cyclosporin-sensitive pore in the inner mitochondrial membrane , 1994, Journal of bioenergetics and biomembranes.

[24]  K. Gunter,et al.  Transport of calcium by mitochondria , 1994, Journal of bioenergetics and biomembranes.

[25]  G. Dubyak,et al.  Evidence that BCL-2 represses apoptosis by regulating endoplasmic reticulum-associated Ca2+ fluxes. , 1994, Proceedings of the National Academy of Sciences of the United States of America.

[26]  J. Gutteridge,et al.  Biological origin of free radicals, and mechanisms of antioxidant protection. , 1994, Chemico-biological interactions.

[27]  B. Scatton.  The NMDA receptor complex , 1993, Fundamental & clinical pharmacology.

[28]  N. Takeyama,et al.  Oxidative damage to mitochondria is mediated by the Ca(2+)-dependent inner-membrane permeability transition. , 1993, The Biochemical journal.

[29]  J. Mccormack,et al.  The role of intramitochondrial Ca2+ in the regulation of oxidative phosphorylation in mammalian tissues. , 1993, Biochemical Society transactions.

[30]  C. Richter.  Pro‐oxidants and mitochondrial Ca2+: their relationship to apoptosis and oncogenesis , 1993, FEBS letters.

[31]  S. Chakraborti,et al.  Role of a membrane-associated serine esterase in the oxidant activation of phospholipase A2 by t-butyl hydroperoxide. , 1993, The Biochemical journal.

[32]  John Calvin Reed,et al.  Conversion of lytic to persistent alphavirus infection by the bcl-2 cellular oncogene , 1993, Nature.

[33]  M. A. Matlib,et al.  A role for the mitochondrial Na(+)-Ca2+ exchanger in the regulation of oxidative phosphorylation in isolated heart mitochondria. , 1993, The Journal of biological chemistry.

[34]  A. Vercesi,et al.  Calcium-dependent mitochondrial oxidative damage promoted by 5-aminolevulinic acid. , 1992, Biochimica et biophysica acta.

[35]  E. Albano,et al.  Mitochondrial damage and its role in causing hepatocyte injury during stimulation of lipid peroxidation by iron nitriloacetate. , 1992, Archives of biochemistry and biophysics.

[36]  C. Richter,et al.  'Pore' formation is not required for the hydroperoxide-induced Ca2+ release from rat liver mitochondria. , 1992, The Biochemical journal.

[37]  D. J. Reed,et al.  Cyclosporin A protects hepatocytes subjected to high Ca2+ and oxidative stress , 1992, FEBS letters.

[38]  W. Fiers,et al.  Cytotoxic activity of tumor necrosis factor is mediated by early damage of mitochondrial functions. Evidence for the involvement of mitochondrial radical generation. , 1992, The Journal of biological chemistry.

[39]  Y. Antonenko,et al.  Modulation of inner mitochondrial membrane channel activity , 1992, Journal of bioenergetics and biomembranes.

[40]  R. Hansford.  Dehydrogenase activation by Ca2+ in cells and tissues , 1991, Journal of bioenergetics and biomembranes.

[41]  K. Tasaka,et al.  Superoxide anion increases intracellular free calcium in human myometrial cells. , 1990, The Journal of biological chemistry.

[42]  A. Vercesi,et al.  Membrane protein thiol cross-linking associated with the permeabilization of the inner mitochondrial membrane by Ca2+ plus prooxidants. , 1990, The Journal of biological chemistry.

[43]  M. Theus,et al.  Cyclosporine A inhibits mitochondrial pyridine nucleotide hydrolysis and calcium release. , 1990, Biochemical pharmacology.

[44]  J. van Marle,et al.  The influence of nifedipine and mioflazine on mitochondrial calcium overload in normoxic and ischaemic guinea-pig hearts. , 1990, European journal of pharmacology.

[45]  M. Crompton,et al.  A heart mitochondrial Ca2(+)-dependent pore of possible relevance to re-perfusion-induced injury. Evidence that ADP facilitates pore interconversion between the closed and open states. , 1990, The Biochemical journal.

[46]  A. Angrilli,et al.  Mechanism of nitrofurantoin toxicity and oxidative stress in mitochondria. , 1988, Biochimica et biophysica acta.

[47]  D. Pfeiffer,et al.  The role of glutathione in the retention of Ca2+ by liver mitochondria. , 1984, The Journal of biological chemistry.

[48]  J. Crapo,et al.  Biology of disease: free radicals and tissue injury. , 1982, Laboratory investigation; a journal of technical methods and pathology.

[49]  S. Orrenius,et al.  Bleb formation in hepatocytes during drug metabolism is caused by disturbances in thiol and calcium ion homeostasis. , 1982, Science.

[50]  C. Richter,et al.  ATP prevents both hydroperoxide-induced hydrolysis of pyridine nucleotides and release of calcium in rat liver mitochondria. , 1981, European journal of biochemistry.

[51]  F. Zoccarato,et al.  Correlated effluxes of adenine nucleotides, Mg2+ and Ca2+ induced in rat-liver mitochondria by external Ca2+ and phosphate. , 1981, European journal of biochemistry.

[52]  E. Carafoli,et al.  Hydroperoxides can modulate the redox state of pyridine nucleotides and the calcium balance in rat liver mitochondria. , 1979, Proceedings of the National Academy of Sciences of the United States of America.

[53]  R. Haworth,et al.  The Ca2+-induced membrane transition in mitochondria. II. Nature of the Ca2+ trigger site. , 1979, Archives of biochemistry and biophysics.

[54]  D. Nicholls.  Calcium transport and porton electrochemical potential gradient in mitochondria from guinea-pig cerebral cortex and rat heart. , 1978, The Biochemical journal.

[55]  G. Kroemer.  [Mitochondrial control of apoptosis]. , 2001, Bulletin de l'Academie nationale de medecine.

[56]  M. Murphy,et al.  Cyclosporin A blocks 6-hydroxydopamine-induced efflux of Ca2+ from mitochondria without inactivating the mitochondrial inner-membrane pore. , 1994, The Biochemical journal.

[57]  G. Kass,et al.  Oxidative stress in mitochondria: its relationship to cellular Ca2+ homeostasis, cell death, proliferation, and differentiation. , 1991, Chemico-biological interactions.

[58]  B. Frei,et al.  Ca2+ release from mitochondria induced by prooxidants. , 1988, Free radical biology & medicine.

[59]  L. B. Chen,et al.  Mitochondrial membrane potential in living cells. , 1988, Annual review of cell biology.

[60]  E. Carafoli.  Intracellular calcium homeostasis. , 1987, Annual review of biochemistry.

[61]  B. Chance,et al.  Reactive oxygen intermediates in biochemistry. , 1986, Annual review of biochemistry.